Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV. - Wikidata - awgldk - TriplyDB

Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.

Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.

http://www.wikidata.org/entity/Q34538178

about

Disturbed T Cell Signaling and Altered Th17 and Regulatory T Cell Subsets in the Pathogenesis of Systemic Lupus Erythematosus Small molecules in the treatment of systemic lupus erythematosus Heat shock protein 90 inhibition by 17-DMAG lessens disease in the MRL/lpr mouse model of systemic lupus erythematosus Abnormalities of T cell signaling in systemic lupus erythematosus.KN-93, an inhibitor of calcium/calmodulin-dependent protein kinase IV, promotes generation and function of Foxp3⁺ regulatory T cells in MRL/lpr mice.Targeting lymphocyte signaling pathways as a therapeutic approach to systemic lupus erythematosus.T cells as a therapeutic target in SLE.The dysregulation of cytokine networks in systemic lupus erythematosus.Decrease of Functional Activated T and B Cells and Treatment of Glomerulonephitis in Lupus-Prone Mice Using a Natural Flavonoid Astilbin Cutting edge: Calcium/Calmodulin-dependent protein kinase type IV is essential for mesangial cell proliferation and lupus nephritis.T cell signaling abnormalities contribute to aberrant immune cell function and autoimmunity.Metabolic regulation of organelle homeostasis in lupus T cells Calcium/calmodulin-dependent protein kinase IV suppresses IL-2 production and regulatory T cell activity in lupus Cutting Edge: Nanogel-Based Delivery of an Inhibitor of CaMK4 to CD4+ T Cells Suppresses Experimental Autoimmune Encephalomyelitis and Lupus-like Disease in Mice.The cAMP response element modulator (CREM) regulates TH2 mediated inflammation Taming lupus-a new understanding of pathogenesis is leading to clinical advances.Activation of mTOR (mechanistic target of rapamycin) in rheumatic diseases.CaMK4-dependent activation of AKT/mTOR and CREM-α underlies autoimmunity-associated Th17 imbalance Treatment of systemic lupus erythematosus: new advances in targeted therapy.Treatment of systemic lupus erythematosus: new therapeutic avenues and blind alleys.A monoclonal antibody against CD86 and its protection in a murine lupus nephritis model of chronic graft-versus-host disease.Upcoming Therapeutic Targets in Cutaneous Lupus Erythematous.Empowering Regulatory T Cells in Autoimmunity.New insights into the immunopathogenesis of systemic lupus erythematosus.CREM Alpha Enhances IL-21 Production in T Cells In Vivo and In Vitro.Belimumab efficacy is 'mild' but market potential still great†: anticipating us approval of the first lupus drug since 1957.Calcium/Calmodulin-Dependent Kinase IV Facilitates the Recruitment of Interleukin-17-Producing Cells to Target Organs Through the CCR6/CCL20 Axis in Th17 Cell-Driven Inflammatory Diseases.The role of CaMK4 in immune responses.Calcium/Calmodulin-Dependent Protein Kinase IV (CaMKIV) Mediates Acute Skeletal Muscle Inflammatory Response.Jieduquyuziyin prescription suppresses IL-17 production and Th17 activity in MRL/lpr mice by inhibiting expression of Ca(2+)/calmodulin-dependent protein kinase-4.Lupus Nephritis IgG Induction of Calcium/Calmodulin-Dependent Protein Kinase IV Expression in Podocytes and Alteration of Their Function.Aberrant T Cell Signaling and Subsets in Systemic Lupus Erythematosus.Calcium/Calmodulin Kinase IV Controls the Function of Both T Cells and Kidney Resident Cells

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P2860

Suppression of autoimmunity and organ pathology in lupus-prone mice upon inhibition of calcium/calmodulin-dependent protein kinase type IV.

http://www.wikidata.org/entity/Q34538178

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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Suppression of autoimmunity an ...... endent protein kinase type IV.

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Arthritis and Rheumatism

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Suppression of autoimmunity an ...... pendent protein kinase type IV

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George C Tsokos

http://www.w3.org/2001/XMLSchema#string

José C Crispín

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Katalin Kis-Toth

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Yuang-Taung Juang

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P2860

Calmodulin-dependent kinase IV links Toll-like receptor 4 signaling with survival pathway of activated dendritic cells

Primer3 on the WWW for general users and for biologist programmers

Systemic lupus erythematosus serum IgG increases CREM binding to the IL-2 promoter and suppresses IL-2 production through CaMKIV

Pathogenesis of human systemic lupus erythematosus: recent advances.

Suppression of skin and kidney disease by inhibition of spleen tyrosine kinase in lupus-prone mice

Lupus nephritis: lessons from murine models

Expanded double negative T cells in patients with systemic lupus erythematosus produce IL-17 and infiltrate the kidneys

Studies on the role of tumor necrosis factor in murine and human autoimmunity.

Interferons as pathogenic effectors in autoimmunity.

HMGB1 release induced by liver ischemia involves Toll-like receptor 4 dependent reactive oxygen species production and calcium-mediated signaling

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523-529

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10.1002/ART.30085

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2

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63

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Kunihiro Ichinose

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2011-02-01T00:00:00Z

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47451415

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20954187

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3030625

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