Cerebral amyloid angiopathy increases susceptibility to infarction after focal cerebral ischemia in Tg2576 mice - Wikidata - awgldk - TriplyDB

Cerebral amyloid angiopathy increases susceptibility to infarction after focal cerebral ischemia in Tg2576 mice

Cerebral amyloid angiopathy increases susceptibility to infarction after focal cerebral ischemia in Tg2576 mice

http://www.wikidata.org/entity/Q34889214

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Cerebral Small Vessel Disease: Targeting Oxidative Stress as a Novel Therapeutic Strategy?Ischemic brain injury in cerebral amyloid angiopathy Heparan sulfate proteoglycans mediate Aβ-induced oxidative stress and hypercontractility in cultured vascular smooth muscle cells Neurovascular Specifications in the Alzheimer-Like Brain of Mice Affected by Focal Cerebral Ischemia: Implications for Future Therapies.Cerebral blood flow regulation and neurovascular dysfunction in Alzheimer disease.The effect of Scutellaria baicalensis stem-leaf flavonoids on spatial learning and memory in chronic cerebral ischemia-induced vascular dementia of rats.Physiologic Reelin does not play a strong role in protection against acute stroke.Identification of a peptide recognizing cerebrovascular changes in mouse models of Alzheimer's disease.Mouse models of neurodegenerative disease: preclinical imaging and neurovascular component.

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P2860

Cerebral amyloid angiopathy increases susceptibility to infarction after focal cerebral ischemia in Tg2576 mice

http://www.wikidata.org/entity/Q34889214

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cerebral amyloid angiopathy in ...... rebral ischemia in Tg2576 mice

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Cerebral amyloid angiopathy in ...... rebral ischemia in Tg2576 mice

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STROKEAHA.114.006078

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Cerebral amyloid angiopathy in ...... rebral ischemia in Tg2576 mice

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P2093

Ananth K Vellimana

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Andrew W Johnson

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Byung Hee Han

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Eric Milner

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Jacob K Greenberg

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Meng-Liang Zhou

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Structural and functional disruption of vascular smooth muscle cells in a transgenic mouse model of amyloid angiopathy

Neurovascular regulation in the normal brain and in Alzheimer's disease

Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein.

Beta-amyloid precursor protein transgenic mice that harbor diffuse A beta deposits but do not form plaques show increased ischemic vulnerability: role of inflammation

Age-dependent changes in brain, CSF, and plasma amyloid (beta) protein in the Tg2576 transgenic mouse model of Alzheimer's disease.

Vasoactive effects of A beta in isolated human cerebrovessels and in a transgenic mouse model of Alzheimer's disease: role of inflammation.

Age-dependent cerebrovascular dysfunction in a transgenic mouse model of cerebral amyloid angiopathy.

Biochemical and immunohistochemical analysis of an Alzheimer's disease mouse model reveals the presence of multiple cerebral Abeta assembly forms throughout life.

Soluble amyloid-beta, effect on cerebral arteriolar regulation and vascular cells

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3064-3069

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scholarly article

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10.1161/STROKEAHA.114.006078

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10

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45

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David M. Holtzman

Gregory J Zipfel

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2014-09-04T00:00:00Z

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25190447

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cerebral infarction

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4288816

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