Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage. - Wikidata - awgldk - TriplyDB

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

http://www.wikidata.org/entity/Q34639033

about

Ribosomal Protein S6 Phosphorylation in the Nervous System: From Regulation to Function mTOR inhibition suppresses established epilepsy in a mouse model of cortical dysplasia.Dysfunctional epileptic neuronal circuits and dysmorphic dendritic spines are mitigated by platelet-activating factor receptor antagonism mTOR activation is a biomarker and a central pathway to autoimmune disorders, cancer, obesity, and aging Effect of Chronic Administration of Low Dose Rapamycin on Development and Immunity in Young Rats Virulence factor activity relationships (VFARs): a bioinformatics perspective.mTORC1 Is a Local, Postsynaptic Voltage Sensor Regulated by Positive and Negative Feedback Pathways.Wortmannin Attenuates Seizure-Induced Hyperactive PI3K/Akt/mTOR Signaling, Impaired Memory, and Spine Dysmorphology in Rats Importance of neonatal immunoglobulin transfer for hippocampal development and behaviour in the newborn pig PTEN deletion from adult-generated dentate granule cells disrupts granule cell mossy fiber axon structure.mTOR signaling in epilepsy: insights from malformations of cortical development Analysis of Proteins That Rapidly Change Upon Mechanistic/Mammalian Target of Rapamycin Complex 1 (mTORC1) Repression Identifies Parkinson Protein 7 (PARK7) as a Novel Protein Aberrantly Expressed in Tuberous Sclerosis Complex (TSC)Rapamycin prevents acute dendritic injury following seizures.Impact of rapamycin on status epilepticus induced hippocampal pathology and weight gain.Neuroprotective levels of IGF-1 exacerbate epileptogenesis after brain injury.Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.Screening the Molecular Framework Underlying Local Dendritic mRNA Translation.WONOEP appraisal: Biomarkers of epilepsy-associated comorbidities.Proteomic Analysis After Status Epilepticus Identifies UCHL1 as Protective Against Hippocampal Injury.Spatiotemporal profile of Map2 and microglial changes in the hippocampal CA1 region following pilocarpine-induced status epilepticus.Inflammatory aspects of epileptogenesis: contribution of molecular inflammatory mechanisms.mTOR referees memory and disease through mRNA repression and competition.TIMP3 Attenuates the Loss of Neural Stem Cells, Mature Neurons and Neurocognitive Dysfunction in Traumatic Brain Injury.The effect of early life status epilepticus on ultrasonic vocalizations in mice Early cardiac electrographic and molecular remodeling in a model of status epilepticus and acquired epilepsy.Neural Progenitor Cells Rptor Ablation Impairs Development but Benefits to Seizure-Induced Behavioral Abnormalities.Commonalities in epileptogenic processes from different acute brain insults: Do they translate?Mechanistic target of rapamycin complex 1 and 2 in human temporal lobe epilepsy.A Mercaptoacetamide-Based Class II Histone Deacetylase Inhibitor Increases Dendritic Spine Density via RasGRF1/ERK Pathway.Status Epilepticus Triggers Time-Dependent Alterations in Microglia Abundance and Morphological Phenotypes in the Hippocampus.mTOR-dependent alterations of Kv1.1 subunit expression in the neuronal subset-specific Pten knockout mouse model of cortical dysplasia with epilepsy.Signaling Pathways and Cellular Mechanisms Regulating Mossy Fiber Sprouting in the Development of Epilepsy.Effects of rapamycin and curcumin on inflammation and oxidative stress in vitro and in vivo - in search of potential anti-epileptogenic strategies for temporal lobe epilepsy

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Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

http://www.wikidata.org/entity/Q34639033

description

2013 nî lūn-bûn

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2013 թուականի Մարտին հրատարակուած գիտական յօդուած

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2013 թվականի մարտին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@ast

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@en

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@nl

type

label

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@ast

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@en

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@nl

prefLabel

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@ast

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@en

Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@nl

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JOURNAL.PONE.0057808

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Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.

@en

P2093

Amy L Brewster

http://www.w3.org/2001/XMLSchema#string

Anne E Anderson

http://www.w3.org/2001/XMLSchema#string

Fabiola Vanegas

http://www.w3.org/2001/XMLSchema#string

Joaquin N Lugo

http://www.w3.org/2001/XMLSchema#string

Vinit V Patil

http://www.w3.org/2001/XMLSchema#string

Wai L Lee

http://www.w3.org/2001/XMLSchema#string

Yan Qian

http://www.w3.org/2001/XMLSchema#string

P2860

S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

Reversal of learning deficits in a Tsc2+/- mouse model of tuberous sclerosis

The MAP2/Tau family of microtubule-associated proteins

Modification of seizure activity by electrical stimulation. II. Motor seizure

Upstream and downstream of mTOR

Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB

Mapping the spatio-temporal pattern of the mammalian target of rapamycin (mTOR) activation in temporal lobe epilepsy

Pten regulates neuronal arborization and social interaction in mice.

Enduring, handling-evoked enhancement of hippocampal memory function and glucocorticoid receptor expression involves activation of the corticotropin-releasing factor type 1 receptor

Involvement of mTOR kinase in cytokine-dependent microglial activation and cell proliferation

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23536771

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status epilepticus

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3594232

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