KLF4, p21 and context-dependent opposing forces in cancer.
about
Krüppel-like factors: three fingers in controlALDH1A1 maintains ovarian cancer stem cell-like properties by altered regulation of cell cycle checkpoint and DNA repair network signalingChallenges to the clinical application of pluripotent stem cells: towards genomic and functional stabilityKrüppel-like factor 4 regulates genetic stability in mouse embryonic fibroblastsHNF4 alpha orchestrates a set of 14 genes to down-regulate cell proliferation in kidney cellsThe human synMuv-like protein LIN-9 is required for transcription of G2/M genes and for entry into mitosisMammalian Krüppel-like factors in health and diseasesThe diverse functions of Krüppel-like factors 4 and 5 in epithelial biology and pathobiologyCancer Stem Cell Hierarchy in Glioblastoma MultiformeEndothelial Transdifferentiation of Tumor Cells Triggered by the Twist1-Jagged1-KLF4 Axis: Relationship between Cancer Stemness and AngiogenesisCommon stemness regulators of embryonic and cancer stem cellsClinical Potential of microRNA-7 in CancerThe genomic stability of induced pluripotent stem cells3,6-Epidioxy-1,10-bisaboladiene inhibits G1 -specific transcription through Swi4/Swi6 and Mbp1/Swi6 via the Hog1 stress pathway in yeast.eIF4GI links nutrient sensing by mTOR to cell proliferation and inhibition of autophagyDeficiency of the Kruppel-like factor KLF4 correlates with increased cell proliferation and enhanced skin tumorigenesisAblation of PRMT6 reveals a role as a negative transcriptional regulator of the p53 tumor suppressorAP-2 factors act in concert with Notch to orchestrate terminal differentiation in skin epidermisConditional deletion of the mouse Klf4 gene results in corneal epithelial fragility, stromal edema, and loss of conjunctival goblet cellsDissecting direct reprogramming through integrative genomic analysisp21 in cancer: intricate networks and multiple activitiesA lentiviral functional proteomics approach identifies chromatin remodeling complexes important for the induction of pluripotency.Chromosome missegregation rate predicts whether aneuploidy will promote or suppress tumors.Interplay between arginine methylation and ubiquitylation regulates KLF4-mediated genome stability and carcinogenesis.Atomic force microscopy study of the specific adhesion between a colloid particle and a living melanoma cell: Effect of the charge and the hydrophobicity of the particle surfaceKnockdown of MBP-1 in human foreskin fibroblasts induces p53-p21 dependent senescence.Nuclear expression of KLF6 tumor suppressor factor is highly associated with overexpression of ERBB2 oncoprotein in ductal breast carcinomasPlatelet factor 4 regulation of monocyte KLF4 in experimental cerebral malaria.RECLU: a pipeline to discover reproducible transcriptional start sites and their alternative regulation using capped analysis of gene expression (CAGE).Expression of Krüppel-like factor 4 in breast cancer tissues and its effects on the proliferation of breast cancer MDA-MB-231 cells.YAP-Hippo signalling downstream of leukemia inhibitory factor receptor: implications for breast cancer.KLF4 Plays an Essential Role in Corneal Epithelial Homeostasis by Promoting Epithelial Cell Fate and Suppressing Epithelial-Mesenchymal Transition.Dependence of p53-deficient cells on the DHX9 DExH-box helicase.Inhibition of KLF4 by Statins Reverses Adriamycin-Induced Metastasis and Cancer Stemness in Osteosarcoma CellsSingle-nucleotide polymorphisms in the p53 signaling pathway.Differential control of Notch1 gene transcription by Klf4 and Sp3 transcription factors in normal versus cancer-derived keratinocytes.Mammary cancer chemoprevention by withaferin A is accompanied by in vivo suppression of self-renewal of cancer stem cellsEKLF directly activates the p21WAF1/CIP1 gene by proximal promoter and novel intronic regulatory regions during erythroid differentiationGenetic deletion of Klf4 in the mouse intestinal epithelium ameliorates dextran sodium sulfate-induced colitis by modulating the NF-κB pathway inflammatory responseKLF family members regulate intrinsic axon regeneration ability.
P2860
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P2860
KLF4, p21 and context-dependent opposing forces in cancer.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
KLF4, p21 and context-dependent opposing forces in cancer.
@ast
KLF4, p21 and context-dependent opposing forces in cancer.
@en
KLF4, p21 and context-dependent opposing forces in cancer.
@nl
type
label
KLF4, p21 and context-dependent opposing forces in cancer.
@ast
KLF4, p21 and context-dependent opposing forces in cancer.
@en
KLF4, p21 and context-dependent opposing forces in cancer.
@nl
prefLabel
KLF4, p21 and context-dependent opposing forces in cancer.
@ast
KLF4, p21 and context-dependent opposing forces in cancer.
@en
KLF4, p21 and context-dependent opposing forces in cancer.
@nl
P356
P1476
KLF4, p21 and context-dependent opposing forces in cancer.
@en
P2093
Benjamin D Rowland
Daniel S Peeper
P2888
P356
10.1038/NRC1780
P407
P577
2006-01-01T00:00:00Z
P6179
1053134180