Longitudinal behavioral, cross-sectional transcriptional and histopathological characterization of a knock-in mouse model of Huntington's disease with 140 CAG repeats.

Longitudinal behavioral, cross-sectional transcriptional and histopathological characterization of a knock-in mouse model of Huntington's disease with 140 CAG repeats.

Item
http://www.wikidata.org/entity/Q34699467
Mouse models of polyglutamine diseases: review and data table. Part IA broad phenotypic screen identifies novel phenotypes driven by a single mutant allele in Huntington's disease CAG knock-in mice.Differential loss of thalamostriatal and corticostriatal input to striatal projection neuron types prior to overt motor symptoms in the Q140 knock-in mouse model of Huntington's disease.Comprehensive behavioral and molecular characterization of a new knock-in mouse model of Huntington's disease: zQ175.Transcriptional modulator H2A histone family, member Y (H2AFY) marks Huntington disease activity in man and mouseAbnormal burst patterns of single neurons recorded in the substantia nigra reticulata of behaving 140 CAG Huntington's disease miceDysregulation of dopamine receptor D2 as a sensitive measure for Huntington disease pathology in model miceUse of a force-sensing automated open field apparatus in a longitudinal study of multiple behavioral deficits in CAG140 Huntington's disease model miceQuantitative Electroencephalographic Analysis Provides an Early-Stage Indicator of Disease Onset and Progression in the zQ175 Knock-In Mouse Model of Huntington's Disease.A Longitudinal Motor Characterisation of the HdhQ111 Mouse Model of Huntington's DiseaseLoss of corticostriatal and thalamostriatal synaptic terminals precedes striatal projection neuron pathology in heterozygous Q140 Huntington's disease mice.Long-term analyses of innervation and neuromuscular integrity in the Trembler-J mouse model of Charcot-Marie-Tooth diseaseCholinergic interneurons in the Q140 knockin mouse model of Huntington's disease: Reductions in dendritic branching and thalamostriatal input.Cell-specific deletion of PGC-1α from medium spiny neurons causes transcriptional alterations and age-related motor impairment.Disrupted striatal neuron inputs and outputs in Huntington's disease.
P2860

Longitudinal behavioral, cross-sectional transcriptional and histopathological characterization of a knock-in mouse model of Huntington's disease with 140 CAG repeats.

Item
http://www.wikidata.org/entity/Q34699467
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@ast
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@en
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@nl
type
label
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@ast
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@en
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@nl
prefLabel
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@ast
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@en
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P921
P932
P356
P1433
P1476
Longitudinal behavioral, cross ...... disease with 140 CAG repeats.
@en
P2093
P2860
P304
P31
P356
10.1016/J.EXPNEUROL.2010.12.017
P407
P433
P478
P577
P5875
P698
P921
P932