IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling - Wikidata - awgldk - TriplyDB

IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling

IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling

http://www.wikidata.org/entity/Q34539832

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A Mouse Model of Zika Virus Pathogenesis Zika Virus Disrupts Phospho-TBK1 Localization and Mitosis in Human Neuroepithelial Stem Cells and Radial Glia Vaginal Exposure to Zika Virus during Pregnancy Leads to Fetal Brain Infection The Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific Manner The regulation of inflammation by interferons and their STATs Regulation of mitochondrial biogenesis and its intersection with inflammatory responses MAVS is essential for primary CD4+ T cell immunity but not for recall T cell responses following an attenuated West Nile virus infection RNA-Seq analysis of chikungunya virus infection and identification of granzyme A as a major promoter of arthritic inflammation Chemokine Receptor Ccr7 Restricts Fatal West Nile Virus Encephalitis Tissue tropisms, infection kinetics, histologic lesions, and antibody response of the MR766 strain of Zika virus in a murine model Potent inhibition of Junín virus infection by interferon in murine cells.Hantavirus interferon regulation and virulence determinants T-helper 17 cell cytokines and interferon type I: partners in crime in systemic lupus erythematosus?Oas1b-dependent Immune Transcriptional Profiles of West Nile Virus Infection in the Collaborative Cross.Type I interferon in the pathogenesis of lupus.Zika Virus Persistently Infects and Is Basolaterally Released from Primary Human Brain Microvascular Endothelial Cells.Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways.The TIR-domain containing adaptor TRAM is required for TLR7 mediated RANTES production.Viral pathogen-associated molecular patterns regulate blood-brain barrier integrity via competing innate cytokine signals.Interferon regulatory factor 5-dependent immune responses in the draining lymph node protect against West Nile virus infection.Age-dependent myeloid dendritic cell responses mediate resistance to la crosse virus-induced neurological disease.Investigation of the caspase-dependent mitochondrial apoptotic pathway in mononuclear cells of patients with systemic Lupus erythematosus.IKKβ is an IRF5 kinase that instigates inflammation Interferon regulatory factors: critical mediators of human lupus.Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.A temporal gate for viral enhancers to co-opt Toll-like-receptor transcriptional activation pathways upon acute infection Control of hepatitis C virus replication in mouse liver-derived cells by MAVS-dependent production of type I and type III interferons.Oropouche virus infection and pathogenesis are restricted by MAVS, IRF-3, IRF-7, and type I interferon signaling pathways in nonmyeloid cells.IFIT1 Differentially Interferes with Translation and Replication of Alphavirus Genomes and Promotes Induction of Type I Interferon Dengue Virus NS Proteins Inhibit RIG-I/MAVS Signaling by Blocking TBK1/IRF3 Phosphorylation: Dengue Virus Serotype 1 NS4A Is a Unique Interferon-Regulating Virulence Determinant.Selective Blockade of Interferon-α and -β Reveals Their Non-Redundant Functions in a Mouse Model of West Nile Virus Infection Functional Analysis of Chicken IRF7 in Response to dsRNA Analog Poly(I:C) by Integrating Overexpression and Knockdown Constitutively Active MAVS Inhibits HIV-1 Replication via Type I Interferon Secretion and Induction of HIV-1 Restriction Factors.The role of type I interferons in intestinal infection, homeostasis, and inflammation.Full Spectrum of LPS Activation in Alveolar Macrophages of Healthy Volunteers by Whole Transcriptomic Profiling.Amelioration of Japanese encephalitis by blockage of 4-1BB signaling is coupled to divergent enhancement of type I/II IFN responses and Ly-6C(hi) monocyte differentiation.The interferon regulatory factors as novel potential targets in the treatment of cardiovascular diseases.Interferon-Regulatory Factor 5-Dependent Signaling Restricts Orthobunyavirus Dissemination to the Central Nervous System Emerging roles of interferon-stimulated genes in the innate immune response to hepatitis C virus infection Pre-vaccination inflammation and B-cell signalling predict age-related hyporesponse to hepatitis B vaccination

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IRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signaling

http://www.wikidata.org/entity/Q34539832

description

2013 nî lūn-bûn

@nan

2013 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հունվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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JOURNAL.PPAT.1003118

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IRF-3, IRF-5, and IRF-7 coordi ...... s downstream of MAVS signaling

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Albert Huang

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Alissa Lancaster

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Ashlee V Moses

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Klaus Früh

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Larissa Thackray

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Lisa Clepper

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Margaret M Brassil

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Mehul S Suthar

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Sarah C Vick

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P2860

Type I interferon [corrected] gene induction by the interferon regulatory factor family of transcription factors

Characterization of the 2'-5'-oligoadenylate synthetase ubiquitin-like family

CHAC1/MGC4504 is a novel proapoptotic component of the unfolded protein response, downstream of the ATF4-ATF3-CHOP cascade

Mapping a dynamic innate immunity protein interaction network regulating type I interferon production

Regulation of innate antiviral defenses through a shared repressor domain in RIG-I and LGP2

Multiple regulatory domains of IRF-5 control activation, cellular localization, and induction of chemokines that mediate recruitment of T lymphocytes

Interferon response factors 3 and 7 protect against Chikungunya virus hemorrhagic fever and shock

A diverse range of gene products are effectors of the type I interferon antiviral response

The IRF family, revisited

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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e1003118

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10.1371/JOURNAL.PPAT.1003118

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1

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9

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Michael S. Diamond

Herbert W. "Skip" Virgin

Helen M. Lazear

Janko Nikolich-Žugich

Courtney Wilkins

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2013-01-03T00:00:00Z

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234090057

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23300459

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3536698

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