Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
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Cancer incidence in the population exposed to dioxin after the "Seveso accident": twenty years of follow-upTCDD and cancer: a critical review of epidemiologic studiesEnvironmental Ligands of the Aryl Hydrocarbon Receptor and Their Effects in Models of Adult Liver Progenitor CellsMammalian models of chemically induced primary malignancies exploitable for imaging-based preclinical theragnostic researchEffect of 2,3,7,8-tetrachlorodibenzo-p-dioxin administration and high-fat diet on the body weight and hepatic estrogen metabolism in female C3H/HeN miceNrf2 protects against 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced oxidative injury and steatohepatitisDynamic zebrafish interactome reveals transcriptional mechanisms of dioxin toxicitySU5416, a VEGF receptor inhibitor and ligand of the AHR, represents a new alternative for immunomodulationDevelopment of a convenient in vivo hepatotoxin assay using a transgenic zebrafish line with liver-specific DsRed expressionTransgenic rat models for mutagenesis and carcinogenesisInhibition of UV-C light-induced apoptosis in liver cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin.TCDD promotes lung tumors via attenuation of apoptosis through activation of the Akt and ERK1/2 signaling pathways.Dioxin and estrogen signaling in lung adenocarcinoma cells with different aryl hydrocarbon receptor/estrogen receptor α phenotypes.The environmental toxicant 2,3,7,8-tetrachlorodibenzo-p-dioxin disturbs the establishment and maintenance of cell polarity in preimplantation rat embryos.The activated aryl hydrocarbon receptor synergizes mitogen-induced murine liver hyperplasiaFungicide prochloraz and environmental pollutant dioxin induce the ABCG2 transporter in bovine mammary epithelial cells by the arylhydrocarbon receptor signaling pathway.Xenobiotic metabolism, disposition, and regulation by receptors: from biochemical phenomenon to predictors of major toxicities2,3,7,8-Tetrachlorodibenzo-p-dioxin promotes BHV-1 infection in mammalian cells by interfering with iron homeostasis regulationIn utero exposure to TCDD alters Wnt signaling during mouse prostate development: linking ventral prostate agenesis to downregulated β-catenin signalingResveratrol prevents epigenetic silencing of BRCA-1 by the aromatic hydrocarbon receptor in human breast cancer cellsAryl hydrocarbon receptor ligands in cancer: friend and foe.The AhR Ligand, TCDD, Regulates Androgen Receptor Activity Differently in Androgen-Sensitive versus Castration-Resistant Human Prostate Cancer Cells.The mammalian circadian system is resistant to dioxinRole of the Aryl Hydrocarbon Receptor in Colon Neoplasia.Polycyclic aromatic hydrocarbons (PAHs) mediate transcriptional activation of the ATP binding cassette transporter ABCB6 gene via the aryl hydrocarbon receptor (AhR).Ameliorative effect of supplementation with L-glutamine on oxidative stress, DNA damage, cell viability and hepatotoxicity induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in rat hepatocyte cultures.Altering HIF-1α through 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure affects coronary vessel developmentTreatment of mice with the Ah receptor agonist and human carcinogen dioxin results in altered numbers and function of hematopoietic stem cells.Role of the aryl hydrocarbon receptor in carcinogenesis and potential as a drug target.Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME.Activation of the aryl hydrocarbon receptor by TCDD inhibits senescence: a tumor promoting event?Aryl hydrocarbon receptor and lung cancer.The role of oxidative stress in carcinogenesis induced by metals and xenobiotics.Comparative analysis of AhR-mediated TCDD-elicited gene expression in human liver adult stem cells.Gap junctional intercellular communication as a target for liver toxicity and carcinogenicity.Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis-persistent pollutants affect programmed cell death.Role of the arylhydrocarbon receptor in lung disease.Environmental factors in causing human cancers: emphasis on tumorigenesis.Mode of action and dose-response framework analysis for receptor-mediated toxicity: The aryl hydrocarbon receptor as a case study.Endocrine-disrupting chemicals and skin manifestations.
P2860
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P2860
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
description
2006 nî lūn-bûn
@nan
2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@ast
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@en
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@nl
type
label
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@ast
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@en
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@nl
prefLabel
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@ast
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@en
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@nl
P356
P1476
Carcinogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in experimental models.
@en
P2093
Dieter Schrenk
Stefanie Knerr
P304
P356
10.1002/MNFR.200600006
P577
2006-10-01T00:00:00Z