Regulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter. - Wikidata - awgldk - TriplyDB

Regulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter.

Regulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter.

http://www.wikidata.org/entity/Q34809257

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Neuroimmunomodulation in the Gut: Focus on Inflammatory Bowel Disease The Neuromodulation of the Intestinal Immune System and Its Relevance in Inflammatory Bowel Disease Interleukin-1beta upregulates functional expression of neurokinin-1 receptor (NK-1R) via NF-kappaB in astrocytes.Substance P modulates colitis-associated fibrosis Metalloproteinases and transforming growth factor-alpha mediate substance P-induced mitogen-activated protein kinase activation and proliferation in human colonocytes.Full-length and truncated neurokinin-1 receptor expression and function during monocyte/macrophage differentiation.Induction of colitis causes inflammatory responses in fat depots: evidence for substance P pathways in human mesenteric preadipocytes Expression of functional NK1 receptors in human alveolar macrophages: superoxide anion production, cytokine release and involvement of NF-kappaB pathway.Autocrine hemokinin-1 functions as an endogenous adjuvant for IgE-mediated mast cell inflammatory responses Substance P mediates pro-inflammatory cytokine release form mesenteric adipocytes in Inflammatory Bowel Disease patients Immunomodulatory properties of substance P: the gastrointestinal system as a model.Role of neuropeptides in inflammatory bowel disease.Melanin-concentrating hormone as a mediator of intestinal inflammation.Interleukin-12 (IL-12) and IL-23 induction of substance p synthesis in murine T cells and macrophages is subject to IL-10 and transforming growth factor beta regulation.Neurokinin-1 receptor mRNA expression differences in brains of HIV-infected individuals Substance P stimulates cyclooxygenase-2 and prostaglandin E2 expression through JAK-STAT activation in human colonic epithelial cells.Substance P (SP) enhances CCL5-induced chemotaxis and intracellular signaling in human monocytes, which express the truncated neurokinin-1 receptor (NK1R)Effect of Corneal Nerve Ablation on Immune Tolerance Induced by Corneal Allografts, Oral Immunization, or Anterior Chamber Injection of Antigens.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Tachykinins and Neurokinin Receptors in Bone Marrow Functions: Neural-Hematopoietic Link.Substance P and the regulation of inflammation in infections and inflammatory bowel disease.Targeting tachykinin receptors for the treatment of functional gastrointestinal disorders with a focus on irritable bowel syndrome.Gastrointestinal neuroendocrine peptides/amines in inflammatory bowel disease.The Role of NFkB in Drug Addiction: Beyond Inflammation.Neuropeptide substance P and the immune response.Reciprocal Regulation of Substance P and IL-12/IL-23 and the Associated Cytokines, IFNγ/IL-17: A Perspective on the Relevance of This Interaction to Multiple Sclerosis.A novel activity for substance P: stimulation of peroxisome proliferator-activated receptor-gamma protein expression in human monocytes and macrophages.Role of Spinal Microglia in Visceral Hyperalgesia and NK1R Up-Regulation in a Rat Model of Chronic Stress (Gastroenterology 2009;136:1339-1348).Cytokine upregulation of proteinase-activated-receptors 2 and 4 expression mediated by p38 MAP kinase and inhibitory kappa B kinase beta in human endothelial cells.Role of spinal microglia in visceral hyperalgesia and NK1R up-regulation in a rat model of chronic stress Increased expression of 5-HT3 and NK 1 receptors in 5-fluorouracil-induced mucositis in mouse jejunum.Role of G protein-coupled receptors-microRNA interactions in gastrointestinal pathophysiology.Induction of Contrasuppressor Cells and Loss of Immune Privilege Produced by Corneal Nerve Ablation

P2860

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P2860

Regulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter.

http://www.wikidata.org/entity/Q34809257

description

2003 nî lūn-bûn

@nan

2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի փետրվարին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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type

label

Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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prefLabel

Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

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Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

@en

Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

@nl

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PNAS.0530112100

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Regulation of the NK-1 recepto ...... -kappa B site on its promoter.

@en

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Amy Pan

http://www.w3.org/2001/XMLSchema#string

Andreas Mykoniatis

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Asia Pasha

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Charalabos Pothoulakis

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Chi-Chi Wang

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Dezheng Zhao

http://www.w3.org/2001/XMLSchema#string

Ignazio Castagliuolo

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Jennifer Liu

http://www.w3.org/2001/XMLSchema#string

Leyla Valenick

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Simos Simeonidis

http://www.w3.org/2001/XMLSchema#string

P2860

The NF-kappa B and I kappa B proteins: new discoveries and insights

Cytokine response by human monocytes to Clostridium difficile toxin A and toxin B

The primary structure and gene organization of human substance P and neuromedin K receptors

Missing pieces in the NF-kappaB puzzle

NF-kappa B: a lesson in family values

Clostridium difficile toxin A induces the release of neutrophil chemotactic factors from rat peritoneal macrophages: role of interleukin-1beta, tumor necrosis factor alpha, and leukotrienes.

Fecal lactoferrin, interleukin-1beta, and interleukin-8 are elevated in patients with severe Clostridium difficile colitis

Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease.

The neuropeptide substance P activates p38 mitogen-activated protein kinase resulting in IL-6 expression independently from NF-kappa B.

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2957-2962

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10.1073/PNAS.0530112100

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5

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100

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10894148

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12594338

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151448

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