NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats - Wikidata - awgldk - TriplyDB

NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats

NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats

http://www.wikidata.org/entity/Q34570408

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Peripheral chemoreception and arterial pressure responses to intermittent hypoxia Hypoxia-inducible factors and hypertension: lessons from sleep apnea syndrome Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxia Hypoxia-inducible factor 2α (HIF-2α) heterozygous-null mice exhibit exaggerated carotid body sensitivity to hypoxia, breathing instability, and hypertension Intermittent hypoxia augments acute hypoxic sensing via HIF-mediated ROS Enhanced neuropeptide Y synthesis during intermittent hypoxia in the rat adrenal medulla: role of reactive oxygen species-dependent alterations in precursor peptide processing Reactive oxygen species and the brain in sleep apnea.Hypoxia. 4. Hypoxia and ion channel function Protecting against vascular disease in brain Xanthine oxidase mediates hypoxia-inducible factor-2α degradation by intermittent hypoxia.Chronic intermittent hypoxia increases rat sternohyoid muscle NADPH oxidase expression with attendant modest oxidative stress.HIF-1α activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidase Sensory plasticity of the carotid body: role of reactive oxygen species and physiological significance.Relationship between expression of NADPH oxidase 2 and invasion and prognosis of human gastric cancer Reactive oxygen species are involved in BMP-induced dendritic growth in cultured rat sympathetic neurons.Commitment to glycolysis sustains survival of NO-producing inflammatory dendritic cells.Sympatho-adrenal activation by chronic intermittent hypoxia.Making sense of oxidative stress in obstructive sleep apnea: mediator or distracter?CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia.Manganese superoxide dismutase depletion in murine hematopoietic stem cells perturbs iron homeostasis, globin switching, and epigenetic control in erythrocyte precursor cells.Neural regulation of hypoxia-inducible factors and redox state drives the pathogenesis of hypertension in a rodent model of sleep apnea Calpain activation by ROS mediates human ether-a-go-go-related gene protein degradation by intermittent hypoxia.The role of hypoxia-inducible factors in oxygen sensing by the carotid body Intermittent hypoxia from obstructive sleep apnea may cause neuronal impairment and dysfunction in central nervous system: the potential roles played by microglia.Impairment of pancreatic β-cell function by chronic intermittent hypoxia Endothelin-1 mediates attenuated carotid baroreceptor activity by intermittent hypoxia.Peripheral chemoreceptors: function and plasticity of the carotid body.Sensing hypoxia: physiology, genetics and epigenetics.Understanding the biology of reactive oxygen species and their link to cancer: NADPH oxidases as novel pharmacological targets.Protein Thiol Redox Signaling in Monocytes and Macrophages.Carotid body chemoreflex: a driver of autonomic abnormalities in sleep apnoea.Mitochondrial-targeted nitroxides disrupt mitochondrial architecture and inhibit expression of peroxiredoxin 3 and FOXM1 in malignant mesothelioma cells.C/EBP homologous binding protein (CHOP) underlies neural injury in sleep apnea model.Moderate inhibition of mitochondrial function augments carotid body hypoxic sensitivity.Targeting mitochondrial reactive oxygen species to modulate hypoxia-induced pulmonary hypertension.NADPH oxidase 4 modulates hepatic responses to lipopolysaccharide mediated by Toll-like receptor-4.DNA methylation in the central and efferent limbs of the chemo reflex requires carotid body neural activity.The role of mitochondrial ROS in the aging brain.PKA activity exacerbates hypoxia-induced ROS formation and hypoxic injury in PC-12 cells.Complement-induced activation of the cardiac NLRP3 inflammasome in sepsis.

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NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats

http://www.wikidata.org/entity/Q34570408

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

NADPH oxidase 2 mediates inter ...... blood pressure changes in rats

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NADPH oxidase 2 mediates inter ...... blood pressure changes in rats

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NADPH oxidase 2 mediates inter ...... blood pressure changes in rats

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NADPH oxidase 2 mediates inter ...... blood pressure changes in rats

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Antioxidants & Redox Signaling

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NADPH oxidase 2 mediates inter ...... blood pressure changes in rats

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Balaraman Kalyanaraman

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Brian Kinsman

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Ganesh K Kumar

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Guoxiang Yuan

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Jayasri Nanduri

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Joy Joseph

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Nanduri R Prabhakar

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Shakil A Khan

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P2860

How mitochondria produce reactive oxygen species

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Targeting and regulation of reactive oxygen species generation by Nox family NADPH oxidases.

The mitochondrial production of reactive oxygen species: mechanisms and implications in human pathology.

Superoxide radical and iron modulate aconitase activity in mammalian cells.

Glutaredoxin 2 catalyzes the reversible oxidation and glutathionylation of mitochondrial membrane thiol proteins: implications for mitochondrial redox regulation and antioxidant DEFENSE.

Thiol oxidation and loss of mitochondrial complex I precede excitatory amino acid-mediated neurodegeneration.

Association of sleep-disordered breathing, sleep apnea, and hypertension in a large community-based study. Sleep Heart Health Study.

S-nitrosation of mitochondrial complex I depends on its structural conformation.

Induction of sensory long-term facilitation in the carotid body by intermittent hypoxia: implications for recurrent apneas

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10.1089/ARS.2010.3213

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14

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20618070

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3038125

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