shRNA knockdown of Bmi-1 reveals a critical role for p21-Rb pathway in NSC self-renewal during development. - Wikidata - awgldk - TriplyDB

shRNA knockdown of Bmi-1 reveals a critical role for p21-Rb pathway in NSC self-renewal during development.

shRNA knockdown of Bmi-1 reveals a critical role for p21-Rb pathway in NSC self-renewal during development.

http://www.wikidata.org/entity/Q34764944

about

Identification of a polymorphism in the RING finger of human Bmi-1 that causes its degradation by the ubiquitin-proteasome system Epigenetic choreographers of neurogenesis in the adult mammalian brain Epigenetic regulation of neurogenesis in the adult mammalian brain Signaling mechanisms regulating adult neural stem cells and neurogenesis BMI-1 promotes ewing sarcoma tumorigenicity independent of CDKN2A repression FoxO3 regulates neural stem cell homeostasis Yin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implication Post-Translational Modifications of Histones in Vertebrate Neurogenesis A Novel Aspect of Tumorigenesis-BMI1 Functions in Regulating DNA Damage Response Polycomb Group (PcG) Proteins and Human Cancers: Multifaceted Functions and Therapeutic Implications PRC2 during vertebrate organogenesis: a complex in transition Unlocking epigenetic codes in neurogenesis Histone methylation makes its mark on longevity Targeted disruption in mice of a neural stem cell-maintaining, KRAB-Zn finger-encoding gene that has rapidly evolved in the human lineage Speech sound processing deficits and training-induced neural plasticity in rats with dyslexia gene knockdown Epithelial-mesenchymal transition and cancer stemness: the Twist1-Bmi1 connection A novel KRAB domain-containing zinc finger transcription factor ZNF431 directly represses Patched1 transcription Ezh2, the histone methyltransferase of PRC2, regulates the balance between self-renewal and differentiation in the cerebral cortex A novel function of the proneural factor Ascl1 in progenitor proliferation identified by genome-wide characterization of its targets Coordinated control of self-renewal and differentiation of neural stem cells by Myc and the p19ARF-p53 pathway GFAP-Cre-mediated transgenic activation of Bmi1 results in pituitary tumors Electrical coupling regulates layer 1 interneuron microcircuit formation in the neocortex Bmi1 regulates auditory hair cell survival by maintaining redox balance.The role of microRNAs in glioma initiation and progression.Rare somatic cells from human breast tissue exhibit extensive lineage plasticity Transcriptional Regulation of Arabidopsis Polycomb Repressive Complex 2 Coordinates Cell-Type Proliferation and Differentiation.Bmi-1 regulates autoreactive CD4+ T cell survival in immune thrombocytopenia patients.Role of p16INK4a and BMI-1 in oxidative stress-induced premature senescence in human dental pulp stem cells.Spred1, a negative regulator of Ras-MAPK-ERK, is enriched in CNS germinal zones, dampens NSC proliferation, and maintains ventricular zone structure.p38 MAPK-Mediated Bmi-1 down-regulation and defective proliferation in ATM-deficient neural stem cells can be restored by Akt activation.Transcriptional role of androgen receptor in the expression of long non-coding RNA Sox2OT in neurogenesis.Bmi1 regulates mitochondrial function and the DNA damage response pathway REST interacts with Cbx proteins and regulates polycomb repressive complex 1 occupancy at RE1 elements Down-regulation of Sp1 suppresses cell proliferation, clonogenicity and the expressions of stem cell markers in nasopharyngeal carcinoma.Bmi1 is expressed in postnatal myogenic satellite cells, controls their maintenance and plays an essential role in repeated muscle regeneration Polycomb group proteins: multi-faceted regulators of somatic stem cells and cancer.A small molecule accelerates neuronal differentiation in the adult rat.The Ink4a/Arf locus is a barrier to direct neuronal transdifferentiation The microRNA cluster miR-106b~25 regulates adult neural stem/progenitor cell proliferation and neuronal differentiation.Bmi-1 absence causes premature brain degeneration

P2860

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P2860

shRNA knockdown of Bmi-1 reveals a critical role for p21-Rb pathway in NSC self-renewal during development.

http://www.wikidata.org/entity/Q34764944

description

2007 nî lūn-bûn

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2007 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2007 թվականի հունիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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type

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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prefLabel

shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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J.STEM.2007.04.001

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shRNA knockdown of Bmi-1 revea ...... lf-renewal during development.

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Christopher A Fasano

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Ihor R Lemischka

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John T Dimos

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Natalia B Ivanova

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Natalia Lowry

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Sally Temple

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87-99

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scholarly article

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10.1016/J.STEM.2007.04.001

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1

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1

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2007-06-01T00:00:00Z

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5481969

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18371338

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