Interferon regulatory factor 3 and type I interferons are protective in alcoholic liver injury in mice by way of crosstalk of parenchymal and myeloid cells. - Wikidata - awgldk - TriplyDB

Interferon regulatory factor 3 and type I interferons are protective in alcoholic liver injury in mice by way of crosstalk of parenchymal and myeloid cells.

Interferon regulatory factor 3 and type I interferons are protective in alcoholic liver injury in mice by way of crosstalk of parenchymal and myeloid cells.

http://www.wikidata.org/entity/Q34753189

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Identifying Novel Targets for Treatment of Liver Fibrosis: What Can We Learn from Injured Tissues which Heal Without a Scar?Alcoholic liver disease: pathogenesis, management, and novel targets for therapy Leaky gut and the liver: a role for bacterial translocation in nonalcoholic steatohepatitis IL-1 receptor antagonist ameliorates inflammasome-dependent alcoholic steatohepatitis in mice Role of innate immunity in the development of hepatocellular carcinoma Alcoholic liver disease: pathogenesis and new therapeutic targets.Chronic alcohol consumption potentiates the development of diabetes through pancreatic β-cell dysfunction MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases.TRIF Differentially Regulates Hepatic Steatosis and Inflammation/Fibrosis in Mice Role of interleukin-22 in liver diseases.Fibrogenesis in alcoholic liver disease.Inhibition of TLR8- and TLR4-induced Type I IFN induction by alcohol is different from its effects on inflammatory cytokine production in monocytes Both bone marrow-derived and non-bone marrow-derived cells contribute to AIM2 and NLRP3 inflammasome activation in a MyD88-dependent manner in dietary steatohepatitis.Toll-like receptor-mediated signaling cascade as a regulator of the inflammation network during alcoholic liver disease.Pathogenesis of alcoholic liver disease: role of oxidative metabolism.Mechanisms of alcohol-mediated hepatotoxicity in human-immunodeficiency-virus-infected patients.Melatonin attenuates the TLR4-mediated inflammatory response through MyD88- and TRIF-dependent signaling pathways in an in vivo model of ovarian cancer Alcohol and inflammation and infection: clinical and experimental systems--summary of the 2010 Alcohol and Immunology Research Interest Group Meeting Hepatoprotective and anti-inflammatory cytokines in alcoholic liver disease Metabolic danger signals, uric acid and ATP, mediate inflammatory cross-talk between hepatocytes and immune cells in alcoholic liver disease.Toll-like receptors in liver fibrosis: cellular crosstalk and mechanisms Immunity and inflammatory signaling in alcoholic liver disease.New Approaches for Studying Alcoholic Liver Disease The intestinal microbiome and the leaky gut as therapeutic targets in alcoholic liver disease.Binge ethanol and liver: new molecular developments Autophagy in alcohol-induced liver diseases.Inflammation in alcoholic liver disease.Differences in innate immune signaling between alcoholic and non-alcoholic steatohepatitis Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME.STING-IRF3 pathway links endoplasmic reticulum stress with hepatocyte apoptosis in early alcoholic liver disease.NOX4 Regulates CCR2 and CCL2 mRNA Stability in Alcoholic Liver Disease.Gut-liver axis and sensing microbes.Hepatic stellate cells and innate immunity in alcoholic liver disease.Role of innate immunity and the microbiota in liver fibrosis: crosstalk between the liver and gut.Role of toll-like receptors in immune activation and tolerance in the liver.Hepatitis C, innate immunity and alcohol: friends or foes?Scoparone Inhibits LPS-Simulated Inflammatory Response by Suppressing IRF3 and ERK in BV-2 Microglial Cells.Inhibition of IRF3 expression reduces TGF-β1-induced proliferation of hepatic stellate cells.Chronic Alcohol Consumption Causes Liver Injury in High-Fructose-Fed Male Mice Through Enhanced Hepatic Inflammatory Response.ADAR1 Prevents Liver Injury from Inflammation and Suppresses Interferon Production in Hepatocytes.

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P2860

Interferon regulatory factor 3 and type I interferons are protective in alcoholic liver injury in mice by way of crosstalk of parenchymal and myeloid cells.

http://www.wikidata.org/entity/Q34753189

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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name

Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells.

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Interferon regulatory factor 3 ...... parenchymal and myeloid cells

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P2093

Angela Dolganiuc

http://www.w3.org/2001/XMLSchema#string

Bharath Nath

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Donna Catalano

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Evelyn Kurt-Jones

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Gyongyi Szabo

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Jan Petrasek

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Karen Kodys

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Timea Csak

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P2860

Essential role of tumor necrosis factor alpha in alcohol-induced liver injury in mice

Interleukin-10: new perspectives on an old cytokine

LPS-TLR4 signaling to IRF-3/7 and NF-kappaB involves the toll adapters TRAM and TRIF

Kupffer cell activation by lipopolysaccharide in rats: role for lipopolysaccharide binding protein and toll-like receptor 4

TLR4 enhances TGF-beta signaling and hepatic fibrosis

Role of Kupffer cells in alcoholic hepatitis.

Endotoxin and Kupffer cell activation in alcoholic liver disease.

Toll-like receptor-4 signaling and Kupffer cells play pivotal roles in the pathogenesis of non-alcoholic steatohepatitis.

Activation of the innate immune system and alcoholic liver disease: effects of ethanol per se or enhanced intestinal translocation of bacterial toxins induced by ethanol?

Kupffer cell heterogeneity: functional properties of bone marrow derived and sessile hepatic macrophages.

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649-660

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scholarly article

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10.1002/HEP.24059

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2

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53

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Pranoti S. Mandrekar

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2011-01-10T00:00:00Z

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49792515

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21274885

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3069538

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