Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

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Experimental autoimmune encephalomyelitis from a tissue energy perspective.Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies.

P2860

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Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

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http://www.wikidata.org/entity/Q37417523

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

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Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@nl

type

Item

label

Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@en

Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@nl

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Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@en

Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@nl

P1476

Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

@en

P2093

Benjamin Schattling

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Birgit Engeland

http://www.w3.org/2001/XMLSchema#string

Dirk Isbrandt

http://www.w3.org/2001/XMLSchema#string

Holger Lerche

http://www.w3.org/2001/XMLSchema#string

Walid Fazeli

http://www.w3.org/2001/XMLSchema#string

Yuanyuan Liu

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P2860

De novo pathogenic SCN8A mutation identified by whole-genome sequencing of a family quartet affected by infantile epileptic encephalopathy and SUDEP

Mutation of a new sodium channel gene, Scn8a, in the mouse mutant ‘motor endplate disease’

Neuronal death and perinatal lethality in voltage-gated sodium channel alpha(II)-deficient mice

Convulsive seizures and SUDEP in a mouse model of SCN8A epileptic encephalopathy.

Changed distribution of sodium channels along demyelinated axons

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger

Sodium accumulation is associated with disability and a progressive course in multiple sclerosis.

Impaired action potential initiation in GABAergic interneurons causes hyperexcitable networks in an epileptic mouse model carrying a human Na(V)1.1 mutation

Four novel tarantula toxins as selective modulators of voltage-gated sodium channel subtypes.

Sodium channel expression within chronic multiple sclerosis plaques.

P304

e89810

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scholarly article

P356

10.1172/JCI.INSIGHT.89810

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Manuel A. Friese

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2016-11-17T00:00:00Z

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27882351

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P921

multiple sclerosis

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5111507

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Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

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P2860

P2860

Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.

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type

label

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932