A genetic basis for the variation in the vulnerability of cancer to DNA damage. - Wikidata - awgldk - TriplyDB

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

http://www.wikidata.org/entity/Q34523304

about

PI3K pathway mutations are associated with longer time to local progression after radioembolization of colorectal liver metastases.Non-homologous end-joining protein expression screen from radiosensitive cancer patients yields a novel DNA double strand break repair phenotype.Androgen receptor as a mediator and biomarker of radioresistance in triple-negative breast cancer.Radiogenomics and radiotherapy response modeling.p62/SQSTM1-Dr. Jekyll and Mr. Hyde that prevents oxidative stress but promotes liver cancer.The changing paradigm of tumour response to irradiation.A general mechanistic model enables predictions of the biological effectiveness of different qualities of radiation.Across the tree of life, radiation resistance is governed by antioxidant Mn2+, gauged by paramagnetic resonance.Molecular Signatures of Radiation Response in Breast Cancer: Towards Personalized Decision-Making in Radiation Treatment.RNF126 as a biomarker of a poor prognosis in invasive breast cancer and CHK1 inhibitor efficacy in breast cancer cells.Radiotherapy increases plasma levels of tumoral cell-free DNA in non-small cell lung cancer patients.Machine Learning and Radiogenomics: Lessons Learned and Future Directions.Scorpins in the DNA Damage Response.

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P2860

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

http://www.wikidata.org/entity/Q34523304

description

2016 nî lūn-bûn

@nan

2016 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

name

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

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A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@en

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@nl

type

label

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@ast

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@en

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@nl

prefLabel

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@ast

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@en

A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@nl

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Nature Communications

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A genetic basis for the variation in the vulnerability of cancer to DNA damage.

@en

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Alykhan Shamji

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Bradley E Pearson

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Brian D Yard

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Craig D Peacock

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Daniel P Raymond

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Drew J Adams

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Eui Kyu Chie

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Francisco Almeida

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Jaime H Cheah

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James Phillips

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P2860

Role of the androgen receptor in breast cancer and preclinical analysis of enzalutamide

Mechanisms of change in gene copy number

The mRNA surveillance protein hSMG-1 functions in genotoxic stress response pathways in mammalian cells

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

RAD21L, a novel cohesin subunit implicated in linking homologous chromosomes in mammalian meiosis

Phase II trial of bicalutamide in patients with androgen receptor-positive, estrogen receptor-negative metastatic Breast Cancer

Comprehensive molecular characterization of human colon and rectal cancer

Improved survival with vemurafenib in melanoma with BRAF V600E mutation

Autophagy suppresses tumorigenesis through elimination of p62

Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy

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scholarly article

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10.1038/NCOMMS11428

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7

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Nathan A Pennell

Stuart Schreiber

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2016-04-25T00:00:00Z

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301645265

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1017987629

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