Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury. - Wikidata - awgldk - TriplyDB

Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury.

Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury.

http://www.wikidata.org/entity/Q34520738

about

Transcriptional regulation by poly(ADP-ribose) polymerase-1 during T cell activation.Global analysis of transcriptional regulation by poly(ADP-ribose) polymerase-1 and poly(ADP-ribose) glycohydrolase in MCF-7 human breast cancer cells Mechanisms of vascular aging: new perspectives.Role of the mitochondrion in programmed necrosis Nitrosative stress and pharmacological modulation of heart failure.Role of poly(ADP-ribose) polymerase 1 (PARP-1) in cardiovascular diseases: the therapeutic potential of PARP inhibitors Transcriptional control by PARP-1: chromatin modulation, enhancer-binding, coregulation, and insulation Therapeutic applications of PARP inhibitors: anticancer therapy and beyond Inflammation and endothelial dysfunction during aging: role of NF-kappaB Pharmacological Strategies to Retard Cardiovascular Aging Inhibition of the activity of poly (ADP-ribose) polymerase reduces heart ischaemia/reperfusion injury via suppressing JNK-mediated AIF translocation.PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegeneration Melatonin, clock genes and mitochondria in sepsis.Computational simulations to predict creatine kinase-associated factors: protein-protein interaction studies of brain and muscle types of creatine kinases.Activator protein-1 signalling pathway and apoptosis are modulated by poly(ADP-ribose) polymerase-1 in experimental colitis.Poly(ADP-ribose) polymerase-1 hyperactivation and impairment of mitochondrial respiratory chain complex I function in reperfused mouse hearts.PARP1 is required for adhesion molecule expression in atherogenesis.Olaparib protects cardiomyocytes against oxidative stress and improves graft contractility during the early phase after heart transplantation in rats.Role of oxidative and nitrosative stress, longevity genes and poly(ADP-ribose) polymerase in cardiovascular dysfunction associated with aging.Poly(ADP-ribose) polymerase 1 inhibition protects human aortic endothelial cells against LPS-induced inflammation response.

P2860

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P2860

Absence of poly(ADP-ribose)polymerase-1 alters nuclear factor-kappa B activation and gene expression of apoptosis regulators after reperfusion injury.

http://www.wikidata.org/entity/Q34520738

description

2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի մայիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

@ast

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

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type

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Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

@ast

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

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prefLabel

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

@ast

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

@en

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2003-00011.ZINGARELLI

P1433

Molecular Medicine

P1476

Absence of poly(ADP-ribose)pol ...... tors after reperfusion injury.

@en

P2093

Alvin Denenberg

http://www.w3.org/2001/XMLSchema#string

Basilia Zingarelli

http://www.w3.org/2001/XMLSchema#string

Michael O'Connor

http://www.w3.org/2001/XMLSchema#string

Paul W Hake

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Sue Kong

http://www.w3.org/2001/XMLSchema#string

P2860

ICE-LAP6, a novel member of the ICE/Ced-3 gene family, is activated by the cytotoxic T cell protease granzyme B

The I kappa B kinase (IKK) and NF-kappa B: key elements of proinflammatory signalling

BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

The enzymatic and DNA binding activity of PARP-1 are not required for NF-kappa B coactivator function

Interaction of Oct-1 and automodification domain of poly(ADP-ribose) synthetase

Poly(ADP-ribose) polymerase binds with transcription enhancer factor 1 to MCAT1 elements to regulate muscle-specific transcription

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

Poly(ADP-ribosyl)ation reactions in the regulation of nuclear functions

Poly (ADP-ribose) polymerase, nitric oxide and cell death.

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143-153

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scholarly article

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10.2119/2003-00011.ZINGARELLI

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2003-05-01T00:00:00Z

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14571322

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reperfusion injury

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1430828

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