Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells - Wikidata - awgldk - TriplyDB

Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells

Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells

http://www.wikidata.org/entity/Q34693000

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Tamoxifen Resistance: Emerging Molecular Targets Epithelial-Mesenchymal Transition (EMT) and Regulation of EMT Factors by Steroid Nuclear Receptors in Breast Cancer: A Review and in Silico Investigation The Role of MicroRNAs as Predictors of Response to Tamoxifen Treatment in Breast Cancer Patients Roles for miRNAs in endocrine resistance in breast cancer MiR-200, a new star miRNA in human cancer Mechanisms of endocrine resistance in breast cancer: an overview of the proposed roles of noncoding RNA The microRNA-200 family: small molecules with novel roles in cancer development, progression and therapy Long-term exposure of MCF-7 breast cancer cells to ethanol stimulates oncogenic features Clinicopathological and prognostic implications of the miR-200 family in patients with epithelial ovarian cancer.Computational analysis identifies a sponge interaction network between long non-coding RNAs and messenger RNAs in human breast cancer.Association of microRNA-93, 190, 200b and receptor status in core biopsies from stage III breast cancer patients.miR-200c overexpression is associated with better efficacy of EGFR-TKIs in non-small cell lung cancer patients with EGFR wild-type.miR-200b inhibits prostate cancer EMT, growth and metastasis MicroRNA-125b upregulation confers aromatase inhibitor resistance and is a novel marker of poor prognosis in breast cancer MiR-125b regulates epithelial-mesenchymal transition via targeting Sema4C in paclitaxel-resistant breast cancer cells.Functional role of miR-10b in tamoxifen resistance of ER-positive breast cancer cells through down-regulation of HDAC4.MicroRNA as tools and therapeutics in lung cancer MicroRNA-200 Family Profile: A Promising Ancillary Tool for Accurate Cancer Diagnosis miRNAs regulated by estrogens, tamoxifen, and endocrine disruptors and their downstream gene targets.miR-378a-3p modulates tamoxifen sensitivity in breast cancer MCF-7 cells through targeting GOLT1A.Silibinin suppresses EMT-driven erlotinib resistance by reversing the high miR-21/low miR-200c signature in vivo miRNA clusters as therapeutic targets for hormone-resistant breast cancer.Multidrug-resistant breast cancer: current perspectives.The novel role of miRNAs for tamoxifen resistance in human breast cancer.Impact of Histone Deacetylase Inhibitors on microRNA Expression and Cancer Therapy: A Review.MicroRNAs and Cancer Drug Resistance.miR-200c: a versatile watchdog in cancer progression, EMT, and drug resistance.Roles and epigenetic regulation of epithelial-mesenchymal transition and its transcription factors in cancer initiation and progression.Exosomal miR-221/222 enhances tamoxifen resistance in recipient ER-positive breast cancer cells.Prognostic role of microRNA-221 in various human malignant neoplasms: a meta-analysis of 20 related studies Anacardic Acid, Salicylic Acid, and Oleic Acid Differentially Alter Cellular Bioenergetic Function in Breast Cancer Cells.Prognostic role of microRNA-145 in various human malignant neoplasms: a meta-analysis of 18 related studies.A Potential Prognostic Long Noncoding RNA Signature to Predict Recurrence among ER-positive Breast Cancer Patients Treated with Tamoxifen.Role of the EZH2/miR-200 axis in STAT3-mediated OSCC invasion.miR-200b as a prognostic factor targets multiple members of RAB family in glioma.Knockdown of miR-194-5p inhibits cell proliferation, migration and invasion in breast cancer by regulating the Wnt/β-catenin signaling pathway

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P2860

Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells

http://www.wikidata.org/entity/Q34693000

description

2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Reduced expression of miR-200 ...... LY2 human breast cancer cells

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Lacey M Litchfield

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Numan Al-Rayyan

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Penn Muluhngwi

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Tissa T Manavalan

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Yun Teng

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P2860

miR-200 enhances mouse breast cancer cell colonization to form distant metastases

Loss of miR-200c: A Marker of Aggressiveness and Chemoresistance in Female Reproductive Cancers

The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

Gemcitabine sensitivity can be induced in pancreatic cancer cells through modulation of miR-200 and miR-21 expression by curcumin or its analogue CDF

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells

SIP1/ZEB2 induces EMT by repressing genes of different epithelial cell-cell junctions

The nuclear factor kappa B inhibitor parthenolide restores ICI 182,780 (Faslodex; fulvestrant)-induced apoptosis in antiestrogen-resistant breast cancer cells.

The miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identity

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Carolyn M. Klinge

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