Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells. - Wikidata - awgldk - TriplyDB

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

http://www.wikidata.org/entity/Q34479204

about

The ability to generate senescent progeny as a mechanism underlying breast cancer cell heterogeneity Genome-wide transcriptional reorganization associated with senescence-to-immortality switch during human hepatocellular carcinogenesis SIP1 is downregulated in hepatocellular carcinoma by promoter hypermethylation Overexpression of ZEB2 in peritumoral liver tissue correlates with favorable survival after curative resection of hepatocellular carcinoma.Novel anti-HER2 monoclonal antibodies: synergy and antagonism with tumor necrosis factor-α.Protein expression of ZEB2 in renal cell carcinoma and its prognostic significance in patient survival Common telomerase reverse transcriptase promoter mutations in hepatocellular carcinomas from different geographical locations.SIP1 is a downstream effector of GADD45G in senescence induction and growth inhibition of liver tumor cells.SIP1 protein protects cells from DNA damage-induced apoptosis and has independent prognostic value in bladder cancer.ZEB/miR-200 feedback loop: at the crossroads of signal transduction in cancer.EMT-activating transcription factors in cancer: beyond EMT and tumor invasiveness.Oncogenic roles of EMT-inducing transcription factors.Short stretches of rare codons regulate translation of the transcription factor ZEB2 in cancer cells.Expanding roles of ZEB factors in tumorigenesis and tumor progression.Reactive oxygen species generation is essential for cisplatin-induced accelerated senescence in hepatocellular carcinoma.Aflatoxin genotoxicity is associated with a defective DNA damage response bypassing p53 activation.Inhibition of Akt signaling in hepatoma cells induces apoptotic cell death independent of Akt activation status.Transforming growth factor-beta induces senescence in hepatocellular carcinoma cells and inhibits tumor growth.HCV core inhibits hepatocellular carcinoma cell replicative senescence through downregulating microRNA-138 expression.Large liver cell change in hepatitis B virus-related liver cirrhosis.Development of a novel zebrafish xenograft model in ache mutants using liver cancer cell lines.

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P2860

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

http://www.wikidata.org/entity/Q34479204

description

2006 nî lūn-bûn

@nan

2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@ast

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@en

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@nl

type

label

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@ast

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@en

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@nl

prefLabel

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@ast

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@en

Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells.

@nl

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PNAS.0510877103

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Proceedings of the National Academy of Sciences of the United States of America

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Reprogramming of replicative senescence in hepatocellular carcinoma-derived cells

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Bala Gur

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Cengiz Yakicier

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Isik Yulug

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Mehmet Ozturk

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Mesut Tez

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Mine Mumcuoglu

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Nuri Ozturk

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Ozden Yalcin

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Tamer Yagci

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P2860

Multiple tumor suppressor pathways negatively regulate telomerase

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Regulation of Smad signaling through a differential recruitment of coactivators and corepressors by ZEB proteins

The clonal evolution of tumor cell populations

Myc activates telomerase

Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors

A survey of telomerase activity in human cancer

The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion

Growth of human hepatoma cells lines with differentiated functions in chemically defined medium

SIP1, a novel zinc finger/homeodomain repressor, interacts with Smad proteins and binds to 5'-CACCT sequences in candidate target genes.

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