Epstein-Barr virus immediate-early protein BZLF1 inhibits tumor necrosis factor alpha-induced signaling and apoptosis by downregulating tumor necrosis factor receptor 1
about
Modulation of tumor necrosis factor by microbial pathogens.The C-mer gene is induced by Epstein-Barr virus immediate-early protein BRLF1Modulation of p53 activity by IkappaBalpha: evidence suggesting a common phylogeny between NF-kappaB and p53 transcription factors.Subversion of cytokine networks by virally encoded decoy receptorsEpstein-Barr virus WZhet DNA can induce lytic replication in epithelial cells in vitro, although WZhet is not detectable in many human tissues in vivo.Kaposi's sarcoma-associated herpesvirus K-bZIP represses gene transcription via SUMO modification.Interferon regulatory factor 7 is negatively regulated by the Epstein-Barr virus immediate-early gene, BZLF-1Epstein-Barr virus evades CD4+ T cell responses in lytic cycle through BZLF1-mediated downregulation of CD74 and the cooperation of vBcl-2.The Epstein-Barr virus BZLF1 protein inhibits tumor necrosis factor receptor 1 expression through effects on cellular C/EBP proteinsAdenovirus RIDalphabeta complex inhibits lipopolysaccharide signaling without altering TLR4 cell surface expression.Amino acids in the basic domain of Epstein-Barr virus ZEBRA protein play distinct roles in DNA binding, activation of early lytic gene expression, and promotion of viral DNA replication.The human papillomavirus 16 E6 protein can either protect or further sensitize cells to TNF: effect of doseAnalysis of an ankyrin-like region in Epstein Barr Virus encoded (EBV) BZLF-1 (ZEBRA) protein: implications for interactions with NF-κB and p53.Host genetics of Epstein-Barr virus infection, latency and disease.Epstein-Barr virus (EBV) latent membrane protein-1 down-regulates tumor necrosis factor-alpha (TNF-alpha) receptor-1 and confers resistance to TNF-alpha-induced apoptosis in T cells: implication for the progression to T-cell lymphoma in EBV-associatThe Epstein-Barr Virus Lytic Protein BZLF1 as a Candidate Target Antigen for Vaccine Development.Epstein-Barr virus transcription activator Rta upregulates decoy receptor 3 expression by binding to its promoterAn Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral ReplicationSalivary and serum IL-10, TNF-α, TGF-β, VEGF levels in oropharyngeal squamous cell carcinoma and correlation with HPV and EBV infections.CCAAT/enhancer binding proteins alpha and beta regulate the tumor necrosis factor receptor 1 gene promoter.Cross-regulation between herpesviruses and the TNF superfamily membersReduced display of tumor necrosis factor receptor I at the host cell surface supports infection with Chlamydia trachomatis.Tumour necrosis factor receptors and apoptosis of alveolar macrophages during early infection with attenuated and virulent Mycobacterium bovis.Cellular immediate-early gene expression occurs kinetically upstream of Epstein-Barr virus bzlf1 and brlf1 following cross-linking of the B cell antigen receptor in the Akata Burkitt lymphoma cell line.Construction and expression of a spliced variant of Epstein-Barr virus bzlf1 and preparation of its polyclonal antibody.Functional interaction between Epstein-Barr virus replication protein Zta and host DNA damage response protein 53BP1.Mutations of amino acids in the DNA-recognition domain of Epstein-Barr virus ZEBRA protein alter its sub-nuclear localization and affect formation of replication compartments.Comparative genomic analysis of three white spot syndrome virus isolates of different virulence.Virus Infection and Death Receptor-Mediated Apoptosis.Herpes simplex virus type 1 infection activates the Epstein-Barr virus replicative cycle via a CREB-dependent mechanism.Decoy receptor 3, upregulated by Epstein-Barr virus latent membrane protein 1, enhances nasopharyngeal carcinoma cell migration and invasion.The Immunomodulatory Capacity of an Epstein-Barr Virus Abortive Lytic Cycle: Potential Contribution to Viral Tumorigenesis.Therapies based on targeting Epstein-Barr virus lytic replication for EBV-associated malignancies.Investigating genetic-and-epigenetic networks, and the cellular mechanisms occurring in Epstein-Barr virus-infected human B lymphocytes via big data mining and genome-wide two-sided NGS data identification
P2860
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P2860
Epstein-Barr virus immediate-early protein BZLF1 inhibits tumor necrosis factor alpha-induced signaling and apoptosis by downregulating tumor necrosis factor receptor 1
description
2004 nî lūn-bûn
@nan
2004 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@ast
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@en
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@nl
type
label
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@ast
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@en
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@nl
prefLabel
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@ast
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@en
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@nl
P2093
P2860
P1433
P1476
Epstein-Barr virus immediate-e ...... mor necrosis factor receptor 1
@en
P2093
Amy Mauser
Shannon C Kenney
Thomas E Morrison
P2860
P304
P356
10.1128/JVI.78.1.544-549.2004
P407
P577
2004-01-01T00:00:00Z