The key involvement of estrogen receptor β and G-protein-coupled receptor 30 in the neuroprotective action of daidzein.
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Estrogen biology: new insights into GPER function and clinical opportunitiesDibutyl Phthalate (DBP)-Induced Apoptosis and Neurotoxicity are Mediated via ...Cognitive Effects of Soy Isoflavones in Patients with Alzheimer's Disease.Concentration-dependent opposite effects of 1-benzyl-1,2,3,4-tetrahydroisoquinoline on markers of apoptosis: in vitro and ex vivo studies.Salsolinol, an endogenous compound triggers a two-phase opposing action in the central nervous system.Lithospermic acid attenuates 1-methyl-4-phenylpyridine-induced neurotoxicity by blocking neuronal apoptotic and neuroinflammatory pathways.International Union of Basic and Clinical Pharmacology. XCVII. G Protein-Coupled Estrogen Receptor and Its Pharmacologic ModulatorsThe Crucial Involvement of Retinoid X Receptors in DDE Neurotoxicity.Molecular mechanisms underlying the memory-enhancing effects of estradiol.Estrogen Receptor Beta and 2-arachidonoylglycerol Mediate the Suppressive Effects of Estradiol on Frequency of Postsynaptic Currents in Gonadotropin-Releasing Hormone Neurons of Metestrous Mice: An Acute Slice Electrophysiological Study.Risks and benefits related to alimentary exposure to xenoestrogens.Oxabicycloheptene Sulfonate Protects Against β-Amyloid-induced Toxicity by Activation of PI3K/Akt and ERK Signaling Pathways Via GPER1 in C6 Cells.Apoptosis Induced by the UV Filter Benzophenone-3 in Mouse Neuronal Cells Is Mediated via Attenuation of Erα/Pparγ and Stimulation of Erβ/Gpr30 Signaling.Low dietary soy isoflavonoids increase hippocampal spine synapse density in ovariectomized rats.GPER modulators: Opportunity Nox on the heels of a class Akt.Selective Aryl Hydrocarbon Receptor Modulator 3,3'-Diindolylmethane Impairs AhR and ARNT Signaling and Protects Mouse Neuronal Cells Against Hypoxia.Estrogen receptors and estetrol-dependent neuroprotective actions: a pilot study.Steroid and Xenobiotic Receptor Signalling in Apoptosis and Autophagy of the Nervous System.Physiological and brain alterations produced by high-fat diet in male and female rats can be modulated by increased levels of estradiol during critical periods of development.Benzophenone-3 Impairs Autophagy, Alters Epigenetic Status, and Disrupts Retinoid X Receptor Signaling in Apoptotic Neuronal Cells.Exposure to increased levels of estradiol during development can have long-term effects on the response to undernutrition in female rats.Estradiol Increases Glutamate and GABA Neurotransmission into GnRH Neurons via Retrograde NO-Signaling in Proestrous Mice during the Positive Estradiol Feedback Period
P2860
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P2860
The key involvement of estrogen receptor β and G-protein-coupled receptor 30 in the neuroprotective action of daidzein.
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
The key involvement of estroge ...... protective action of daidzein.
@en
The key involvement of estroge ...... protective action of daidzein.
@nl
type
label
The key involvement of estroge ...... protective action of daidzein.
@en
The key involvement of estroge ...... protective action of daidzein.
@nl
prefLabel
The key involvement of estroge ...... protective action of daidzein.
@en
The key involvement of estroge ...... protective action of daidzein.
@nl
P2093
P1433
P1476
The key involvement of estroge ...... protective action of daidzein.
@en
P2093
A K Wojtowicz
J Rzemieniec
M Lenartowicz
W Krzeptowski
P304
P356
10.1016/J.NEUROSCIENCE.2013.02.005
P407
P577
2013-02-16T00:00:00Z