Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice. - Wikidata - awgldk - TriplyDB

Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.

Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.

http://www.wikidata.org/entity/Q34633873

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Wnt signaling in cartilage development and diseases: lessons from animal studies On the evolutionary relationship between chondrocytes and osteoblasts Fibroblast growth factor signaling in skeletal development and disease The multifaceted role of the vasculature in endochondral fracture repair Cell Death in Chondrocytes, Osteoblasts, and Osteocytes Regulation of Long Bone Growth in Vertebrates; It Is Time to Catch Up Advances in Skeletal Dysplasia Genetics Quiescent Bone Lining Cells Are a Major Source of Osteoblasts During Adulthood.Specific ablation of mouse Fam20C in cells expressing type I collagen leads to skeletal defects and hypophosphatemia.Dual pathways to endochondral osteoblasts: a novel chondrocyte-derived osteoprogenitor cell identified in hypertrophic cartilage The developmental basis of mesenchymal stem/stromal cells (MSCs).Chondrocytes-Specific Expression of Osteoprotegerin Modulates Osteoclast Formation in Metaphyseal Bone Deficiency and Also Transgenic Overexpression of Timp-3 Both Lead to Compromised Bone Mass and Architecture In Vivo.Histone Deacetylases in Bone Development and Skeletal Disorders.Static osteogenesis does not precede dynamic osteogenesis in periosteal ossification of Pleurodeles (Caudata, Amphibia) and Pogona (Squamata, Lepidosauria).Chondrocytes Directly Transform into Bone Cells in Mandibular Condyle Growth.The Mighty Chondrocyte: No Bones about It.Conditional Deletion of Prolyl Hydroxylase Domain-Containing Protein 2 (Phd2) Gene Reveals Its Essential Role in Chondrocyte Function and Endochondral Bone Formation.Rapid modification of the bone microenvironment following short-term treatment with Cabozantinib in vivo.Acid ceramidase treatment enhances the outcome of autologous chondrocyte implantation in a rat osteochondral defect model.Transcriptional profiling of cortical versus cancellous bone from mechanically-loaded murine tibiae reveals differential gene expression.Understanding craniosynostosis as a growth disorder Tissue engineering strategies for promoting vascularized bone regeneration.Ihha induces hybrid cartilage-bone cells during zebrafish jawbone regeneration.Smpd3 Expression in both Chondrocytes and Osteoblasts Is Required for Normal Endochondral Bone Development.Basic science of osteoarthritis.β-catenin activity in late hypertrophic chondrocytes locally orchestrates osteoblastogenesis and osteoclastogenesis.Clec11a/osteolectin is an osteogenic growth factor that promotes the maintenance of the adult skeleton.Emerging roles for long noncoding RNAs in skeletal biology and disease.Healing of a Large Long-Bone Defect through Serum-Free In Vitro Priming of Human Periosteum-Derived Cells Fate of growth plate hypertrophic chondrocytes: death or lineage extension?Chondrogenesis and osteogenesis are one continuous developmental and lineage defined biological process.Expression of RMRP RNA is regulated in chondrocyte hypertrophy and determines chondrogenic differentiation.Reverse engineering development: crosstalk opportunities between developmental biology and tissue engineering.microRNA Regulation of Skeletal Development.Co-localization of Cell Lineage Markers and the Tomato Signal.BMP signalling in skeletal development, disease and repair.SOX9 and the many facets of its regulation in the chondrocyte lineage.Bone repair and stem cells Signaling pathways regulating cartilage growth plate formation and activity.

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P2860

Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.

http://www.wikidata.org/entity/Q34633873

description

2014 nî lūn-bûn

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2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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type

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

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JOURNAL.PGEN.1004820

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Chondrocytes transdifferentiat ...... and fracture healing in mice.

@en

P2093

Benoit de Crombrugghe

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Henry Adams

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Klaus von der Mark

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Stephen Henry

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William Norton

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Xin Zhou

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P2860

The novel zinc finger-containing transcription factor osterix is required for osteoblast differentiation and bone formation

Generalized lacZ expression with the ROSA26 Cre reporter strain

Improved monomeric red, orange and yellow fluorescent proteins derived from Discosoma sp. red fluorescent protein

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Canonical Wnt/beta-catenin signaling prevents osteoblasts from differentiating into chondrocytes

Sequential roles of Hedgehog and Wnt signaling in osteoblast development

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.

Specific expression of Cre recombinase in hypertrophic cartilage under the control of a BAC-Col10a1 promoter.

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