about
Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophyThe yeast kinome displays scale free topology with functional hub clustersThe beneficial role of proteolysis in skeletal muscle growth and stress adaptationMetacaspase Yca1 is required for clearance of insoluble protein aggregates.Expression of utrophin A mRNA correlates with the oxidative capacity of skeletal muscle fiber types and is regulated by calcineurin/NFAT signalingCD34 promotes satellite cell motility and entry into proliferation to facilitate efficient skeletal muscle regeneration.A non-death role of the yeast metacaspase: Yca1p alters cell cycle dynamics.Reconstructing regulatory kinase pathways from phosphopeptide data: a bioinformatics approach.Strain-dependent myeloid hyperplasia, growth deficiency, and accelerated cell cycle in mice lacking the Rb-related p107 gene.Wnt11 promotes cardiomyocyte development by caspase-mediated suppression of canonical Wnt signalsSevere cardiomyopathy in mice lacking dystrophin and MyoDWnt/β-catenin controls follistatin signalling to regulate satellite cell myogenic potential.Getting to the heart of the matter: exploring opportunities for gene therapy treatment of dystrophic cardiomyopathy.Caspase 3 cleavage of Pax7 inhibits self-renewal of satellite cellsIsolation of phosphoproteins.The non-death role of metacaspase proteases.Evolution of caspase-mediated cell death and differentiation: twins separated at birth.Yeast proteinopathy models: a robust tool for deciphering the basis of neurodegeneration.Cardiotrophin-1 maintains the undifferentiated state in skeletal myoblasts.Temporal activation of XRCC1-mediated DNA repair is essential for muscle differentiation.Cardiotrophin 1 stimulates beneficial myogenic and vascular remodeling of the heart.A novel whole-cell lysate kinase assay identifies substrates of the p38 MAPK in differentiating myoblasts.Parole terms for a killer: directing caspase3/CAD induced DNA strand breaks to coordinate changes in gene expression.Erratum to: The beneficial role of proteolysis in skeletal muscle growth and stress adaptation.Phosphorylation-dependent structural alterations in the small hsp30 chaperone are associated with cellular recovery.Glucocorticoid treatment alleviates dystrophic myofiber pathology by activation of the calcineurin/NF-AT pathway.The post-natal heart contains a myocardial stem cell population.Calcineurin-NFAT signaling, together with GABP and peroxisome PGC-1{alpha}, drives utrophin gene expression at the neuromuscular junction.Caspase signaling, a conserved inductive cue for metazoan cell differentiation.Cell death proteins: an evolutionary role in cellular adaptation before the advent of apoptosis.Regeneration and myogenic cell proliferation correlate with taurine levels in dystrophin- and MyoD-deficient muscles.Quantitative proteomic analysis of dystrophic dog muscle.Expression of murine muscle-enriched A-type lamin-interacting protein (MLIP) is regulated by tissue-specific alternative transcription start sitesThe metacaspase Yca1 maintains proteostasis through multiple interactions with the ubiquitin systemCaspase Cleavage of Gelsolin Is an Inductive Cue for Pathologic Cardiac HypertrophyActivation of JNK1 contributes to dystrophic muscle pathogenesisEpinephrine administration stimulates GLUT4 translocation but reduces glucose transport in muscleMyoD is required for myogenic stem cell function in adult skeletal muscleNeural stem cell differentiation is dependent upon endogenous caspase 3 activityIdentification of candidate regulators of embryonic stem cell differentiation by comparative phosphoprotein affinity profiling
P50
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P50
description
researcher ORCID ID = 0000-0002-6824-8569
@en
wetenschapper
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name
Lynn A Megeney
@ast
Lynn A Megeney
@en
Lynn A Megeney
@es
Lynn A Megeney
@nl
type
label
Lynn A Megeney
@ast
Lynn A Megeney
@en
Lynn A Megeney
@es
Lynn A Megeney
@nl
prefLabel
Lynn A Megeney
@ast
Lynn A Megeney
@en
Lynn A Megeney
@es
Lynn A Megeney
@nl
P106
P1153
6603794381
P31
P496
0000-0002-6824-8569