about
Identification and characterization of nucleolin as a COUP-TFII coactivator of retinoic acid receptor β transcription in breast cancer cellsBinding of type II nuclear receptors and estrogen receptor to full and half-site estrogen response elements in vitroEstradiol downregulates miR-21 expression and increases miR-21 target gene expression in MCF-7 breast cancer cellsEstrogenic control of mitochondrial function and biogenesisThe aryl hydrocarbon receptor interacts with estrogen receptor alpha and orphan receptors COUP-TFI and ERRalpha1The aryl hydrocarbon receptor (AHR)/AHR nuclear translocator (ARNT) heterodimer interacts with naturally occurring estrogen response elementsResponse element sequence modulates estrogen receptor alpha and beta affinity and activity5-Aza-2-deoxycytidine and trichostatin A increase COUP-TFII expression in antiestrogen-resistant breast cancer cell lines.Sex differences in estrogen receptor subcellular location and activity in lung adenocarcinoma cellsDehydroepiandrosterone-induces miR-21 transcription in HepG2 cells through estrogen receptor β and androgen receptorDHEA metabolites activate estrogen receptors alpha and betaReduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cellsLigand-dependent differences in estrogen receptor beta-interacting proteins identified in lung adenocarcinoma cells corresponds to estrogenic responses.Targeting the intracellular MUC1 C-terminal domain inhibits proliferation and estrogen receptor transcriptional activity in lung adenocarcinoma cells.Differential expression of microRNA expression in tamoxifen-sensitive MCF-7 versus tamoxifen-resistant LY2 human breast cancer cells.miRNAs regulated by estrogens, tamoxifen, and endocrine disruptors and their downstream gene targets.Multiple roles of COUP-TFII in cancer initiation and progressionEstradiol stimulates transcription of nuclear respiratory factor-1 and increases mitochondrial biogenesis.Endocrine disruptors fludioxonil and fenhexamid stimulate miR-21 expression in breast cancer cells.Estradiol and tamoxifen regulate NRF-1 and mitochondrial function in mouse mammary gland and uterus.Estrogen Regulation of MicroRNA ExpressionCOUP-TFII inhibits NFkappaB activation in endocrine-resistant breast cancer cells.Activity and intracellular location of estrogen receptors alpha and beta in human bronchial epithelial cells.β-D-glucan inhibits endocrine-resistant breast cancer cell proliferation and alters gene expressionMicroRNA-21 promotes cell transformation by targeting the programmed cell death 4 gene.Short heterodimer partner (SHP) orphan nuclear receptor inhibits the transcriptional activity of aryl hydrocarbon receptor (AHR)/AHR nuclear translocator (ARNT).Estrogen response element sequence impacts the conformation and transcriptional activity of estrogen receptor alpha.Effects of estradiol and 4-hydroxytamoxifen on the conformation, thermal stability, and DNA recognition of estrogen receptor beta.Comparison of transcriptional synergy of estrogen receptors alpha and beta from multiple tandem estrogen response elements.Differential impact of flanking sequences on estradiol- vs 4-hydroxytamoxifen-liganded estrogen receptor binding to estrogen responsive element DNA.Transcription profiling of estrogen target genes in young and old mouse uterus.Stability of the ligand-estrogen receptor interaction depends on estrogen response element flanking sequences and cellular factors.Phosphorylation of purified estradiol-liganded estrogen receptor by casein kinase II increases estrogen response element binding but does not alter ligand stability.The miR-29 transcriptome in endocrine-sensitive and resistant breast cancer cells.Identification of miRNAs as biomarkers for acquired endocrine resistance in breast cancer.Effect of nonpersistent pesticides on estrogen receptor, androgen receptor, and aryl hydrocarbon receptor.MUC1/A and MUC1/B splice variants differentially regulate inflammatory cytokine expressionSphingosine-1-phosphate receptor-3 signaling up-regulates epidermal growth factor receptor and enhances epidermal growth factor receptor-mediated carcinogenic activities in cultured lung adenocarcinoma cells.Inhibition of non-small-cell lung cancer growth by combined fulvestrant and vandetanib.Repression of activated aryl hydrocarbon receptor-induced transcriptional activation by 5alpha-dihydrotestosterone in human prostate cancer LNCaP and human breast cancer T47D cells.
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Carolyn M Klinge
@nl
Carolyn M Klinge
@sl
Carolyn M. Klinge
@en
Carolyn M. Klinge
@es
type
label
Carolyn M Klinge
@nl
Carolyn M Klinge
@sl
Carolyn M. Klinge
@en
Carolyn M. Klinge
@es
prefLabel
Carolyn M Klinge
@nl
Carolyn M Klinge
@sl
Carolyn M. Klinge
@en
Carolyn M. Klinge
@es
P108
P106
P1153
7004207221
P21
P31
P496
0000-0002-3358-4378