Enhanced growth of a murine coronavirus in transformed mouse cells.
about
P1343
Functional and genetic analysis of coronavirus replicase-transcriptase proteinsRole of Endocytosis and Low pH in Murine Hepatitis Virus Strain A59 Cell EntryCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionCloning of the mouse hepatitis virus (MHV) receptor: expression in human and hamster cell lines confers susceptibility to MHVSeveral members of the mouse carcinoembryonic antigen-related glycoprotein family are functional receptors for the coronavirus mouse hepatitis virus-A59Bgp2, a new member of the carcinoembryonic antigen-related gene family, encodes an alternative receptor for mouse hepatitis virusesHigh-Resolution Analysis of Coronavirus Gene Expression by RNA Sequencing and Ribosome Profiling.Coronavirus species specificity: murine coronavirus binds to a mouse-specific epitope on its carcinoembryonic antigen-related receptor glycoprotein.Induction of apoptosis in murine coronavirus-infected cultured cells and demonstration of E protein as an apoptosis inducer.Intracellular complexes of viral spike and cellular receptor accumulate during cytopathic murine coronavirus infections.Resistance to murine hepatitis virus strain 3 is dependent on production of nitric oxideIdentification of mouse hepatitis virus papain-like proteinase 2 activity.Colocalization and membrane association of murine hepatitis virus gene 1 products and De novo-synthesized viral RNA in infected cells.Recombinant mouse hepatitis virus strain A59 from cloned, full-length cDNA replicates to high titers in vitro and is fully pathogenic in vivoSelective tropism of a neurotropic coronavirus for ependymal cells, neurons, and meningeal cells.Accessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Murine coronavirus replication induces cell cycle arrest in G0/G1 phase.Palmitoylations on murine coronavirus spike proteins are essential for virion assembly and infectivity.RNA replication of mouse hepatitis virus takes place at double-membrane vesicles.Murine coronavirus replication-induced p38 mitogen-activated protein kinase activation promotes interleukin-6 production and virus replication in cultured cells.Murine hepatitis virus strain 1 produces a clinically relevant model of severe acute respiratory syndrome in A/J mice.Comparative analysis of RNA genomes of mouse hepatitis viruses.Specific binding of host cellular proteins to multiple sites within the 3' end of mouse hepatitis virus genomic RNA.Persistent infection of cultured cells with mouse hepatitis virus (MHV) results from the epigenetic expression of the MHV receptor.Loss of resistance to murine hepatitis virus strain 3 infection after treatment with corticosteroids is associated with induction of macrophage procoagulant activity.Murine coronavirus membrane fusion is blocked by modification of thiols buried within the spike proteinTopographic changes in SARS coronavirus-infected cells at late stages of infectionAn In Vivo Selection Identifies Listeria monocytogenes Genes Required to Sense the Intracellular Environment and Activate Virulence Factor Expression.Induction of monocyte procoagulant activity by murine hepatitis virus type 3 parallels disease susceptibility in mice.Lung epithelial cells have virus-specific and shared gene expression responses to infection by diverse respiratory viruses.Requirements for CEACAMs and cholesterol during murine coronavirus cell entry.Coronavirus mRNA transcription: UV light transcriptional mapping studies suggest an early requirement for a genomic-length templateConformational change of the coronavirus peplomer glycoprotein at pH 8.0 and 37 degrees C correlates with virus aggregation and virus-induced cell fusion.Proteolytic cleavage of the E2 glycoprotein of murine coronavirus: activation of cell-fusing activity of virions by trypsin and separation of two different 90K cleavage fragments.Proteolytic cleavage of the E2 glycoprotein of murine coronavirus: host-dependent differences in proteolytic cleavage and cell fusion.Coronavirus minus-strand RNA synthesis and effect of cycloheximide on coronavirus RNA synthesis.Genetic resistance to mouse hepatitis virus correlates with absence of virus-binding activity on target tissuesCell-free translation of murine coronavirus RNA.Differentiated phenotypes of primary murine alveolar epithelial cells and their susceptibility to infection by respiratory viruses.Genetic analysis of murine hepatitis virus strain JHM.
P2860
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P2860
Enhanced growth of a murine coronavirus in transformed mouse cells.
description
1972 nî lūn-bûn
@nan
1972 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1972 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1972年の論文
@ja
1972年論文
@yue
1972年論文
@zh-hant
1972年論文
@zh-hk
1972年論文
@zh-mo
1972年論文
@zh-tw
1972年论文
@wuu
name
Enhanced growth of a murine coronavirus in transformed mouse cells.
@ast
Enhanced growth of a murine coronavirus in transformed mouse cells.
@en
Enhanced growth of a murine coronavirus in transformed mouse cells.
@nl
type
label
Enhanced growth of a murine coronavirus in transformed mouse cells.
@ast
Enhanced growth of a murine coronavirus in transformed mouse cells.
@en
Enhanced growth of a murine coronavirus in transformed mouse cells.
@nl
prefLabel
Enhanced growth of a murine coronavirus in transformed mouse cells.
@ast
Enhanced growth of a murine coronavirus in transformed mouse cells.
@en
Enhanced growth of a murine coronavirus in transformed mouse cells.
@nl
P2860
P1476
Enhanced growth of a murine coronavirus in transformed mouse cells
@en
P2093
Sturman LS
Takemoto KK
P2860
P304
P407
P577
1972-10-01T00:00:00Z