Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo. - Wikidata - awgldk - TriplyDB

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

http://www.wikidata.org/entity/Q34508519

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AID and APOBECs span the gap between innate and adaptive immunity Tetherin/BST-2: Restriction Factor or Immunomodulator?Noninfectious retrovirus particles drive the APOBEC3/Rfv3 dependent neutralizing antibody response Type I interferon signaling is required for the APOBEC3/Rfv3-dependent neutralizing antibody response but not innate retrovirus restriction.Immunoglobulin somatic hypermutation by APOBEC3/Rfv3 during retroviral infection Toll-like receptor 7 controls the anti-retroviral germinal center response Two genetic determinants acquired late in Mus evolution regulate the inclusion of exon 5, which alters mouse APOBEC3 translation efficiency Mouse knockout models for HIV-1 restriction factors.Persistent Friend virus replication and disease in Apobec3-deficient mice expressing functional B-cell-activating factor receptor.Fv1 restriction and retrovirus vaccine immunity in Apobec3-deficient 129P2 mice.Requirement for Fc effector mechanisms in the APOBEC3/Rfv3-dependent neutralizing antibody response Brain tumor eradication and prolonged survival from intratumoral conversion of 5-fluorocytosine to 5-fluorouracil using a nonlytic retroviral replicating vector Negative impact of IFN-γ on early host immune responses to retroviral infection.Tetherin/BST-2 promotes dendritic cell activation and function during acute retrovirus infection.APOBEC3 inhibition of mouse mammary tumor virus infection: the role of cytidine deamination versus inhibition of reverse transcription Restriction by SAMHD1 Limits cGAS/STING-Dependent Innate and Adaptive Immune Responses to HIV-1.Ribonuclease L is not critical for innate restriction and adaptive immunity against Friend retrovirus infection Humoral immunity in the Friend retrovirus infection model.IFN-α treatment inhibits acute Friend retrovirus replication primarily through the antiviral effector molecule Apobec3.Viral subversion of APOBEC3s: Lessons for anti-tumor immunity and tumor immunotherapy.

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Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

http://www.wikidata.org/entity/Q34508519

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@ast

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@en

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@nl

type

label

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@ast

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@en

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@nl

prefLabel

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@ast

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@en

Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@nl

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JIMMUNOL.1001143

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Journal of Immunology

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Innate retroviral restriction by Apobec3 promotes antibody affinity maturation in vivo.

@en

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Kim J Hasenkrug

http://www.w3.org/2001/XMLSchema#string

Mauricio Montano

http://www.w3.org/2001/XMLSchema#string

Robert L Benitez

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Warner C Greene

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P2860

The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA

Human APOBEC3F is another host factor that blocks human immunodeficiency virus type 1 replication

Human immunodeficiency virus type 1 cDNAs produced in the presence of APOBEC3G exhibit defects in plus-strand DNA transfer and integration

Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcripts

DNA deamination mediates innate immunity to retroviral infection

Hypermutation of HIV-1 DNA in the absence of the Vif protein

HIV-1 Vif blocks the antiviral activity of APOBEC3G by impairing both its translation and intracellular stability

Polyclonal B cell activation in infections: infectious agents' devilry or defense mechanism of the host?

Apobec3 encodes Rfv3, a gene influencing neutralizing antibody control of retrovirus infection

Newly synthesized APOBEC3G is incorporated into HIV virions, inhibited by HIV RNA, and subsequently activated by RNase H

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scholarly article

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10.4049/JIMMUNOL.1001143

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2

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Mario L. Santiago

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2010-06-21T00:00:00Z

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44691296

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