Transforming growth factor-beta induces senescence in hepatocellular carcinoma cells and inhibits tumor growth.
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Noncoding RNA control of cellular senescenceRole of NADPH oxidases in the redox biology of liver fibrosisBiochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular systemComplexities of TGF-β targeted cancer therapy.TGF-β Signaling in Bone Remodeling and Osteosarcoma ProgressionTransforming Growth Factor-Beta and Oxidative Stress Interplay: Implications in Tumorigenesis and Cancer ProgressionGrowth arrest and DNA damage 45G down-regulation contributes to Janus kinase/signal transducer and activator of transcription 3 activation and cellular senescence evasion in hepatocellular carcinomaNADPH oxidase subunit 4-mediated reactive oxygen species contribute to cycling hypoxia-promoted tumor progression in glioblastoma multiformeGenome-wide transcriptional reorganization associated with senescence-to-immortality switch during human hepatocellular carcinogenesisThe FYVE domain of Smad Anchor for Receptor Activation (SARA) is required to prevent skin carcinogenesis, but not in mouse developmentThe liver-specific tumor suppressor STAT5 controls expression of the reactive oxygen species-generating enzyme NOX4 and the proapoptotic proteins PUMA and BIM in mice.Serotonin Activated Hepatic Stellate Cells Contribute to Sex Disparity in Hepatocellular Carcinoma.Lentivirus-mediated Nox4 shRNA invasion and angiogenesis and enhances radiosensitivity in human glioblastoma.TGF-β1 induces senescence of bone marrow mesenchymal stem cells via increase of mitochondrial ROS productionSuppression of Transforming Growth Factor-β Signaling Delays Cellular Senescence and Preserves the Function of Endothelial Cells Derived from Human Pluripotent Stem Cells.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Coordinated control of senescence by lncRNA and a novel T-box3 co-repressor complex.4-O-Methylhonokiol Protects HaCaT Cells from TGF-β1-Induced Cell Cycle Arrest by Regulating Canonical and Non-Canonical Pathways of TGF-β SignalingOxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.Negative regulation of NADPH oxidase 4 by hydrogen peroxide-inducible clone 5 (Hic-5) proteinNovel anti-HER2 monoclonal antibodies: synergy and antagonism with tumor necrosis factor-α.Are there roles for brain cell senescence in aging and neurodegenerative disorders?TGF-β signaling is often attenuated during hepatotumorigenesis, but is retained for the malignancy of hepatocellular carcinoma cellsTGF-beta1 does not induce senescence of multipotent mesenchymal stromal cells and has similar effects in early and late passages.2, 3, 7, 8-Tetrachlorodibenzo-P-dioxin (TCDD) induces premature senescence in human and rodent neuronal cells via ROS-dependent mechanismsHepatitis B virus core promoter mutations contribute to hepatocarcinogenesis by deregulating SKP2 and its target, p21Regulation of TGF-β1-Induced Pro-Apoptotic Signaling by Growth Factor Receptors and Extracellular Matrix Receptor Integrins in the Liver.miR-34a induces cellular senescence via modulation of telomerase activity in human hepatocellular carcinoma by targeting FoxM1/c-Myc pathwayIn Vivo Senescence in the Sbds-Deficient Murine Pancreas: Cell-Type Specific Consequences of Translation Insufficiency.Sirtuin 6 promotes transforming growth factor-β1/H2O2/HOCl-mediated enhancement of hepatocellular carcinoma cell tumorigenicity by suppressing cellular senescence.Transactivation of the TIEG1 confers growth inhibition of transforming growth factor-β-susceptible hepatocellular carcinoma cells.Chronic inflammation, immune escape, and oncogenesis in the liver: a unique neighborhood for novel intersections.Insights Into SMAD4 Loss in Pancreatic Cancer From Inducible Restoration of TGF-β Signaling.The complexities of TGF-β action during mammary and squamous cell carcinogenesis.c-Jun N-terminal kinase inhibitor favors transforming growth factor-β to antagonize hepatitis B virus X protein-induced cell growth promotion in hepatocellular carcinomaSIP1 is a downstream effector of GADD45G in senescence induction and growth inhibition of liver tumor cells.Co-expression of LASS2 and TGF-β1 predicts poor prognosis in hepatocellular carcinoma.Dual inhibiting senescence and epithelial-to-mesenchymal transition by erythropoietin preserve tubular epithelial cell regeneration and ameliorate renal fibrosis in unilateral ureteral obstruction.TGF-β and NF-κB signaling pathway crosstalk potentiates corneal epithelial senescence through an RNA stress responseIntrahepatic microcirculatory disorder, parenchymal hypoxia and NOX4 upregulation result in zonal differences in hepatocyte apoptosis following lipopolysaccharide- and D-galactosamine-induced acute liver failure in rats.
P2860
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P2860
Transforming growth factor-beta induces senescence in hepatocellular carcinoma cells and inhibits tumor growth.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@en
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@nl
type
label
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@en
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@nl
prefLabel
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@en
Transforming growth factor-bet ...... lls and inhibits tumor growth.
@nl
P2093
P2860
P356
P1433
P1476
Transforming growth factor-bet ...... ells and inhibits tumor growth
@en
P2093
Burcu Cingoz
Mehmet Ozturk
Mine Mumcuoglu
Ozge Gursoy-Yuzugullu
P2860
P304
P356
10.1002/HEP.23769
P407
P577
2010-09-01T00:00:00Z