A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy.
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A friend within the heart: natriuretic peptide receptor signalingDefective cellular trafficking of missense NPR-B mutants is the major mechanism underlying acromesomelic dysplasia-type MaroteauxSildenafil citrate increases myocardial cGMP content in rat heart, decreases its hypertrophic response to isoproterenol and decreases myocardial leak of creatine kinase and troponin TNatriuretic Peptides in the Regulation of Cardiovascular Physiology and Metabolic EventsNatriuretic Peptides and Cardiometabolic HealthPhosphodiesterases and cardiac cGMP: evolving roles and controversiesAtrial natriuretic peptide in cardiovascular biology and disease (NPPA)Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivoCardiac hypertrophy in transgenic rats expressing a dominant-negative mutant of the natriuretic peptide receptor B.cGMP-phosphodiesterase activity is up-regulated in response to pressure overload of rat ventriclesAtrial natriuretic peptide induces natriuretic peptide receptor-cGMP-dependent protein kinase interactionSmooth muscle-selective deletion of guanylyl cyclase-A prevents the acute but not chronic effects of ANP on blood pressureHypertension in mice lacking the proatrial natriuretic peptide convertase corinNatriuretic peptides and the genomics of left-ventricular hypertrophy.Natriuretic Peptide Signaling via Guanylyl Cyclase (GC)-A: An Endogenous Protective Mechanism of the Heart.Human hypertension is characterized by a lack of activation of the antihypertensive cardiac hormones ANP and BNP.KMUP-1 attenuates isoprenaline-induced cardiac hypertrophy in rats through NO/cGMP/PKG and ERK1/2/calcineurin A pathwaysCardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.Influence of natriuretic peptide receptor-1 on survival and cardiac hypertrophy during developmentClass IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy.Novel mutations in natriuretic peptide receptor-2 gene underlie acromesomelic dysplasia, type maroteaux.Protein kinase G I and heart failure: Shifting focus from vascular unloading to direct myocardial antiremodeling effects.Myocardial remodelling: pharmacological targets.Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.Natriuretic peptides: their structures, receptors, physiologic functions and therapeutic applicationsStructure, signaling mechanism and regulation of the natriuretic peptide receptor guanylate cyclase.Regulation of cardiac angiotensin-converting enzyme and angiotensin AT1 receptor gene expression in Npr1 gene-disrupted miceCommon variations in noncoding regions of the human natriuretic peptide receptor A gene have quantitative effects.Increased atherosclerosis and smooth muscle cell hypertrophy in natriuretic peptide receptor A-/-apolipoprotein E-/- mice.The evolving role of nesiritide in advanced or decompensated heart failure.Progressive cardiac hypertrophy and dysfunction in atrial natriuretic peptide receptor (GC-A) deficient miceNitric oxide-cyclic GMP signaling in stem cell differentiationGuanylyl cyclase / atrial natriuretic peptide receptor-A: role in the pathophysiology of cardiovascular regulation.Natriuretic peptide receptor guanylyl cyclase-A protects podocytes from aldosterone-induced glomerular injuryCardioprotective actions of cyclic GMP: lessons from genetic animal models.Regression of copper-deficient heart hypertrophy: reduction in the size of hypertrophic cardiomyocytes.Effects of ozone and particulate matter on cardiac mechanics: role of the atrial natriuretic peptide gene.Inhibitors of cyclic nucleotide phosphodiesterase 3 and 5 as therapeutic agents in heart failure.Membrane guanylyl cyclase receptors: an updateRegulation and therapeutic targeting of peptide-activated receptor guanylyl cyclases.
P2860
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P2860
A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@ast
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@en
A genetic model provides evidence that the receptor for atrial natriuretic peptide
@nl
type
label
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@ast
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@en
A genetic model provides evidence that the receptor for atrial natriuretic peptide
@nl
prefLabel
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@ast
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@en
A genetic model provides evidence that the receptor for atrial natriuretic peptide
@nl
P2093
P2860
P356
P1476
A genetic model provides evide ...... ntricular myocyte hypertrophy.
@en
P2093
P2860
P304
P356
10.1073/PNAS.051625598
P407
P577
2001-02-13T00:00:00Z