Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα. - Wikidata - awgldk - TriplyDB

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

http://www.wikidata.org/entity/Q34469234

about

Regulation of Estrogen Receptor α Expression in the Hypothalamus by Sex Steroids: Implication in the Regulation of Energy Homeostasis Role of non-neuronal cells in body weight and appetite control CCL5/RANTES contributes to hypothalamic insulin signaling for systemic insulin responsiveness through CCR5.Monomeric GLP-1/GIP/glucagon triagonism corrects obesity, hepatosteatosis, and dyslipidemia in female mice.Liver ERα regulates AgRP neuronal activity in the arcuate nucleus of female mice 17α-Estradiol Alleviates Age-related Metabolic and Inflammatory Dysfunction in Male Mice Without Inducing Feminization The sexual dimorphism of obesity.PGC-1α activity in nigral dopamine neurons determines vulnerability to α-synuclein Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice Low-carbohydrate, high-fat diets have sex-specific effects on bone health in rats.Hypothalamic inflammation and gliosis in obesity Unsaturated Fatty Acids Stimulate Tumor Growth through Stabilization of β-Catenin.Serum retinol binding protein 4 is negatively related to estrogen in Chinese women with obesity: a cross-sectional study.Discovery of tumor-specific irreversible inhibitors of stearoyl CoA desaturase PGC-1α expression in murine AgRP neurons regulates food intake and energy balance.Synergism between PGC-1α and estrogen in the survival of endometrial cancer cells via the mitochondrial pathway Sexually dimorphic brain fatty acid composition in low and high fat diet-fed mice.Adult Neurogenesis in the Female Mouse Hypothalamus: Estradiol and High-Fat Diet Alter the Generation of Newborn Neurons Expressing Estrogen Receptor α Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice Kinase Suppressor of Ras 2 (KSR2) expression in the brain regulates energy balance and glucose homeostasis.Sex differences in microglial CX3CR1 signalling determine obesity susceptibility in mice.High-Fat Diet Induces Unexpected Fatal Uterine Infections in Mice with aP2-Cre-mediated Deletion of Estrogen Receptor Alpha.A sexually dimorphic hypothalamic response to chronic high-fat diet consumption.Nutrient-sensing mechanisms in hypothalamic cell models: neuropeptide regulation and neuroinflammation in male- and female-derived cell lines.The effects of oestrogens and their receptors on cardiometabolic health.Glia: silent partners in energy homeostasis and obesity pathogenesis.Roles of estrogen receptor-alpha in mediating life span: the hypothalamic deregulation hypothesis.Anti-aging drugs reduce hypothalamic inflammation in a sex-specific manner.Hypothalamic Inflammation and Energy Balance Disruptions: Spotlight on Chemokines.Estrogen and insulin transport through the blood-brain barrier The effects of 17 alpha-estradiol to inhibit inflammation in vitro.Neuronal Dnmt1 Deficiency Attenuates Diet-Induced Obesity in Mice.Activation of Estrogen Response Element-Independent ERα Signaling Protects Female Mice From Diet-Induced Obesity.The brain needs interleukin-6 (IL-6) to maintain a "healthy" energy balance. Focus on "IL-6 ameliorates defective leptin sensitivity in DIO ventromedial hypothalamic nucleus neurons".Hypothalamic inflammation in obesity and metabolic disease.Estrogen receptor alpha protects pancreatic beta cells from apoptosis by preserving mitochondrial function and suppressing endoplasmic reticulum stress.Nucleus accumbens inflammation mediates anxiodepressive behavior and compulsive sucrose seeking elicited by saturated dietary fat.[Hypothalamic inflammation and energy balance deregulations: focus on chemokines.]Hallmarks of Brain Aging: Adaptive and Pathological Modification by Metabolic States.Astrocytes, emerging stars of energy homeostasis

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Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

http://www.wikidata.org/entity/Q34469234

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

@ast

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

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Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

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type

label

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

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Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

@en

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

@nl

prefLabel

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

@ast

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

@en

Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

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J.CELREP.2014.09.025

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Hypothalamic PGC-1α protects against high-fat diet exposure by regulating ERα.

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Aaron Frank

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Biff F Palmer

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Brian Finan

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Carlos Rodriguez Navas

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Dan L Li

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Deborah J Clegg

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Esther Fuente-Martin

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Eugenia Morselli

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Lisa Hahner

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Matthias H Tschöp

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P2860

Neuroprotective and disease-modifying effects of the ketogenic diet

Inflammation and metabolic disorders

Mutation analysis of peroxisome proliferator-activated receptor-gamma coactivator-1 (PGC-1) and relationships of identified amino acid polymorphisms to Type II diabetes mellitus

Estrogen resistance caused by a mutation in the estrogen-receptor gene in a man

Effect of single and compound knockouts of estrogen receptors alpha (ERalpha) and beta (ERbeta) on mouse reproductive phenotypes

An ADIOL-ERβ-CtBP transrepression pathway negatively regulates microglia-mediated inflammation

Defects in adaptive energy metabolism with CNS-linked hyperactivity in PGC-1alpha null mice

Definition of metabolic syndrome: Report of the National Heart, Lung, and Blood Institute/American Heart Association conference on scientific issues related to definition

Metabolic control through the PGC-1 family of transcription coactivators

Inflammatory mechanisms in obesity

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633-645

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10.1016/J.CELREP.2014.09.025

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2

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9

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Cristina García-Cáceres

Michael Neinast

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267270320

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25373903

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