Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE. - Wikidata - awgldk - TriplyDB

Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.

Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.

http://www.wikidata.org/entity/Q34552395

about

Astrocytes differentially respond to inflammatory autoimmune insults and imbalances of neural activity.Inhibition of microglia activation as a phenotypic assay in early drug discovery A Chronic Longitudinal Characterization of Neurobehavioral and Neuropathological Cognitive Impairment in a Mouse Model of Gulf War Agent Exposure.Retinal ganglion cell loss is accompanied by antibody depositions and increased levels of microglia after immunization with retinal antigens Inflammation-mediated memory dysfunction and effects of a ketogenic diet in a murine model of multiple sclerosis Estrogen receptor β ligand therapy activates PI3K/Akt/mTOR signaling in oligodendrocytes and promotes remyelination in a mouse model of multiple sclerosis.Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.Inhibition of glutamate carboxypeptidase II (GCPII) activity as a treatment for cognitive impairment in multiple sclerosis.Bringing CLARITY to gray matter atrophy.Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.Reduction in parvalbumin-positive interneurons and inhibitory input in the cortex of mice with experimental autoimmune encephalomyelitis Inflammation subverts hippocampal synaptic plasticity in experimental multiple sclerosis.Axonal degeneration in multiple sclerosis: can we predict and prevent permanent disability?Smaller cornu ammonis 2-3/dentate gyrus volumes and elevated cortisol in multiple sclerosis patients with depressive symptoms.Interleukin-1β and interleukin-1 receptor antagonist appear in grey matter additionally to white matter lesions during experimental multiple sclerosis Estriol preserves synaptic transmission in the hippocampus during autoimmune demyelinating disease.Astrocyte CCL2 sustains immune cell infiltration in chronic experimental autoimmune encephalomyelitis.Microglia in the normally aged hippocampus.Cortical atrophy in experimental autoimmune encephalomyelitis: in vivo imaging Oral fingolimod rescues the functional deficits of synapses in experimental autoimmune encephalomyelitis.Automated measurement of nerve fiber density using line intensity scan analysis.Inhibition of Vascular Endothelial Growth Factor Receptor 2 Exacerbates Loss of Lower Motor Neurons and Axons during Experimental Autoimmune Encephalomyelitis.LINGO-1 antibody ameliorates myelin impairment and spatial memory deficits in experimental autoimmune encephalomyelitis mice Astrocyte matricellular proteins that control excitatory synaptogenesis are regulated by inflammatory cytokines and correlate with paralysis severity during experimental autoimmune encephalomyelitis.Saffron ethanolic extract attenuates oxidative stress, spatial learning, and memory impairments induced by local injection of ethidium bromide.Platelet-Activating Factor Receptors Mediate Excitatory Postsynaptic Hippocampal Injury in Experimental Autoimmune Encephalomyelitis Therapeutic testosterone administration preserves excitatory synaptic transmission in the hippocampus during autoimmune demyelinating disease Abnormal NMDA receptor function exacerbates experimental autoimmune encephalomyelitis Cognitive impairment in multiple sclerosis: a forgotten disability remembered.Vitamin D3 attenuates oxidative stress and cognitive deficits in a model of toxic demyelination Saturable leptin transport across the BBB persists in EAE mice Detection of altered hippocampal morphology in multiple sclerosis-associated depression using automated surface mesh modeling.Activation of Wnt signaling promotes hippocampal neurogenesis in experimental autoimmune encephalomyelitis.Synaptic plasticity in multiple sclerosis and in experimental autoimmune encephalomyelitis.Brain atrophy correlates with functional outcome in a murine model of multiple sclerosis.Therapeutic laquinimod treatment decreases inflammation, initiates axon remyelination, and improves motor deficit in a mouse model of multiple sclerosis.An animal model of cortical and callosal pathology in multiple sclerosis.Matrix metalloproteinase activity stimulates N-cadherin shedding and the soluble N-cadherin ectodomain promotes classical microglial activation Microglial activation and chronic neurodegeneration.Olfactory dysfunction: its early temporal relationship and neural correlates in the pathogenesis of Alzheimer's disease.

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Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.

http://www.wikidata.org/entity/Q34552395

description

2010 nî lūn-bûn

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2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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type

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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LABINVEST.2010.6

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Laboratory Investigation

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Hippocampal CA1 atrophy and sy ...... immune encephalomyelitis, EAE.

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Andrea A Avedisian

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Marina O Ziehn

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Rhonda R Voskuhl

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Seema Tiwari-Woodruff

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P2860

The blood-brain barrier in health and chronic neurodegenerative disorders

Grey matter pathology in multiple sclerosis.

Evidence of axonal damage in the early stages of multiple sclerosis and its relevance to disability.

Evidence for widespread axonal damage at the earliest clinical stage of multiple sclerosis.

Selective dysfunction of hippocampal CA1 astrocytes contributes to delayed neuronal damage after transient forebrain ischemia.

Dendritic and synaptic pathology in experimental autoimmune encephalomyelitis.

Paradox of Ca2+ signaling, cell death and stroke.

Cognitive impairment and neurophysiological correlates in MS.

Immunopathogenesis and immunotherapy of multiple sclerosis.

Neuropsychology of multiple sclerosis.

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labinvest.2010.6

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10.1038/LABINVEST.2010.6

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5

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1015684602

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experimental autoimmune encephalomyelitis

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3033772

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