Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes. - Wikidata - awgldk - TriplyDB

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

http://www.wikidata.org/entity/Q34668034

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Cardiac cAMP: production, hydrolysis, modulation and detection Therapeutic potential of PDE modulation in treating heart disease Phosphodiesterases and cardiac cGMP: evolving roles and controversies The transcription factor GATA-6 regulates pathological cardiac hypertrophy Role of Ca2+/calmodulin-stimulated cyclic nucleotide phosphodiesterase 1 in mediating cardiomyocyte hypertrophy.Concerted regulation of cGMP and cAMP phosphodiesterases in early cardiac hypertrophy induced by angiotensin II Cardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.Cardiac cyclic nucleotide phosphodiesterases: function, regulation, and therapeutic prospects.Targeting cyclic nucleotide phosphodiesterase in the heart: therapeutic implications.Distinct patterns of constitutive phosphodiesterase activity in mouse sinoatrial node and atrial myocardium.Conserved expression and functions of PDE4 in rodent and human heart.Increased cardiac myocyte PDE5 levels in human and murine pressure overload hypertrophy contribute to adverse LV remodeling Cyclic nucleotide phosphodiesterases: important signaling modulators and therapeutic targets.Regulation of sarcoplasmic reticulum Ca2+ ATPase 2 (SERCA2) activity by phosphodiesterase 3A (PDE3A) in human myocardium: phosphorylation-dependent interaction of PDE3A1 with SERCA2.Expression, activity, and pro-hypertrophic effects of PDE5A in cardiac myocytes.Myocardial Response to Milrinone in Single Right Ventricle Heart Disease.cGMP-hydrolytic activity and its inhibition by sildenafil in normal and failing human and mouse myocardium Combination therapy with beta-adrenergic receptor antagonists and phosphodiesterase inhibitors for chronic heart failure.PDE1C deficiency antagonizes pathological cardiac remodeling and dysfunction.Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics.Clinical and molecular genetics of the phosphodiesterases (PDEs).Cardiac Adenylyl Cyclase and Phosphodiesterase Expression Profiles Vary by Age, Disease, and Chronic Phosphodiesterase Inhibitor Treatment.PDE3 inhibition in dilated cardiomyopathy.Cyclic nucleotide phosphodiesterase (PDE) isozymes as targets of the intracellular signalling network: benefits of PDE inhibitors in various diseases and perspectives for future therapeutic developments.Phosphodiesterases and subcellular compartmentalized cAMP signaling in the cardiovascular system.Phosphodiesterases and cyclic GMP regulation in heart muscle.Calmodulin in a heartbeat.Ca(2+)/calmodulin-activated phosphodiesterase 1A is highly expressed in rabbit cardiac sinoatrial nodal cells and regulates pacemaker function.Cardiac Phosphodiesterases and Their Modulation for Treating Heart Disease.Generation and phenotypic characterization of Pde1a mutant mice.Phosphodiesterase 1: A Unique Drug Target for Degenerative Diseases and Cognitive Dysfunction.Controlling myocyte cGMP: phosphodiesterase 1 joins the fray.Imaging of PDE2- and PDE3-Mediated cGMP-to-cAMP Cross-Talk in Cardiomyocytes.cAMP hydrolysis by Cam-PDE 1 Roles of PDE1 in Pathological Cardiac Remodeling and Dysfunction.

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P2860

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

http://www.wikidata.org/entity/Q34668034

description

2007 nî lūn-bûn

@nan

2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2007年の論文

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2007年論文

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2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

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2007年論文

@zh-tw

2007年论文

@wuu

name

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@ast

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@en

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@nl

type

label

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

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Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@en

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@nl

prefLabel

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@ast

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@en

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@nl

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P1433

Journal of Biological Chemistry

P1476

Cyclic nucleotide phosphodiesterase PDE1C1 in human cardiac myocytes.

@en

P2093

Fabrice Vandeput

http://www.w3.org/2001/XMLSchema#string

Judith Krall

http://www.w3.org/2001/XMLSchema#string

Kim N McCaw

http://www.w3.org/2001/XMLSchema#string

Lothar Uher

http://www.w3.org/2001/XMLSchema#string

Matthew A Movsesian

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Przemyslaw B Radwanski

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Ryan Hambleton

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Sharon L Wolda

http://www.w3.org/2001/XMLSchema#string

Vincent Florio

http://www.w3.org/2001/XMLSchema#string

P2860

Isolation and characterization of cDNAs corresponding to two human calcium, calmodulin-regulated, 3',5'-cyclic nucleotide phosphodiesterases

cGMP-stimulated cyclic nucleotide phosphodiesterase regulates the basal calcium current in human atrial myocytes

Sildenafil citrate increases myocardial cGMP content in rat heart, decreases its hypertrophic response to isoproterenol and decreases myocardial leak of creatine kinase and troponin T

Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice

Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes.

Role of sodium-calcium exchanger in modulating the action potential of ventricular myocytes from normal and failing hearts.

PDE7A is expressed in human B-lymphocytes and is up-regulated by elevation of intracellular cAMP.

Sarcoplasmic reticulum-associated cyclic adenosine 5'-monophosphate phosphodiesterase activity in normal and failing human hearts.

A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy.

Potentiation of slow component of delayed rectifier K(+) current by cGMP via two distinct mechanisms: inhibition of phosphodiesterase 3 and activation of protein kinase G.

P304

32749-32757

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scholarly article

P356

10.1074/JBC.M703173200

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P407

P433

45

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P478

282

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P577

2007-08-28T00:00:00Z

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P698

17726023

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