Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease.
about
Neuroimmunomodulation in the Gut: Focus on Inflammatory Bowel DiseaseTachykinin receptors and tachykinin receptor antagonistsHuman colonic anti-secretory activity of the potent NK(1) antagonist, SR140333: assessment of potential anti-diarrhoeal activity in food allergy and inflammatory bowel diseaseSubstance P regulates migration in rat intestinal epithelial cellsSubstance P-evoked Cl(-) secretion in guinea pig distal colonic epithelia: interaction with PGE(2).Tachykinins potently stimulate human small bowel blood flow: a laser Doppler flowmetry study in humans.Motilin receptor density in inflamed and noninflamed tissue in rabbit TNBS-induced colitis.Gene expression profiling of inflammatory bladder disorders.Targeted deletion of neuropeptide Y (NPY) modulates experimental colitisEnhanced immunoglobulin A response and protection against Salmonella enterica serovar typhimurium in the absence of the substance P receptor.Substance P up-regulates macrophage inflammatory protein-1beta expression in human T lymphocytesInterleukin-1beta upregulates functional expression of neurokinin-1 receptor (NK-1R) via NF-kappaB in astrocytes.Neurotensin is a proinflammatory neuropeptide in colonic inflammationDifferential expression of neurokinin-1 receptor by human mucosal and peripheral lymphoid cellsA lay doctor's guide to the inflammatory process in the gastrointestinal tractNeurokinin-1 receptor: functional significance in the immune system in reference to selected infections and inflammation.Neurotrophic and neuroimmune responses to early-life Pseudomonas aeruginosa infection in rat lungs.Substance P receptor binding sites are expressed by glia in vivo after neuronal injury.Neuropeptide neurotensin stimulates intestinal wound healing following chronic intestinal inflammation.Measurement of plasma-derived substance P: biological, methodological, and statistical considerations.Induction of colitis causes inflammatory responses in fat depots: evidence for substance P pathways in human mesenteric preadipocytesEffects of substance P on identified neurons of the rat dorsal motor nucleus of the vagusRegulation of the NK-1 receptor gene expression in human macrophage cells via an NF-kappa B site on its promoter.Characterization of the [125I]-neurokinin A binding site in the circular muscle of human colonProtective effects of neurokinin-1 receptor during colitis in mice: role of the epidermal growth factor receptor.Chronic pancreatitis: the perspective of pain generation by neuroimmune interaction.Effect of inflammation of enteric nerves. Cytokine-induced changes in neurotransmitter content and release.Substance P and calcitonin gene-related peptide (CGRP) in gastrointestinal inflammation.Role of substance P in inflammatory arthritis.Neurokinin-1 receptor expression in inflammatory bowel disease: molecular quantitation and localisation.Expression of interleukin 8 (IL-8) and substance P in human chronic pancreatitisNeuropeptides, mesenteric fat, and intestinal inflammation.Gastrin releasing peptide receptor expression is decreased in patients with Crohn's disease but not in ulcerative colitis.Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance PSubstance P (neurokinin-1) and neurokinin A (neurokinin-2) receptor gene and protein expression in the healthy and inflamed human intestineFunctional consequences of alteration of N-linked glycosylation sites on the neurokinin 1 receptor.Irritable bowel syndrome: methods, mechanisms, and pathophysiology. Neural and neuro-immune mechanisms of visceral hypersensitivity in irritable bowel syndrome.A substance P antagonist, RP-67,580, ameliorates a mouse meningoencephalitic response to Trypanosoma brucei bruceiIncreased substance P responses in dorsal root ganglia and intestinal macrophages during Clostridium difficile toxin A enteritis in rats.Immunomodulatory properties of substance P: the gastrointestinal system as a model.
P2860
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P2860
Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease.
description
1988 nî lūn-bûn
@nan
1988 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@ast
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@en
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@nl
type
label
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@ast
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@en
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@nl
prefLabel
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@ast
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@en
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@nl
P2093
P2860
P356
P1476
Receptor binding sites for sub ...... ive colitis and Crohn disease.
@en
P2093
C R Mantyh
E P Passaro
J E Maggio
M L Welton
P W Mantyh
R P Zimmerman
P2860
P304
P356
10.1073/PNAS.85.9.3235
P407
P577
1988-05-01T00:00:00Z