Severe cardiomyopathy in mice lacking dystrophin and MyoD
about
Role of telomere dysfunction in cardiac failure in Duchenne muscular dystrophyA comparative study of N-glycolylneuraminic acid (Neu5Gc) and cytotoxic T cell (CT) carbohydrate expression in normal and dystrophin-deficient dog and human skeletal muscleAnimal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapyThe mdx mouse model as a surrogate for Duchenne muscular dystrophyIdentification of gene co-regulatory modules and associated cis-elements involved in degenerative heart diseaseA human-specific deletion in mouse Cmah increases disease severity in the mdx model of Duchenne muscular dystrophyCaspase-12 ablation preserves muscle function in the mdx mouse.Experimental models of duchenne muscular dystrophy: relationship with cardiovascular disease.VBP15, a novel anti-inflammatory and membrane-stabilizer, improves muscular dystrophy without side effects.Secondary coronary artery vasospasm promotes cardiomyopathy progression.Regulation of the calpain and ubiquitin-proteasome systems in a canine model of muscular dystrophy.Cardiac function in muscular dystrophy associates with abdominal muscle pathology.Recombinant adeno-associated viral (rAAV) vectors as therapeutic tools for Duchenne muscular dystrophy (DMD).Challenges and opportunities in dystrophin-deficient cardiomyopathy gene therapy.Full-length dystrophin expression in half of the heart cells ameliorates beta-isoproterenol-induced cardiomyopathy in mdx mice.Cardiac phenotype of Duchenne Muscular Dystrophy: insights from cellular studiesProgress in gene therapy of dystrophic heart disease.Exclusive skeletal muscle correction does not modulate dystrophic heart disease in the aged mdx model of Duchenne cardiomyopathyMicrodystrophin gene therapy of cardiomyopathy restores dystrophin-glycoprotein complex and improves sarcolemma integrity in the mdx mouse heart.Prospect of gene therapy for cardiomyopathy in hereditary muscular dystrophy.Genetic disruption of calcineurin improves skeletal muscle pathology and cardiac disease in a mouse model of limb-girdle muscular dystrophy.Cardiac expression of a mini-dystrophin that normalizes skeletal muscle force only partially restores heart function in aged Mdx mice.Organotypic heart slices for cell transplantation and physiological studies.Annexin A6 modifies muscular dystrophy by mediating sarcolemmal repair.Cardiac and respiratory dysfunction in Duchenne muscular dystrophy and the role of second messengers.The utrophin gene is transcriptionally up-regulated in regenerating muscle.Ablation of cyclase-associated protein 2 (CAP2) leads to cardiomyopathy.Electrocardiographic findings in mdx mice: a cardiac phenotype of Duchenne muscular dystrophy.Dystrophic Cardiomyopathy: Complex Pathobiological Processes to Generate Clinical Phenotype.Humanizing the mdx mouse model of DMD: the long and the short of it.
P2860
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P2860
Severe cardiomyopathy in mice lacking dystrophin and MyoD
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@ast
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@en
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@nl
type
label
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@ast
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@en
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@nl
prefLabel
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@ast
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@en
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@nl
P2093
P2860
P356
P1476
Severe cardiomyopathy in mice lacking dystrophin and MyoD
@en
P2093
P2860
P304
P356
10.1073/PNAS.96.1.220
P407
P577
1999-01-01T00:00:00Z