Naturally occurring single amino acid replacements in a regulatory protein alter streptococcal gene expression and virulence in mice.
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Unique Footprint in the scl1.3 Locus Affects Adhesion and Biofilm Formation of the Invasive M3-Type Group A StreptococcusNatural variant of collagen-like protein a in serotype M3 group a Streptococcus increases adherence and decreases invasive potentialDistinct single amino acid replacements in the control of virulence regulator protein differentially impact streptococcal pathogenesisDual-site phosphorylation of the control of virulence regulator impacts group a streptococcal global gene expression and pathogenesisRapid Emergence of a New Clone Impacts the Population at Risk and Increases the Incidence of Type emm89 Group A Streptococcus Invasive Disease.From transcription to activation: how group A streptococcus, the flesh-eating pathogen, regulates SpeB cysteine protease production.Asymptomatic carriage of group A streptococcus is associated with elimination of capsule production.Transcriptional regulation of a bacteriophage encoded extracellular DNase (Spd-3) by Rgg in Streptococcus pyogenes.Adhesin competence repressor (AdcR) from Streptococcus pyogenes controls adaptive responses to zinc limitation and contributes to virulence.A naturally occurring single amino acid replacement in multiple gene regulator of group A Streptococcus significantly increases virulenceCharacterization of the effect of the histidine kinase CovS on response regulator phosphorylation in group A Streptococcus.Mutations in the control of virulence sensor gene from Streptococcus pyogenes after infection in mice lead to clonal bacterial variants with altered gene regulatory activity and virulence.Plant exomics: concepts, applications and methodologies in crop improvement.Phosphorylation events in the multiple gene regulator of group A Streptococcus significantly influence global gene expression and virulenceSerotype M3 and M28 Group A Streptococci Have Distinct Capacities to Evade Neutrophil and TNF-α Responses and to Invade Soft Tissues.Polymorphisms in regulator of protease B (RopB) alter disease phenotype and strain virulence of serotype M3 group A StreptococcusAllele-dependent differences in quorum-sensing dynamics result in variant expression of virulence genes in Staphylococcus aureusAn amino-terminal signal peptide of Vfr protein negatively influences RopB-dependent SpeB expression and attenuates virulence in Streptococcus pyogenes.Contemporary Pharyngeal and Invasive emm1 and Invasive emm12 Group A Streptococcus Isolates Exhibit Similar In Vivo Selection for CovRS Mutants in Mice.The Mga Regulon but Not Deoxyribonuclease Sda1 of Invasive M1T1 Group A Streptococcus Contributes to In Vivo Selection of CovRS Mutations and Resistance to Innate Immune Killing Mechanisms.Structural and functional analysis of RopB: a major virulence regulator in Streptococcus pyogenes.Application of Whole-Genome Sequencing to an Unusual Outbreak of Invasive Group A Streptococcal Disease.Enterococcal Rgg-like regulator ElrR activates expression of the elrA operon.Transcriptome Remodeling Contributes to Epidemic Disease Caused by the Human Pathogen Streptococcus pyogenes.Structural Mechanisms of Peptide Recognition and Allosteric Modulation of Gene Regulation by the RRNPP Family of Quorum-Sensing Regulators.Natural variation in the promoter of the gene encoding the Mga regulator alters host-pathogen interactions in group a Streptococcus carrier strains.Point mutations in the major outer membrane protein drive hypervirulence of a rapidly expanding clone of Campylobacter jejuni.Genomic Landscape of Intrahost Variation in Group A Streptococcus: Repeated and Abundant Mutational Inactivation of the fabT Gene Encoding a Regulator of Fatty Acid SynthesisThe sagA/pel locus does not regulate the expression of the M protein of the M1T1 lineage of group A Streptococcus.Natural disruption of two regulatory networks in serotype M3 group A Streptococcus isolates contributes to the virulence factor profile of this hypervirulent serotype.Quorum sensing in group A StreptococcusUse of a Phosphorylation Site Mutant To Identify Distinct Modes of Gene Repression by the Control of Virulence Regulator (CovR) in Streptococcus pyogenes.The majority of 9,729 group A streptococcus strains causing disease secrete SpeB cysteine protease: pathogenesis implications.Group A streptococcal cysteine protease cleaves epithelial junctions and contributes to bacterial translocation.Consequences of the variability of the CovRS and RopB regulators among Streptococcus pyogenes causing human infections.The Variable Regions of Lactobacillus rhamnosus Genomes Reveal the Dynamic Evolution of Metabolic and Host-Adaptation Repertoires.The prince and the pauper: which one is real? The problem of secondary mutation during mutagenesis in Streptococcus pyogenes.Streptococcal Cysteine Protease-Mediated Cleavage of Desmogleins Is Involved in the Pathogenesis of Cutaneous Infection.Signaling by a Conserved Quorum Sensing Pathway Contributes to Growth Ex Vivo and Oropharyngeal Colonization of Human Pathogen Group A Streptococcus.Causing Skin and Soft Tissue Infections Are Enriched in the Recently Emerged 89 Clade 3 and Are Not Associated With Abrogation of CovRS
P2860
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P2860
Naturally occurring single amino acid replacements in a regulatory protein alter streptococcal gene expression and virulence in mice.
description
2011 nî lūn-bûn
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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Naturally occurring single ami ...... ression and virulence in mice.
@ast
Naturally occurring single ami ...... ression and virulence in mice.
@en
Naturally occurring single ami ...... ression and virulence in mice.
@nl
type
label
Naturally occurring single ami ...... ression and virulence in mice.
@ast
Naturally occurring single ami ...... ression and virulence in mice.
@en
Naturally occurring single ami ...... ression and virulence in mice.
@nl
prefLabel
Naturally occurring single ami ...... ression and virulence in mice.
@ast
Naturally occurring single ami ...... ression and virulence in mice.
@en
Naturally occurring single ami ...... ression and virulence in mice.
@nl
P2093
P2860
P50
P356
P1476
Naturally occurring single ami ...... pression and virulence in mice
@en
P2093
Bryce Suber
Muthiah Kumaraswami
Pranoti Sahasrabhojane
Ronan K Carroll
Samuel A Shelburne
P2860
P304
P356
10.1172/JCI45169
P407
P577
2011-04-01T00:00:00Z