Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury.
about
Transcriptional regulation by poly(ADP-ribose) polymerase-1 during T cell activation.Global analysis of transcriptional regulation by poly(ADP-ribose) polymerase-1 and poly(ADP-ribose) glycohydrolase in MCF-7 human breast cancer cellsMechanisms of vascular aging: new perspectives.Role of the mitochondrion in programmed necrosisNitrosative stress and pharmacological modulation of heart failure.Role of poly(ADP-ribose) polymerase 1 (PARP-1) in cardiovascular diseases: the therapeutic potential of PARP inhibitorsTranscriptional control by PARP-1: chromatin modulation, enhancer-binding, coregulation, and insulationTherapeutic applications of PARP inhibitors: anticancer therapy and beyondInflammation and endothelial dysfunction during aging: role of NF-kappaBPharmacological Strategies to Retard Cardiovascular AgingInhibition of the activity of poly (ADP-ribose) polymerase reduces heart ischaemia/reperfusion injury via suppressing JNK-mediated AIF translocation.PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegenerationMelatonin, clock genes and mitochondria in sepsis.Computational simulations to predict creatine kinase-associated factors: protein-protein interaction studies of brain and muscle types of creatine kinases.Activator protein-1 signalling pathway and apoptosis are modulated by poly(ADP-ribose) polymerase-1 in experimental colitis.Poly(ADP-ribose) polymerase-1 hyperactivation and impairment of mitochondrial respiratory chain complex I function in reperfused mouse hearts.PARP1 is required for adhesion molecule expression in atherogenesis.Olaparib protects cardiomyocytes against oxidative stress and improves graft contractility during the early phase after heart transplantation in rats.Role of oxidative and nitrosative stress, longevity genes and poly(ADP-ribose) polymerase in cardiovascular dysfunction associated with aging.Poly(ADP-ribose) polymerase 1 inhibition protects human aortic endothelial cells against LPS-induced inflammation response.
P2860
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P2860
Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury.
description
2003 nî lūn-bûn
@nan
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@ast
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@en
type
label
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@ast
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@en
prefLabel
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@ast
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@en
P2093
P2860
P1433
P1476
Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.
@en
P2093
Alvin Denenberg
Basilia Zingarelli
Michael O'Connor
Paul W Hake
P2860
P304
P356
10.2119/2003-00011.ZINGARELLI
P407
P577
2003-05-01T00:00:00Z