Multiple regions of the NG2 proteoglycan inhibit neurite growth and induce growth cone collapse
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Glial inhibition of CNS axon regenerationRegulating Axonal Responses to Injury: The Intersection between Signaling Pathways Involved in Axon Myelination and The Inhibition of Axon RegenerationOligodendrocyte Precursor Cells in Spinal Cord Injury: A Review and UpdateLeukocyte common antigen-related phosphatase is a functional receptor for chondroitin sulfate proteoglycan axon growth inhibitorsAdult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury.Analysis of axonal regeneration in the central and peripheral nervous systems of the NG2-deficient mouse.Chondroitin sulfate proteoglycans regulate the growth, differentiation and migration of multipotent neural precursor cells through the integrin signaling pathway.Adipocyte-derived collagen VI affects early mammary tumor progression in vivo, demonstrating a critical interaction in the tumor/stroma microenvironmentContributions of chondroitin sulfate proteoglycans to neurodevelopment, injury, and cancer.Binge-like postnatal alcohol exposure triggers cortical gliogenesis in adolescent ratsEntrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury.Global analysis of neuronal phosphoproteome regulation by chondroitin sulfate proteoglycansNG2 cells are uniformly distributed and NG2 is not required for barrel formation in the somatosensory cortex.NG2 cells are distinct from neurogenic cells in the postnatal mouse subventricular zone.Matrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injuryExtracellular regulators of axonal growth in the adult central nervous systemSpinal Glia Division Contributes to Conditioning Lesion-Induced Axon Regeneration Into the Injured Spinal Cord: Potential Role of Cyclic AMP-Induced Tissue Inhibitor of Metalloproteinase-1The endogenous proteoglycan-degrading enzyme ADAMTS-4 promotes functional recovery after spinal cord injury.Dexamethasone-coated neural probes elicit attenuated inflammatory response and neuronal loss compared to uncoated neural probesNG2, a common denominator for neuroinflammation, blood-brain barrier alteration, and oligodendrocyte precursor response in EAE, plays a role in dendritic cell activationA chemical genetic approach identifies piperazine antipsychotics as promoters of CNS neurite growth on inhibitory substrates.Vesicular release of glutamate from unmyelinated axons in white matter.NG2: a component of the glial scar that inhibits axon growth.NG2-expressing cells in the nervous system: role of the proteoglycan in migration and glial-neuron interaction.Corneal sulfated glycosaminoglycans and their effects on trigeminal nerve growth cone behavior in vitro: roles for ECM in cornea innervation.Role of chondroitin sulfate proteoglycans in axonal conduction in Mammalian spinal cord.Increased chondroitin sulfate proteoglycan expression in denervated brainstem targets following spinal cord injury creates a barrier to axonal regeneration overcome by chondroitinase ABC and neurotrophin-3.The reactions and role of NG2 glia in spinal cord injury.Degradation of chondroitin sulfate proteoglycans potentiates transplant-mediated axonal remodeling and functional recovery after spinal cord injury in adult rats.tPA-mediated generation of plasmin is catalyzed by the proteoglycan NG2.Establishment of neurovascular congruency in the mouse whisker system by an independent patterning mechanism.Molecular mechanisms of scar-sourced axon growth inhibitorsAxonal regrowth after spinal cord injury via chondroitinase and the tissue plasminogen activator (tPA)/plasmin system.An in vitro model of reactive astrogliosis and its effect on neuronal growth.Sugar-dependent modulation of neuronal development, regeneration, and plasticity by chondroitin sulfate proteoglycans.Proliferating NG2 cell-dependent angiogenesis and scar formation alter axon growth and functional recovery after spinal cord injury in mice.Retinoic acid synthesis by NG2 expressing cells promotes a permissive environment for axonal outgrowth.
P2860
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P2860
Multiple regions of the NG2 proteoglycan inhibit neurite growth and induce growth cone collapse
description
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
im Januar 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/01/01)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/01/01)
@nl
наукова стаття, опублікована в січні 2003
@uk
مقالة علمية (نشرت عام 2003)
@ar
name
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@ast
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@en
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@nl
type
label
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@ast
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@en
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@nl
prefLabel
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@ast
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@en
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@nl
P2093
P3181
P1476
Multiple regions of the NG2 pr ...... nd induce growth cone collapse
@en
P2093
Joel M Levine
Yvonne M Ughrin
Zhi Jiang Chen
P304
P3181
P356
10.1523/JNEUROSCI.23-01-00175.2003
P407
P577
2003-01-01T00:00:00Z