Replication licensing promotes cyclin D1 expression and G1 progression in untransformed human cells. - Wikidata - awgldk - TriplyDB

Replication licensing promotes cyclin D1 expression and G1 progression in untransformed human cells.

Replication licensing promotes cyclin D1 expression and G1 progression in untransformed human cells.

http://www.wikidata.org/entity/Q34547649

about

Gli1 protein regulates the S-phase checkpoint in tumor cells via Bid protein, and its inhibition sensitizes to DNA topoisomerase 1 inhibitors A reduction of licensed origins reveals strain-specific replication dynamics in mice Loss of DLG5 promotes breast cancer malignancy by inhibiting the Hippo signaling pathway.Stress-stimulated mitogen-activated protein kinases control the stability and activity of the Cdt1 DNA replication licensing factor Origin licensing and p53 status regulate Cdk2 activity during G(1).Key role of 15-LO/15-HETE in angiogenesis and functional recovery in later stages of post-stroke mice.Hypoxia activates 15-PGDH and its metabolite 15-KETE to promote pulmonary artery endothelial cells proliferation via ERK1/2 signalling.DNA damage response and tumorigenesis in Mcm2-deficient mice Highly stable loading of Mcm proteins onto chromatin in living cells requires replication to unload Mechanism of CRL4(Cdt2), a PCNA-dependent E3 ubiquitin ligase Mitotic UV irradiation induces a DNA replication-licensing defect that potentiates G1 arrest response.Behavior of replication origins in Eukaryota - spatio-temporal dynamics of licensing and firing Cell cycle stage-specific roles of Rad18 in tolerance and repair of oxidative DNA damage Divergent S phase checkpoint activation arising from prereplicative complex deficiency controls cell survival.Simvastatin suppresses the DNA replication licensing factor MCM7 and inhibits the growth of tamoxifen-resistant breast cancer cells.MCM7 promotes cancer progression through cyclin D1-dependent signaling and serves as a prognostic marker for patients with hepatocellular carcinoma.Simvastatin and Atorvastatin inhibit DNA replication licensing factor MCM7 and effectively suppress RB-deficient tumors growth.How dormant origins promote complete genome replication.Dormant origins, the licensing checkpoint, and the response to replicative stresses The contribution of dormant origins to genome stability: from cell biology to human genetics.Prohibitin physically interacts with MCM proteins and inhibits mammalian DNA replication.The High-Affinity Interaction between ORC and DNA that Is Required for Replication Licensing Is Inhibited by 2-Arylquinolin-4-Amines.The licensing checkpoint opens up.Oncogene-Induced Replication Stress Drives Genome Instability and Tumorigenesis.The Temporal Regulation of S Phase Proteins During G1.Lgr5+ intestinal stem cells reside in an unlicensed G1 phase.

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P2860

Replication licensing promotes cyclin D1 expression and G1 progression in untransformed human cells.

http://www.wikidata.org/entity/Q34547649

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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type

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Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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Replication licensing promotes ...... in untransformed human cells.

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P1433

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Replication licensing promotes ...... n in untransformed human cells

@en

P2093

Cyrus Vaziri

http://www.w3.org/2001/XMLSchema#string

Damien M Slater

http://www.w3.org/2001/XMLSchema#string

Kathleen Nevis

http://www.w3.org/2001/XMLSchema#string

Marc Lenburg

http://www.w3.org/2001/XMLSchema#string

Peijun Liu

http://www.w3.org/2001/XMLSchema#string

P2860

Ser727-dependent recruitment of MCM5 by Stat1alpha in IFN-gamma-induced transcriptional activation.

MCM proteins are associated with RNA polymerase II holoenzyme

Binding of cyclin-dependent kinases to ORC and Cdc6p regulates the chromosome replication cycle

The consensus motif for phosphorylation by cyclin D1-Cdk4 is different from that for phosphorylation by cyclin A/E-Cdk2

Chk2/hCds1 functions as a DNA damage checkpoint in G(1) by stabilizing p53

Identification of two residues in MCM5 critical for the assembly of MCM complexes and Stat1-mediated transcription activation in response to IFN-gamma

Evidence for a role of MCM (mini-chromosome maintenance)5 in transcriptional repression of sub-telomeric and Ty-proximal genes in Saccharomyces cerevisiae.

GINS maintains association of Cdc45 with MCM in replisome progression complexes at eukaryotic DNA replication forks.

Mcm7, a subunit of the presumptive MCM helicase, modulates its own expression in conjunction with Mcm1.

DNA replication in eukaryotic cells

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125-136

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scholarly article

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10.4161/CC.8.1.7528

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1

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8

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Jeanette Gowen Cook

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2009-01-01T00:00:00Z

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P5875

23688287

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19106611

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3032797

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