Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes. - Wikidata - awgldk - TriplyDB

Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes.

Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes.

http://www.wikidata.org/entity/Q34721438

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Th1 and Th17 Cells in Tuberculosis: Protection, Pathology, and Biomarkers Versatile myeloid cell subsets contribute to tuberculosis-associated inflammation Reverse translation in tuberculosis: neutrophils provide clues for understanding development of active disease Critical and independent role for SOCS3 in either myeloid or T cells in resistance to Mycobacterium tuberculosis Granulomas and Inflammation: Host-Directed Therapies for Tuberculosis.Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility Oral Administration of Heat-Killed Mycobacterium manresensis Delays Progression toward Active Tuberculosis in C3HeB/FeJ Mice.Intracellular bacillary burden reflects a burst size for Mycobacterium tuberculosis in vivo.Altered cellular infiltration and cytokine levels during early Mycobacterium tuberculosis sigC mutant infection are associated with late-stage disease attenuation and milder immunopathology in mice.P2X7 Receptor in Bone Marrow-Derived Cells Aggravates Tuberculosis Caused by Hypervirulent Mycobacterium bovis.In mice, tuberculosis progression is associated with intensive inflammatory response and the accumulation of Gr-1 cells in the lungs.Host genetics in granuloma formation: human-like lung pathology in mice with reciprocal genetic susceptibility to M. tuberculosis and M. avium.Neutrophils in Tuberculosis: Heterogeneity Shapes the Way?The introduction of mesenchymal stromal cells induces different immunological responses in the lungs of healthy and M. tuberculosis infected mice.The adaptor molecule CARD9 is essential for tuberculosis control.Interleukin-11 drives early lung inflammation during Mycobacterium tuberculosis infection in genetically susceptible mice.Pathological role of interleukin 17 in mice subjected to repeated BCG vaccination after infection with Mycobacterium tuberculosis.B cells delay neutrophil migration toward the site of stimulus: tardiness critical for effective bacillus Calmette-Guérin vaccination against tuberculosis infection in mice.Programmed death-1 (PD-1)-deficient mice are extraordinarily sensitive to tuberculosis.Dichotomy between factors inducing the immunosuppressive enzyme IL-4-induced gene 1 (IL4I1) in B lymphocytes and mononuclear phagocytes Natural transmission of Plasmodium berghei exacerbates chronic tuberculosis in an experimental co-infection model IL-17 and Th17 cells in tuberculosis Asymptomatic cattle naturally infected with Mycobacterium bovis present exacerbated tissue pathology and bacterial dissemination.B cells regulate neutrophilia during Mycobacterium tuberculosis infection and BCG vaccination by modulating the interleukin-17 response.Cell-mediated immune responses in tuberculosis.Genetic and functional characterization of the mouse Trl3 locus in defense against tuberculosis.Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection.Comparing efficacy of BCG/lactoferrin primary vaccination versus booster regimen.Neutrophil Extracellular Traps are Involved in the Innate Immune Response to Infection with Leptospira.Th17 cells in human disease.Bacillary replication and macrophage necrosis are determinants of neutrophil recruitment in tuberculosis.Ecto-5'-Nucleotidase (CD73) Deficiency in Mycobacterium tuberculosis-Infected Mice Enhances Neutrophil Recruitment Gr-1dimCD11b+ immature myeloid-derived suppressor cells but not neutrophils are markers of lethal tuberculosis infection in mice Enhanced protection to Mycobacterium tuberculosis infection in IL-10-deficient mice is accompanied by early and enhanced Th1 responses in the lung.Lung necrosis and neutrophils reflect common pathways of susceptibility to Mycobacterium tuberculosis in genetically diverse, immune-competent mice.Hypervirulent Mycobacterium tuberculosis strain triggers necrotic lung pathology associated with enhanced recruitment of neutrophils in resistant C57BL/6 mice Mouse models of human TB pathology: roles in the analysis of necrosis and the development of host-directed therapies.Gene Expression Profiling of Tuberculous Meningitis Co-infected with HIV Early innate immunity determines outcome of Mycobacterium tuberculosis pulmonary infection in rabbits Dominant role of the sst1 locus in pathogenesis of necrotizing lung granulomas during chronic tuberculosis infection and reactivation in genetically resistant hosts.

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P2860

Genetically determined susceptibility to tuberculosis in mice causally involves accelerated and enhanced recruitment of granulocytes.

http://www.wikidata.org/entity/Q34721438

description

2006 nî lūn-bûn

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2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2006 թվականի հուլիսին հրատարակված գիտական հոդված

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2006年の論文

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Genetically determined suscept ...... d recruitment of granulocytes.

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Infection and Immunity

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Christine Keller

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Corinna Hermann

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Stefan Ehlers

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P2860

Roles of PLC-beta2 and -beta3 and PI3Kgamma in chemoattractant-mediated signal transduction

Severe mycobacterial and Salmonella infections in interleukin-12 receptor-deficient patients

Tuberculosis toxin blocking phagosome maturation inhibits a novel Ca2+/calmodulin-PI3K hVPS34 cascade

Consensus statement. Global burden of tuberculosis: estimated incidence, prevalence, and mortality by country. WHO Global Surveillance and Monitoring Project

CSF-1 as a regulator of macrophage activation and immune responses

Essential role of phosphoinositide 3-kinase delta in neutrophil directional movement

Ipr1 gene mediates innate immunity to tuberculosis.

Controlling the false discovery rate in behavior genetics research

The adaptor protein p40(phox) as a positive regulator of the superoxide-producing phagocyte oxidase.

Genotype frequencies of the +874T-->A single nucleotide polymorphism in the first intron of the interferon-gamma gene in a sample of Sicilian patients affected by tuberculosis.

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10.1128/IAI.00057-06

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16790804

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Akafuba bulwadde

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