Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury.
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Understanding the NG2 Glial Scar after Spinal Cord InjuryNG2+ progenitors derived from embryonic stem cells penetrate glial scar and promote axonal outgrowth into white matter after spinal cord injuryA surviving intact branch stabilizes remaining axon architecture after injury as revealed by in vivo imaging in the mouse spinal cord.NG2-glia and their functions in the central nervous system.Toll-Like Receptors and Dectin-1, a C-Type Lectin Receptor, Trigger Divergent Functions in CNS Macrophages.Disrupting protein tyrosine phosphatase σ does not prevent sympathetic axonal dieback following myocardial infarction.Signaling pathways regulating neuron-glia interaction and their implications in Alzheimer's disease.The reactions and role of NG2 glia in spinal cord injury.Temporal and Regional Expression of Glucose-Dependent Insulinotropic Peptide and Its Receptor in Spinal Cord Injured Rats.STAT3 and SOCS3 regulate NG2 cell proliferation and differentiation after contusive spinal cord injury.The Role of the Oligodendrocyte Lineage in Acute Brain Trauma."Targeting astrocytes in CNS injury and disease: A translational research approach".Cell biology of spinal cord injury and repair.Synapsing with NG2 cells (polydendrocytes), unappreciated barrier to axon regeneration?Extrinsic and intrinsic regulation of axon regeneration at a crossroads.Neurite Outgrowth Assay.Proliferating NG2 cell-dependent angiogenesis and scar formation alter axon growth and functional recovery after spinal cord injury in mice.Disruption of perineuronal nets increases the frequency of sharp wave ripple events.Maximizing functional axon repair in the injured central nervous system: Lessons from neuronal development.Can injured adult CNS axons regenerate by recapitulating development?The diversity and disparity of the glial scar.Retinoic acid synthesis by NG2 expressing cells promotes a permissive environment for axonal outgrowth.Lack of NG2 exacerbates neurological outcome and modulates glial responses after traumatic brain injury.The Biology of Regeneration Failure and Success After Spinal Cord Injury.Locomotor recovery following contusive spinal cord injury does not require oligodendrocyte remyelination
P2860
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P2860
Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury.
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2014 nî lūn-bûn
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2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
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2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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Entrapment via synaptic-like c ...... lure after spinal cord injury.
@ast
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@en
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@nl
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label
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@ast
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@en
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@nl
prefLabel
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@ast
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@en
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@nl
P2093
P2860
P1476
Entrapment via synaptic-like c ...... lure after spinal cord injury.
@en
P2093
Amanda Tran
Angela R Filous
C James Howell
Joel M Levine
Sarah A Busch
Seong-il Lee
Shin H Kang
Teresa A Evans
William B Stallcup
P2860
P304
16369-16384
P356
10.1523/JNEUROSCI.1309-14.2014
P407
P577
2014-12-01T00:00:00Z