Blockade of TNF-α rapidly inhibits pain responses in the central nervous system. - Wikidata - awgldk - TriplyDB

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

http://www.wikidata.org/entity/Q34621522

about

TNF biology, pathogenic mechanisms and emerging therapeutic strategies Response to peripheral immune stimulation within the brain: magnetic resonance imaging perspective of treatment success A Neurologist's Guide to TNF Biology and to the Principles behind the Therapeutic Removal of Excess TNF in Disease Osteoarthritis joint pain: the cytokine connection Reflex principles of immunological homeostasis Population Explosions of Tiger Moth Lead to Lepidopterism Mimicking Infectious Fever Outbreaks Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agents Emerging targets in neuroinflammation-driven chronic pain Emerging roles of resolvins in the resolution of inflammation and pain Interleukin-1 inhibition and fatigue in primary Sjögren's syndrome--a double blind, randomised clinical trial From synovial tissue to peripheral blood: myeloid related protein 8/14 is a sensitive biomarker for effective treatment in early drug development in patients with rheumatoid arthritis Detection of enthesitis in children with enthesitis-related arthritis: dolorimetry compared to ultrasonography Maintaining brain health by monitoring inflammatory processes: a mechanism to promote successful aging Normothermic Mouse Functional MRI of Acute Focal Thermostimulation for Probing Nociception Imaging pain in arthritis: advances in structural and functional neuroimaging.Activity and connectivity changes of central projection areas revealed by functional magnetic resonance imaging in NaV1.8-deficient mice upon cold signaling.Effect of Trichilia monadelpha (Meliaceae) extracts on bone histomorphology in complete Freund's adjuvant-induced arthritis.Animal models of rheumatoid pain: experimental systems and insights.Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.Mapping the immunological homunculus Comparing Five Year Out-Come in Two Cohorts of Patients with Early Rheumatoid Arthritis - A BARFOT Study.Measurements in the Blood of BDNF for RA Patients and in Response to Anti-TNF Treatment Help Us to Clarify the Magnitude of Centrally Related Pain and to Explain the Relief of This Pain upon Treatment.Inflammation in intervertebral disc degeneration and regeneration Brain region-specific alterations in the gene expression of cytokines, immune cell markers and cholinergic system components during peripheral endotoxin-induced inflammation.Resolvins are potent analgesics for arthritic pain Resolving TRPV1- and TNF-α-mediated spinal cord synaptic plasticity and inflammatory pain with neuroprotectin D1.The brain in chronic pain: clinical implications.Antioxidant Effect of Spirulina (Arthrospira) maxima on Chronic Inflammation Induced by Freund's Complete Adjuvant in Rats A pilot study on biomarkers for tendinopathy: lower levels of serum TNF-α and other cytokines in females but not males with Achilles tendinopathy.Neural reflexes in inflammation and immunity In Vivo Assessment of Phage and Linezolid Based Implant Coatings for Treatment of Methicillin Resistant S. aureus (MRSA) Mediated Orthopaedic Device Related Infections.Peripheral inflammatory disease associated with centrally activated IL-1 system in humans and mice Immune-to-Brain Communication Pathways in Inflammation-Associated Sickness and Depression.Anti-arthritic property of crude extracts of Piptadeniastrum africanum (Mimosaceae) in complete Freund's adjuvant-induced arthritis in rats.Incidence and predictors of secondary fibromyalgia in an early arthritis cohort.Inflaming the brain: CRPS a model disease to understand neuroimmune interactions in chronic pain.Calcitonin gene-related peptide-expressing sensory neurons and spinal microglial reactivity contribute to pain states in collagen-induced arthritis Decreased neuroinflammation correlates to higher vagus nerve activity fluctuations in near-term ovine fetuses: a case for the afferent cholinergic anti-inflammatory pathway?Genomics of pain in osteoarthritis.Neuroinflammation and comorbidity of pain and depression.

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P2860

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

http://www.wikidata.org/entity/Q34621522

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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type

label

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

@en

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

@nl

prefLabel

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

@ast

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

@en

Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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PNAS.1011774108

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Proceedings of the National Academy of Sciences of the United States of America

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Blockade of TNF-α rapidly inhibits pain responses in the central nervous system.

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Andreas Hess

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Arnd Doerfler

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Cornelia Heindl

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Roland Axmann

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Silke Kreitz

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P2860

Biologics for rheumatoid arthritis: an overview of Cochrane reviews

Community structure in social and biological networks

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Oxygenation dependence of the transverse relaxation time of water protons in whole blood at high field

Cytokines in the pathogenesis of rheumatoid arthritis

Complex network measures of brain connectivity: uses and interpretations

Evolving concepts of rheumatoid arthritis

Brain magnetic resonance imaging with contrast dependent on blood oxygenation

TNF-alpha suppresses the expression of clock genes by interfering with E-box-mediated transcription.

Spinal inflammatory hyperalgesia is mediated by prostaglandin E receptors of the EP2 subtype.

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