Anomalous levels of Cl- transporters in the hippocampal subiculum from temporal lobe epilepsy patients make GABA excitatory. - Wikidata - awgldk - TriplyDB

Anomalous levels of Cl- transporters in the hippocampal subiculum from temporal lobe epilepsy patients make GABA excitatory.

Anomalous levels of Cl- transporters in the hippocampal subiculum from temporal lobe epilepsy patients make GABA excitatory.

http://www.wikidata.org/entity/Q34694335

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NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currents Utility of magnetic resonance spectroscopic imaging for human epilepsy Current view on the functional regulation of the neuronal K(+)-Cl(-) cotransporter KCC2 Abnormal chloride homeostasis in the substancia nigra pars reticulata contributes to locomotor deficiency in a model of acute liver injury Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potential Molecular and evolutionary insights into the structural organization of cation chloride cotransporters The RCC1 domain of protein associated with Myc (PAM) interacts with and regulates KCC2 Blocking early GABA depolarization with bumetanide results in permanent alterations in cortical circuits and sensorimotor gating deficits Differences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures.A sensitive period of mice inhibitory system to neonatal GABA enhancement by vigabatrin is brain region dependent High throughput electrophysiology with Xenopus oocytes Potassium dynamics in the epileptic cortex: new insights on an old topic.A novel regulatory locus of phosphorylation in the C terminus of the potassium chloride cotransporter KCC2 that interferes with N-ethylmaleimide or staurosporine-mediated activation.The location of olfactory receptors within olfactory epithelium is independent of odorant volatility and solubility Chloride regulation in the pain pathway Bumetanide, an NKCC1 antagonist, does not prevent formation of epileptogenic focus but blocks epileptic focus seizures in immature rat hippocampus.Anomalous expression of chloride transporters in the sclerosed hippocampus of mesial temporal lobe epilepsy patients Transient muscarinic and glutamatergic stimulation of neural stem cells triggers acute and persistent changes in differentiation.Functional genomics in Drosophila models of human disease.Morpho-physiologic characteristics of dorsal subicular network in mice after pilocarpine-induced status epilepticus Computational modeling reveals dendritic origins of GABA(A)-mediated excitation in CA1 pyramidal neurons.Prevention or modification of epileptogenesis after brain insults: experimental approaches and translational research.A role for intracellular zinc in glioma alteration of neuronal chloride equilibrium.Ovariectomy modulation of morphine analgesia of neuropathic pain is associated with the change of K(+)-Cl(-) cotransporter 2 protein level in spinal dorsal horn.A stereological study of synapse number in the epileptic human hippocampus.STE20/SPS1-related proline/alanine-rich kinase is involved in plasticity of GABA signaling function in a mouse model of acquired epilepsy.Altered GABA signaling in early life epilepsies.Age-dependent changes in intrinsic neuronal excitability in subiculum after status epilepticus.Loss of cation-chloride cotransporter expression in preterm infants with white matter lesions: implications for the pathogenesis of epilepsy Late development of the GABAergic system in the human cerebral cortex and white matter Possible alterations in GABAA receptor signaling that underlie benzodiazepine-resistant seizures.Calcium-activated chloride channels (CaCCs) regulate action potential and synaptic response in hippocampal neurons.Altered inhibition in tuberous sclerosis and type IIb cortical dysplasia.Beyond the CA1 subfield: Local hippocampal shape changes in MRI-negative temporal lobe epilepsy.Modulation of spinal GABAergic analgesia by inhibition of chloride extrusion capacity in mice.Reduction of TrkB expression de novo in the adult mouse impairs epileptogenesis in the kindling model.Rapamycin down-regulates KCC2 expression and increases seizure susceptibility to convulsants in immature rats.Sequel of spontaneous seizures after kainic acid-induced status epilepticus and associated neuropathological changes in the subiculum and entorhinal cortex Detrimental effect of post Status Epilepticus treatment with ROCK inhibitor Y-27632 in a pilocarpine model of temporal lobe epilepsy.WONOEP appraisal: Development of epilepsy biomarkers-What we can learn from our patients?

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Anomalous levels of Cl- transporters in the hippocampal subiculum from temporal lobe epilepsy patients make GABA excitatory.

http://www.wikidata.org/entity/Q34694335

description

2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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type

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Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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prefLabel

Anomalous levels of Cl- transp ...... patients make GABA excitatory.

@ast

Anomalous levels of Cl- transp ...... patients make GABA excitatory.

@en

Anomalous levels of Cl- transp ...... patients make GABA excitatory.

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Proceedings of the National Academy of Sciences of the United States of America

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Anomalous levels of Cl- transp ...... patients make GABA excitatory

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Amici M

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Di Angelantonio S

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Eusebi F

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Miledi R

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Ragozzino D

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Scoppetta C

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Sobrero F

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Spinelli G

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P2860

BDNF-induced TrkB activation down-regulates the K+-Cl- cotransporter KCC2 and impairs neuronal Cl- extrusion

Two developmental switches in GABAergic signalling: the K+-Cl- cotransporter KCC2 and carbonic anhydrase CAVII

NKCC1 transporter facilitates seizures in the developing brain

The K+/Cl- co-transporter KCC2 renders GABA hyperpolarizing during neuronal maturation

Excitatory actions of gaba during development: the nature of the nurture

BDNF modulates GABAA receptors microtransplanted from the human epileptic brain to Xenopus oocytes.

Abnormal GABAA receptors from the human epileptic hippocampal subiculum microtransplanted to Xenopus oocytes.

On the origin of interictal activity in human temporal lobe epilepsy in vitro.

Genetics of epilepsy

Expression of functional neurotransmitter receptors in Xenopus oocytes after injection of human brain membranes

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Addolorata Mascia

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