about
c-Jun NH(2)-terminal kinase is essential for the regulation of AP-1 by tumor necrosis factor.p38α negatively regulates survival and malignant selection of transformed bronchioalveolar stem cellsMechanism of p38 MAP kinase activation in vivo.Protein kinases and phosphatases as therapeutic targets in cancer.The death domain kinase RIP1 is essential for tumor necrosis factor alpha signaling to p38 mitogen-activated protein kinaseC3G knock-down enhances migration and invasion by increasing Rap1-mediated p38α activation, while it impairs tumor growth through p38α-independent mechanisms.JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species.The many faces of p38 mitogen-activated protein kinase in progenitor/stem cell differentiation.A paracrine network regulates the cross-talk between human lung stem cells and the stroma.Could Sox2 be a useful target to treat lung squamous cell carcinoma?Regulation of human lung alveolar multipotent cells by a novel p38α MAPK/miR-17-92 axis.RNA-sequencing-based comparative analysis of human hepatic progenitor cells and their niche from alcoholic steatohepatitis livers.EGF blocks NADPH oxidase activation by TGF-beta in fetal rat hepatocytes, impairing oxidative stress, and cell death.miR-17-92/p38α Dysregulation Enhances Wnt Signaling and Selects Lgr6+ Cancer Stem-like Cells during Lung Adenocarcinoma Progression.Epidermal Growth Factor Impairs the Cytochrome C/Caspase-3 Apoptotic Pathway Induced by Transforming Growth Factorβ in Rat Fetal Hepatocytes Via a Phosphoinositide 3-Kinase–Dependent PathwayGlucocorticoid receptor down-Regulates c-jun amino terminal kinases induced by tumor necrosis factor ? in fetal rat hepatocyte primary culturesp38α MAP kinase is essential in lung stem and progenitor cell proliferation and differentiationp38 mitogen-activated protein kinase mediates tumor necrosis factor-alpha-induced apoptosis in rat fetal brown adipocytesJNK2 is a positive regulator of the cJun transcription factorChemical genetic analysis of the time course of signal transduction by JNKIsolation, culture, and potentiality assessment of lung alveolar stem cellsLgr6: From Stemness to Cancer Progression
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description
researcher ORCID ID = 0000-0002-9252-843X
@en
wetenschapper
@nl
name
Juan-Jose Ventura
@ast
Juan-Jose Ventura
@en
Juan-Jose Ventura
@es
Juan-Jose Ventura
@nl
type
label
Juan-Jose Ventura
@ast
Juan-Jose Ventura
@en
Juan-Jose Ventura
@es
Juan-Jose Ventura
@nl
prefLabel
Juan-Jose Ventura
@ast
Juan-Jose Ventura
@en
Juan-Jose Ventura
@es
Juan-Jose Ventura
@nl
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P106
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7102389035
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P31
P496
0000-0002-9252-843X