Sepsis lethality via exacerbated tissue infiltration and TLR-induced cytokine production by neutrophils is integrin α3β1-dependent. - Wikidata - awgldk - TriplyDB

Sepsis lethality via exacerbated tissue infiltration and TLR-induced cytokine production by neutrophils is integrin α3β1-dependent.

Sepsis lethality via exacerbated tissue infiltration and TLR-induced cytokine production by neutrophils is integrin α3β1-dependent.

http://www.wikidata.org/entity/Q34636844

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Diagnostic value of soluble triggering receptor expressed on myeloid cells in paediatric sepsis: a systematic review MST1-dependent vesicle trafficking regulates neutrophil transmigration through the vascular basement membrane Peripheral blood AKAP7 expression as an early marker for lymphocyte-mediated post-stroke blood brain barrier disruption Regulation of tissue infiltration by neutrophils: role of integrin α3β1 and other factors.Sepsis-induced immune dysfunction: can immune therapies reduce mortality?Immune and regulatory functions of neutrophils in inflammatory bone loss.WISP1-αvβ3 integrin signaling positively regulates TLR-triggered inflammation response in sepsis induced lung injury GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.Neutrophil Extravasation Cascade: What Can We Learn from Two-photon Intravital Imaging?Paracoccidioides brasiliensis induces cytokine secretion in epithelial cells in a protease-activated receptor-dependent (PAR) manner.Histoplasma capsulatum-Induced Cytokine Secretion in Lung Epithelial Cells Is Dependent on Host Integrins, Src-Family Kinase Activation, and Membrane Raft Recruitment.Role of cellular events in the pathophysiology of sepsis.The immune system's role in sepsis progression, resolution, and long-term outcome.From leukocyte recruitment to resolution of inflammation: the cardinal role of integrins.Integrin Cross-Talk Regulates the Human Neutrophil Response to Fungal β-Glucan in the Context of the Extracellular Matrix: A Prominent Role for VLA3 in the Antifungal Response.Potential Immunotherapeutics for Immunosuppression in Sepsis.Recombinant Human Vimentin Binds to P-Selectin and Blocks Neutrophil Capture and Rolling on Platelets and Endothelium.Activated Protein C Attenuates Severe Inflammation by Targeting VLA-3high Neutrophil Subpopulation in Mice.A ligand-specific blockade of the integrin Mac-1 selectively targets pathologic inflammation while maintaining protective host-defense.

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P2860

Sepsis lethality via exacerbated tissue infiltration and TLR-induced cytokine production by neutrophils is integrin α3β1-dependent.

http://www.wikidata.org/entity/Q34636844

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@ast

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@en

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

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type

label

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@ast

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@en

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@nl

prefLabel

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@ast

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@en

Sepsis lethality via exacerbat ...... ls is integrin α3β1-dependent.

@nl

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Sepsis lethality via exacerbat ...... ils is integrin α3β1-dependent

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P2093

Anthony P Pietropaoli

http://www.w3.org/2001/XMLSchema#string

Arnoud Sonnenberg

http://www.w3.org/2001/XMLSchema#string

Hongmei Yang

http://www.w3.org/2001/XMLSchema#string

Kathleen L Falkner

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Kihong Lim

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Minsoo Kim

http://www.w3.org/2001/XMLSchema#string

Pranita P Sarangi

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Yelena V Lerman

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P2860

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

CD14 cooperates with complement receptor 3 to mediate MyD88-independent phagocytosis of Borrelia burgdorferi

Targeting leukocyte integrins in human diseases

The impact of the extracellular matrix on inflammation

Integrin CD11b negatively regulates TLR-triggered inflammatory responses by activating Syk and promoting degradation of MyD88 and TRIF via Cbl-b

Divergent effects of platelet-endothelial cell adhesion molecule-1 and beta 3 integrin blockade on leukocyte transmigration in vivo

Impaired immunity and enhanced resistance to endotoxin in the absence of neutrophil elastase and cathepsin G

Neutrophils and immunity: challenges and opportunities

Beta1 integrins differentially control extravasation of inflammatory cell subsets into the CNS during autoimmunity

Uropod elongation is a common final step in leukocyte extravasation through inflamed vessels.

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3515-3523

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scholarly article

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10.1182/BLOOD-2014-01-552943

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24

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Toll-like receptor

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