Induction of monocyte procoagulant activity by murine hepatitis virus type 3 parallels disease susceptibility in mice.
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Absence of monocyte procoagulant activity during the immune response to influenza virusProtein Interferon-Stimulated Gene 15 Conjugation Delays but Does Not Overcome Coronavirus Proliferation in a Model of Fulminant HepatitisAdhesive receptor Mac-1 coordinates the activation of factor X on stimulated cells of monocytic and myeloid differentiation: an alternative initiation of the coagulation protease cascade.Fibrin depletion decreases inflammation and delays the onset of demyelination in a tumor necrosis factor transgenic mouse model for multiple sclerosis.Resistance to murine hepatitis virus strain 3 is dependent on production of nitric oxideProgrammed death (PD)-1-deficient mice are extremely sensitive to murine hepatitis virus strain-3 (MHV-3) infection.Coupling of the adhesive receptor CD11b/CD18 to functional enhancement of effector macrophage tissue factor response.Regulatory roles of T mu and T gamma cells in the collaborative cellular initiation of the extrinsic coagulation pathway by bacterial lipopolysaccharide.Tissue factor induction in human monocytes. Two distinct mechanisms displayed by different alloantigen-responsive T cell clones.The novel CD4+CD25+ regulatory T cell effector molecule fibrinogen-like protein 2 contributes to the outcome of murine fulminant viral hepatitis.Genome-wide immunity studies in the rabbit: transcriptome variations in peripheral blood mononuclear cells after in vitro stimulation by LPS or PMA-IonomycinThe Fgl2/fibroleukin prothrombinase contributes to immunologically mediated thrombosis in experimental and human viral hepatitisMurine hepatitis virus strain 1 produces a clinically relevant model of severe acute respiratory syndrome in A/J mice.Novel antibody against a glutamic acid-rich human fibrinogen-like protein 2-derived peptide near Ser91 inhibits hfgl2 prothrombinase activityAssociation of mouse fibrinogen-like protein with murine hepatitis virus-induced prothrombinase activity.Pattern of disease after murine hepatitis virus strain 3 infection correlates with macrophage activation and not viral replication.Induction of macrophage procoagulant activity by murine hepatitis virus strain 3: role of tyrosine phosphorylationLoss of resistance to murine hepatitis virus strain 3 infection after treatment with corticosteroids is associated with induction of macrophage procoagulant activity.Monoclonal antiprothrombinase (3D4.3) prevents mortality from murine hepatitis virus (MHV-3) infection.Fulminant hepatic failure in murine hepatitis virus strain 3 infection: tissue-specific expression of a novel fgl2 prothrombinase.Mechanisms for virus-induced liver disease: tumor necrosis factor-mediated pathology independent of natural killer and T cells during murine cytomegalovirus infection.Increased monocyte tissue factor expression in coronary disease.Platelet-activating factor modulates endotoxin-induced macrophage procoagulant activity by a protein kinase C-dependent mechanism.Protective effects of long pentraxin PTX3 on lung injury in a severe acute respiratory syndrome model in mice.Disparate mechanisms of induction of procoagulant activity by live and inactivated bacteria and viruses.16, 16 Dimethyl prostaglandin E2 prevents the development of fulminant hepatitis and blocks the induction of monocyte/macrophage procoagulant activity after murine hepatitis virus strain 3 infectionAllogeneic induction of the human T cell-instructed monocyte procoagulant response is rapid and is elicited by HLA-DR.The Role of FGL2 in the Pathogenesis and Treatment of Hepatitis C Virus Infection.Roles for thrombin and fibrin(ogen) in cytokine/chemokine production and macrophage adhesion in vivo.Selective cellular expression of tissue factor in human tissues. Implications for disorders of hemostasis and thrombosis.Macrophage heterogeneity in thromboplastin response.Monocyte expression of tissue factor and adhesion molecules: the link with accelerated coronary artery disease in patients with chronic renal failure.Macrophage scavenger receptor 1 contributes to pathogenesis of fulminant hepatitis via neutrophil mediated complement activation.Expression of B and T lymphocyte attenuator (BTLA) in macrophages contributes to the fulminant hepatitis caused by murine hepatitis virus strain-3.Mouse hepatitis virus 3 binding to macrophages correlates with resistance to experimental infection.The nucleocapsid protein of murine hepatitis virus type 3 induces transcription of the novel fgl2 prothrombinase gene.Fgl2 deficiency causes neonatal death and cardiac dysfunction during embryonic and postnatal development in mice.
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P2860
Induction of monocyte procoagulant activity by murine hepatitis virus type 3 parallels disease susceptibility in mice.
description
1981 nî lūn-bûn
@nan
1981年の論文
@ja
1981年学术文章
@wuu
1981年学术文章
@zh-cn
1981年学术文章
@zh-hans
1981年学术文章
@zh-my
1981年学术文章
@zh-sg
1981年學術文章
@yue
1981年學術文章
@zh
1981年學術文章
@zh-hant
name
Induction of monocyte procoagu ...... isease susceptibility in mice.
@ast
Induction of monocyte procoagu ...... isease susceptibility in mice.
@en
type
label
Induction of monocyte procoagu ...... isease susceptibility in mice.
@ast
Induction of monocyte procoagu ...... isease susceptibility in mice.
@en
prefLabel
Induction of monocyte procoagu ...... isease susceptibility in mice.
@ast
Induction of monocyte procoagu ...... isease susceptibility in mice.
@en
P2093
P2860
P356
P1476
Induction of monocyte procoagu ...... isease susceptibility in mice.
@en
P2093
J L Leibowitz
T S Edgington
P2860
P304
P356
10.1084/JEM.154.4.1150
P407
P577
1981-10-01T00:00:00Z