c-Jun NH(2)-terminal kinase is essential for the regulation of AP-1 by tumor necrosis factor. - Wikidata - awgldk - TriplyDB

c-Jun NH(2)-terminal kinase is essential for the regulation of AP-1 by tumor necrosis factor.

c-Jun NH(2)-terminal kinase is essential for the regulation of AP-1 by tumor necrosis factor.

http://www.wikidata.org/entity/Q34889343

about

Mutual regulation of c-Jun and ATF2 by transcriptional activation and subcellular localization Identification of PCTA, a TGIF antagonist that promotes PML function in TGF-beta signalling JNK Signaling: Regulation and Functions Based on Complex Protein-Protein Partnerships c-Jun NH2-terminal kinase is required for lineage-specific differentiation but not stem cell self-renewal.The protective action of scutellarin against immunological liver injury induced by concanavalin A and its effect on pro-inflammatory cytokines in mice.Oxidative stress-induced JNK activation contributes to proinflammatory phenotype of aging diabetic mesangial cells.AP-1 (Fra-1/c-Jun)-mediated induction of expression of matrix metalloproteinase-2 is required for 15S-hydroxyeicosatetraenoic acid-induced angiogenesis.TNF and MAP kinase signalling pathways.Regulation of human CYP2C9 expression by electrophilic stress involves activator protein 1 activation and DNA looping.Activation of the Jun N-terminal kinase pathway by friend spleen focus-forming virus and its role in the growth and survival of friend virus-induced erythroleukemia cells.c-Jun N-terminal kinase phosphorylation of stathmin confers protection against cellular stress The dual roles of c-Jun NH2-terminal kinase signaling in Cr(VI)-induced apoptosis in JB6 cells The role of JNK in the development of hepatocellular carcinoma.Requirement of c-Jun NH(2)-terminal kinase for Ras-initiated tumor formation.Novel role for aldose reductase in mediating acute inflammatory responses in the lung.Transforming growth factor-beta-activated kinase 1 regulates natural killer cell-mediated cytotoxicity and cytokine production.Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases.JAB1 determines the response of rheumatoid arthritis synovial fibroblasts to tumor necrosis factor-alpha.JNK initiates a cytokine cascade that causes Pax2 expression and closure of the optic fissure.JNK/AP-1 pathway is involved in tumor necrosis factor-alpha induced expression of vascular endothelial growth factor in MCF7 cells From cellular receptors to transduction-transcription pathways for cytokines: at which level should the inhibition be targeted in inflammation?c-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.Distribution of NF-kappaB-binding sites across human chromosome 22 c-Jun controls histone modifications, NF-kappaB recruitment, and RNA polymerase II function to activate the ccl2 gene.TRAF2 exerts opposing effects on basal and TNFα-induced activation of the classic IKK complex in hematopoietic cells in mice c-Jun N-terminal kinases as potential therapeutic targets.ATF2 on the double - activating transcription factor and DNA damage response protein.Suppressive activity of tiotropium bromide on matrix metalloproteinase production from lung fibroblasts in vitro.TNFR1 signaling kinetics: spatiotemporal control of three phases of IKK activation by posttranslational modification.Diet-induced obesity mediated by the JNK/DIO2 signal transduction pathway Miz1 is a signal- and pathway-specific modulator or regulator (SMOR) that suppresses TNF-alpha-induced JNK1 activation Induction of hepatitis by JNK-mediated expression of TNF-alpha.An initial phase of JNK activation inhibits cell death early in the endoplasmic reticulum stress response.The c-Jun NH2-terminal kinase is essential for epidermal growth factor expression during epidermal morphogenesis.Role of Jnk1 in development of neural precursors revealed by iPSC modeling.JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species.Functional analysis of proteins and protein species using shotgun proteomics and linear mathematics.Mammalian MAPK signal transduction pathways activated by stress and inflammation: a 10-year update.The role of the c-Jun N-terminal Kinase signaling pathway in skin cancer.TNF-α signaling in Fanconi anemia.

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P2860

c-Jun NH(2)-terminal kinase is essential for the regulation of AP-1 by tumor necrosis factor.

http://www.wikidata.org/entity/Q34889343

description

2003 nî lūn-bûn

@nan

2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի ապրիլին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

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2003年论文

@wuu

name

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

@ast

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

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type

label

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

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c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

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prefLabel

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

@ast

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor.

@en

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P356

MCB.23.8.2871-2882.2003

P1433

Molecular and Cellular Biology

P1476

c-Jun NH(2)-terminal kinase is ...... AP-1 by tumor necrosis factor

@en

P2093

Jennifer A Lamb

http://www.w3.org/2001/XMLSchema#string

Norman J Kennedy

http://www.w3.org/2001/XMLSchema#string

Richard A Flavell

http://www.w3.org/2001/XMLSchema#string

Roger J Davis

http://www.w3.org/2001/XMLSchema#string

P2860

Signal transduction by tumor necrosis factor mediated by JNK protein kinases

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

Selective interaction of JNK protein kinase isoforms with transcription factors

The p38/RK mitogen-activated protein kinase pathway regulates interleukin-6 synthesis response to tumor necrosis factor

ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents

ATF-2 contains a phosphorylation-dependent transcriptional activation domain

MKK7 is an essential component of the JNK signal transduction pathway activated by proinflammatory cytokines

MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway

Signal transduction by the JNK group of MAP kinases

TNF-R1 signaling: a beautiful pathway

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2871-2882

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scholarly article

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10.1128/MCB.23.8.2871-2882.2003

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8

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23

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Juan-Jose Ventura

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2003-04-01T00:00:00Z

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12665585

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152565

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