Protein kinase C and the development of diabetic vascular complications. - Wikidata - awgldk - TriplyDB

Protein kinase C and the development of diabetic vascular complications.

Protein kinase C and the development of diabetic vascular complications.

http://www.wikidata.org/entity/Q34570516

about

A vascular gene trap screen defines RasGRP3 as an angiogenesis-regulated gene required for the endothelial response to phorbol esters Sensitization and translocation of TRPV1 by insulin and IGF-I.The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction Hyperglycemia promotes oxidative stress through inhibition of thioredoxin function by thioredoxin-interacting protein Presence and Risk Factors for Glaucoma in Patients with Diabetes.Medical treatment of diabetic retinopathy with somatostatin analogues.HIV replication enhances production of free fatty acids, low density lipoproteins and many key proteins involved in lipid metabolism: a proteomics study Role of calcium-independent phospholipase A2beta in high glucose-induced activation of RhoA, Rho kinase, and CPI-17 in cultured vascular smooth muscle cells and vascular smooth muscle hypercontractility in diabetic animals.Activation of protein kinase C isoforms and its impact on diabetic complications.Structural basis of protein kinase C isoform function PPARγ-induced cardiolipotoxicity in mice is ameliorated by PPARα deficiency despite increases in fatty acid oxidation.The maillard reaction in eye diseases.Total synthesis of bryostatin 1 The bryostatin 1 A-ring acetate is not the critical determinant for antagonism of phorbol ester-induced biological responses.Oxygen free radicals and the penis.Selective PKC beta inhibition with ruboxistaurin and endothelial function in type-2 diabetes mellitus Vascular complications and gene therapy.Quality of life and objective measures of diabetic neuropathy in a prospective placebo-controlled trial of ruboxistaurin and topiramate.Decoding the oxidative stress hypothesis in diabetic embryopathy through proapoptotic kinase signaling.Biochemical pathways for microvascular complications of diabetes mellitus.Pathophysiological role and therapeutic implications of inflammation in diabetic nephropathy Early diabetic neuropathy: triggers and mechanisms.Retrospective data suggests that the higher prevalence of benign paroxysmal positional vertigo in individuals with type 2 diabetes is mediated by hypertension.Ruboxistaurin, a protein kinase C beta inhibitor, as an emerging treatment for diabetes microvascular complications.Phosphorylation of pleckstrin increases proinflammatory cytokine secretion by mononuclear phagocytes in diabetes mellitus.Exercise-induced cardiac performance in autoimmune (type 1) diabetes is associated with a decrease in myocardial diacylglycerol.Experimental mechanisms of diabetic embryopathy and strategies for developing therapeutic interventions.Coenzyme Q10 Attenuates High Glucose-Induced Endothelial Progenitor Cell Dysfunction through AMP-Activated Protein Kinase Pathways.Skin microvascular endothelial dysfunction is associated with type 2 diabetes independently of microalbuminuria and arterial stiffness.Influence of TRPV1 on diabetes-induced alterations in thermal pain sensitivity.Protein kinase C beta inhibition: A novel therapeutic strategy for diabetic microangiopathy.Impact of glucose levels on advanced glycation end products in hemodialysis.Protective effect of short-term genistein supplementation on the early stage in diabetes-induced renal damage.Hyperglycaemia and cardiovascular disease.Tracing the footsteps of glomerular insulin signaling in diabetic kidney disease.Targeting the retinal microcirculation to treat diabetic sight problems.Dietary supplementation with vitamin E ameliorates cardiac failure in type I diabetic cardiomyopathy by suppressing myocardial generation of 8-iso-prostaglandin F2alpha and oxidized glutathione Traditional chinese medicine tang-luo-ning ameliorates sciatic nerve injuries in streptozotocin-induced diabetic rats.Relationship of hemoglobin A1C and mortality in heart failure patients with diabetes.Proteomic characterization of HIV-modulated membrane receptors, kinases and signaling proteins involved in novel angiogenic pathways.

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P2860

Protein kinase C and the development of diabetic vascular complications.

http://www.wikidata.org/entity/Q34570516

description

2001 nî lūn-bûn

@nan

2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2001年の論文

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2001年論文

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2001年論文

@zh-hant

2001年論文

@zh-hk

2001年論文

@zh-mo

2001年論文

@zh-tw

2001年论文

@wuu

name

Protein kinase C and the development of diabetic vascular complications.

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Protein kinase C and the development of diabetic vascular complications.

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Protein kinase C and the development of diabetic vascular complications.

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type

label

Protein kinase C and the development of diabetic vascular complications.

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Protein kinase C and the development of diabetic vascular complications.

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Protein kinase C and the development of diabetic vascular complications.

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prefLabel

Protein kinase C and the development of diabetic vascular complications.

@ast

Protein kinase C and the development of diabetic vascular complications.

@en

Protein kinase C and the development of diabetic vascular complications.

@nl

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J.0742-3071.2001.00638.X

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Diabetic Medicine

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Protein kinase C and the development of diabetic vascular complications.

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Katai N

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King GL

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Way KJ

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P2860

The extended protein kinase C superfamily

Characterization of vascular endothelial growth factor's effect on the activation of protein kinase C, its isoforms, and endothelial cell growth

Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Effects of ramipril on cardiovascular and microvascular outcomes in people with diabetes mellitus: results of the HOPE study and MICRO-HOPE substudy. Heart Outcomes Prevention Evaluation Study Investigators

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation

Localization of protein kinase C isozymes in cardiac myocytes

The c-Fos protein interacts with c-Jun/AP-1 to stimulate transcription of AP-1 responsive genes

Suppression subtractive hybridization identifies high glucose levels as a stimulus for expression of connective tissue growth factor and other genes in human mesangial cells

The molecular heterogeneity of protein kinase C and its implications for cellular regulation

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