ROS inhibit autophagy by downregulating ULK1 mediated by the phosphorylation of p53 in selenite-treated NB4 cells. - Wikidata - awgldk - TriplyDB

ROS inhibit autophagy by downregulating ULK1 mediated by the phosphorylation of p53 in selenite-treated NB4 cells.

ROS inhibit autophagy by downregulating ULK1 mediated by the phosphorylation of p53 in selenite-treated NB4 cells.

http://www.wikidata.org/entity/Q34662354

about

P53 functional abnormality in mesenchymal stem cells promotes osteosarcoma development Neuroblastoma: oncogenic mechanisms and therapeutic exploitation of necroptosis Ultraconserved long non-coding RNA uc.63 in breast cancer Autophagy: In the cROSshairs of cancer.Differential autophagic effects of vital dyes in retinal pigment epithelial ARPE-19 and photoreceptor 661W cells p53-regulated autophagy is controlled by glycolysis and determines cell fate.Nuclear ULK1 promotes cell death in response to oxidative stress through PARP1 p73 regulates basal and starvation-induced liver metabolism in vivo.Setdb1, a novel interactor of ΔNp63, is involved in breast tumorigenesis Metabolic pathways regulated by TAp73 in response to oxidative stress Regulation of ER stress-induced autophagy by GSK3β-TIP60-ULK1 pathway.Selenite inhibits glutamine metabolism and induces apoptosis by regulating GLS1 protein degradation via APC/C-CDH1 pathway in colorectal cancer cells.The critical molecular interconnections in regulating apoptosis and autophagy.Ferrocene-cinchona hybrids with triazolyl-chalcone linkers act as pro-oxidants and sensitize human cancer cell lines to paclitaxel.Serine/Threonine Kinase Unc-51-like Kinase-1 (Ulk1) Phosphorylates the Co-chaperone Cell Division Cycle Protein 37 (Cdc37) and Thereby Disrupts the Stability of Cdc37 Client Proteins.Autophagy regulation and its role in gastric cancer and colorectal cancer.Differential regulated microRNA by wild type and mutant p53 in induced pluripotent stem cells p73 promotes glioblastoma cell invasion by directly activating POSTN (periostin) expression.The p53 tetramer shows an induced-fit interaction of the C-terminal domain with the DNA-binding domain.Sitagliptin-mediated preservation of endothelial progenitor cell function via augmenting autophagy enhances ischaemic angiogenesis in diabetes.GDC-0152-induced autophagy promotes apoptosis in HL-60 cells.p53 and metabolism: from mechanism to therapeutics.

P2860

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P2860

ROS inhibit autophagy by downregulating ULK1 mediated by the phosphorylation of p53 in selenite-treated NB4 cells.

http://www.wikidata.org/entity/Q34662354

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@ast

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@en

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@nl

type

label

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@ast

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@en

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@nl

prefLabel

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@ast

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@en

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells.

@nl

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P1433

Cell Death and Disease

P1476

ROS inhibit autophagy by downr ...... in selenite-treated NB4 cells

@en

P2093

C Xu

http://www.w3.org/2001/XMLSchema#string

J An

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K Hui

http://www.w3.org/2001/XMLSchema#string

K Shi

http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

Y Ci

http://www.w3.org/2001/XMLSchema#string

Y Yang

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P2860

Nutrient-dependent mTORC1 association with the ULK1-Atg13-FIP200 complex required for autophagy

Analysis of DRAM-related proteins reveals evolutionarily conserved and divergent roles in the control of autophagy

mTOR/p70S6K signaling distinguishes routine, maintenance-level autophagy from autophagic cell death during influenza A infection

ULK-Atg13-FIP200 complexes mediate mTOR signaling to the autophagy machinery

ULK1.ATG13.FIP200 complex mediates mTOR signaling and is essential for autophagy

Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

Rapamycin-FKBP specifically blocks growth-dependent activation of and signaling by the 70 kd S6 protein kinases

Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis

Simultaneous induction of non-canonical autophagy and apoptosis in cancer cells by ROS-dependent ERK and JNK activation

AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1

P304

e1542

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P356

10.1038/CDDIS.2014.506

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P478

5

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2014-11-27T00:00:00Z

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25429619

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P921

phosphorylation

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