Inhibitory postsynaptic potentials in lumbar motoneurons remain depolarizing after neonatal spinal cord transection in the rat.
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Serotonergic modulation of post-synaptic inhibition and locomotor alternating pattern in the spinal cordNMR Based Cerebrum Metabonomic Analysis Reveals Simultaneous Interconnected Changes during Chick Embryo IncubationSensory feedback synchronizes motor and sensory neuronal networks in the neonatal rat spinal cordContribution of the potassium-chloride co-transporter KCC2 to the modulation of lumbar spinal networks in mice.Cation-chloride cotransporters in neuronal development, plasticity and diseasePrenatal exposure to fenugreek impairs sensorimotor development and the operation of spinal cord networks in miceDual personality of GABA/glycine-mediated depolarizations in immature spinal cord.Refuting the challenges of the developmental shift of polarity of GABA actions: GABA more exciting than ever!Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons.Activation-Dependent Rapid Postsynaptic Clustering of Glycine Receptors in Mature Spinal Cord Neurons.Timing of developmental sequences in different brain structures: physiological and pathological implications.Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury.Global gene expression analysis of rodent motor neurons following spinal cord injury associates molecular mechanisms with development of postinjury spasticity.Distribution of neuronal KCC2a and KCC2b isoforms in mouse CNS.Spinal cord injury-induced attenuation of GABAergic inhibition in spinal dorsal horn circuits is associated with down-regulation of the chloride transporter KCC2 in rat.NKCC1 cotransporter inactivation underlies embryonic development of chloride-mediated inhibition in mouse spinal motoneuron.Retracing your footsteps: developmental insights to spinal network plasticity following injury.Transmission within several spinal pathways in adults with cerebral palsy.Glucose is an adequate energy substrate for the depolarizing action of GABA and glycine in the neonatal rat spinal cord in vitro.[Calpain as a new therapeutic target for treating spasticity after a spinal cord injury].Changes in spinal inhibitory networks induced by furosemide in humans.Cystic fibrosis transmembrane conductance regulator modulates synaptic chloride homeostasis in motoneurons of the rat spinal cord during neonatal development.Metaplasticity within the spinal cord: Evidence brain-derived neurotrophic factor (BDNF), tumor necrosis factor (TNF), and alterations in GABA function (ionic plasticity) modulate pain and the capacity to learn.
P2860
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P2860
Inhibitory postsynaptic potentials in lumbar motoneurons remain depolarizing after neonatal spinal cord transection in the rat.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@ast
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@en
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@nl
type
label
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@ast
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@en
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@nl
prefLabel
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@ast
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@en
Inhibitory postsynaptic potent ...... l cord transection in the rat.
@nl
P2093
P2860
P356
P1476
Inhibitory postsynaptic potent ...... al cord transection in the rat
@en
P2093
Jean-François Pflieger
Laurent Vinay
Sylvie Liabeuf
P2860
P304
P356
10.1152/JN.00328.2006
P407
P577
2006-06-28T00:00:00Z