Myxoma virus protein M029 is a dual function immunomodulator that inhibits PKR and also conscripts RHA/DHX9 to promote expanded host tropism and viral replication - Wikidata - awgldk - TriplyDB

Myxoma virus protein M029 is a dual function immunomodulator that inhibits PKR and also conscripts RHA/DHX9 to promote expanded host tropism and viral replication

Myxoma virus protein M029 is a dual function immunomodulator that inhibits PKR and also conscripts RHA/DHX9 to promote expanded host tropism and viral replication

http://www.wikidata.org/entity/Q34819569

about

Stress Beyond Translation: Poxviruses and More The biology of DHX9 and its potential as a therapeutic target Therapeutics for Graft-versus-Host Disease: From Conventional Therapies to Novel Virotherapeutic Strategies Poxviruses and the evolution of host range and virulence Adaptive gene amplification as an intermediate step in the expansion of virus host range.Myxoma virus and the Leporipoxviruses: an evolutionary paradigm.Functional analysis of the short isoform of orf virus protein OV20.0.Ectopic expression of vaccinia virus E3 and K3 cannot rescue ectromelia virus replication in rabbit RK13 cells Evasion of the Innate Immune Type I Interferon System by Monkeypox Virus.Myxoma virus M156 is a specific inhibitor of rabbit PKR but contains a loss-of-function mutation in Australian virus isolates Genome scale evolution of myxoma virus reveals host-pathogen adaptation and rapid geographic spread.Myxoma Virus dsRNA Binding Protein M029 Inhibits the Type I IFN-Induced Antiviral State in a Highly Species-Specific Fashion.Activation of the PKR/eIF2α signaling cascade inhibits replication of Newcastle disease virus Ectromelia virus accumulates less double-stranded RNA compared to vaccinia virus in BS-C-1 cells.Poxviruses Utilize Multiple Strategies to Inhibit Apoptosis.Reverse engineering field isolates of myxoma virus demonstrates that some gene disruptions or loss of function do not explain virulence changes observed in the field.AC dielectrophoretic manipulation and electroporation of vaccinia virus using carbon nanoelectrode arrays.Evolution of the DEAD box helicase family in chicken: chickens have no DHX9 ortholog.Recombinant myxoma virus lacking all poxvirus ankyrin-repeat proteins stimulates multiple cellular anti-viral pathways and exhibits a severe decrease in virulence Evolutionary clash between myxoma virus and rabbit PKR in Australia.Identification of host DEAD-box RNA helicases that regulate cellular tropism of oncolytic Myxoma virus in human cancer cells.Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox.Poxviral protein E3-altered cytokine production reveals that DExD/H-box helicase 9 controls Toll-like receptor-stimulated immune responses

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Myxoma virus protein M029 is a dual function immunomodulator that inhibits PKR and also conscripts RHA/DHX9 to promote expanded host tropism and viral replication

http://www.wikidata.org/entity/Q34819569

description

2013 nî lūn-bûn

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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

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Jia Liu

http://www.w3.org/2001/XMLSchema#string

Masmudur M Rahman

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Stefan Rothenburg

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Winnie M Chan

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P2860

Domain structure of human nuclear DNA helicase II (RNA helicase A)

Post-translational modification of the myxoma-virus anti-inflammatory serpin SERP-1 by a virally encoded sialyltransferase

The E3L gene of vaccinia virus encodes an inhibitor of the interferon-induced, double-stranded RNA-dependent protein kinase

The E3L and K3L vaccinia virus gene products stimulate translation through inhibition of the double-stranded RNA-dependent protein kinase by different mechanisms

Analysis of vaccinia virus-host protein-protein interactions: validations of yeast two-hybrid screenings

A role for Z-DNA binding in vaccinia virus pathogenesis

Host-range restriction of vaccinia virus E3L deletion mutant can be overcome in vitro, but not in vivo, by expression of the influenza virus NS1 protein

Nuclear factors are involved in hepatitis C virus RNA replication

IRF3 and IRF7 phosphorylation in virus-infected cells does not require double-stranded RNA-dependent protein kinase R or Ikappa B kinase but is blocked by Vaccinia virus E3L protein

Co-regulation of NF-kappaB and inflammasome-mediated inflammatory responses by myxoma virus pyrin domain-containing protein M013

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249322249

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