about
Macrophages and Alcohol-Related Liver InflammationCellular stress response and innate immune signaling: integrating pathways in host defense and inflammation.Toll-like receptors in the pathogenesis of alcoholic liver disease.Inhibition of heat shock protein 90 alleviates steatosis and macrophage activation in murine alcoholic liver injury.Interferon regulatory factor 3 and type I interferons are protective in alcoholic liver injury in mice by way of crosstalk of parenchymal and myeloid cells.Epigenetic regulation in alcoholic liver disease.Alcohol and inflammatory responses: summary of the 2013 Alcohol and Immunology Research Interest Group (AIRIG) meeting.Summary of the 2014 Alcohol and Immunology Research Interest Group (AIRIG) meetingOxidative stress and inflammation: essential partners in alcoholic liver disease.An essential role for monocyte chemoattractant protein-1 in alcoholic liver injury: regulation of proinflammatory cytokines and hepatic steatosis in mice.Inhibition of heat shock protein (molecular weight 90 kDa) attenuates proinflammatory cytokines and prevents lipopolysaccharide-induced liver injury in mice.Immunity and inflammatory signaling in alcoholic liver disease.Sexual Dimorphism in Alcohol Induced Adipose Inflammation Relates to Liver Injury.Pattern recognition receptors: a contemporary view on liver diseases.MicroRNA expression profile in Lieber-DeCarli diet-induced alcoholic and methionine choline deficient diet-induced nonalcoholic steatohepatitis models in miceVSL#3 probiotic treatment attenuates fibrosis without changes in steatohepatitis in a diet-induced nonalcoholic steatohepatitis model in mice.A recent perspective on alcohol, immunity, and host defense.Signalling pathways in alcohol-induced liver inflammation.Diverse regulation of NF-kappaB and peroxisome proliferator-activated receptors in murine nonalcoholic fatty liver.Alcoholic hepatitis: Translational approaches to develop targeted therapies.Hepatocyte-specific hypoxia-inducible factor-1α is a determinant of lipid accumulation and liver injury in alcohol-induced steatosis in mice.Identifying molecular targets to improve immune function in alcoholic hepatitis.Hepatitis C virus (HCV) core protein-induced, monocyte-mediated mechanisms of reduced IFN-alpha and plasmacytoid dendritic cell loss in chronic HCV infection.Hepatitis C virus core and nonstructural protein 3 proteins induce pro- and anti-inflammatory cytokines and inhibit dendritic cell differentiation.Inhibition of superantigen-induced T cell proliferation and monocyte IL-1 beta, TNF-alpha, and IL-6 production by acute ethanol treatment.Tacrolimus and cyclosporine A inhibit allostimulatory capacity and cytokine production of human myeloid dendritic cells.Acute alcohol consumption inhibits accessory cell function of monocytes and dendritic cells.Toll-like receptor 2 mediates inflammatory cytokine induction but not sensitization for liver injury by Propioni- bacterium acnes.Inhibition of myeloid dendritic cell accessory cell function and induction of T cell anergy by alcohol correlates with decreased IL-12 production.RSA 2004: combined basic research satellite symposium - session four: hepatitis virus and alcohol interactions in immunity and liver disease.Human monocytes, macrophages, and dendritic cells: alcohol treatment methods.Modulation of non-alcoholic steatohepatitis by pattern recognition receptors in mice: the role of toll-like receptors 2 and 4.Moderate alcohol intake in humans attenuates monocyte inflammatory responses: inhibition of nuclear regulatory factor kappa B and induction of interleukin 10.Cytokines and alcohol.Bone marrow-derived immune cells mediate sensitization to liver injury in a myeloid differentiation factor 88-dependent fashion.Acute alcohol exposure exerts anti-inflammatory effects by inhibiting IkappaB kinase activity and p65 phosphorylation in human monocytes.Acute alcohol activates STAT3, AP-1, and Sp-1 transcription factors via the family of Src kinases to promote IL-10 production in human monocytesCritical role of Toll-like receptors and the common TLR adaptor, MyD88, in induction of granulomas and liver injuryImmunoreactive-FSH in human normal gastric mucosaRegulation of human monocyte functions by acute ethanol treatment: decreased tumor necrosis factor-alpha, interleukin-1 beta and elevated interleukin-10, and transforming growth factor-beta production
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0000-0002-4966-7100