Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation - Wikidata - awgldk - TriplyDB

Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation

Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation

http://www.wikidata.org/entity/Q34919012

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Enhancing CNS repair in neurological disease: challenges arising from neurodegeneration and rewiring of the network Molecular dissection of reactive astrogliosis and glial scar formation Oligodendrocyte, Astrocyte, and Microglia Crosstalk in Myelin Development, Damage, and Repair Astrocytes in Oligodendrocyte Lineage Development and White Matter Pathology Remyelination Therapy in Multiple Sclerosis Implications of glial nitric oxide in neurodegenerative diseases Glial development: the crossroads of regeneration and repair in the CNS Innate immunity in the central nervous system Astrocytes: biology and pathology Understanding the NG2 Glial Scar after Spinal Cord Injury The astrocyte in multiple sclerosis revisited Voltage-gated sodium channel Nav 1.5 contributes to astrogliosis in an in vitro model of glial injury via reverse Na+ /Ca2+ exchange.Shiga toxin 1-induced inflammatory response in lipopolysaccharide-sensitized astrocytes is mediated by endogenous tumor necrosis factor alpha.Structural remodeling of fibrous astrocytes after axonal injury.IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system Trafficking of immune cells in the central nervous system Transgenic inhibition of astroglial NF-kappa B leads to increased axonal sparing and sprouting following spinal cord injury.Neuronal I kappa B kinase beta protects mice from autoimmune encephalomyelitis by mediating neuroprotective and immunosuppressive effects in the central nervous system.Estradiol inhibits ongoing autoimmune neuroinflammation and NFkappaB-dependent CCL2 expression in reactive astrocytes NF-κB in the mechanism of brain edema in acute liver failure: studies in transgenic mice.IL7Rα contributes to experimental autoimmune encephalomyelitis through altered T cell responses and nonhematopoietic cell lineages Administration of simvastatin after kainic acid-induced status epilepticus restrains chronic temporal lobe epilepsy.Interleukin-17 (IL-17)-induced microRNA 873 (miR-873) contributes to the pathogenesis of experimental autoimmune encephalomyelitis by targeting A20 ubiquitin-editing enzyme.IFN-γ signaling to astrocytes protects from autoimmune mediated neurological disability The contribution of immune and glial cell types in experimental autoimmune encephalomyelitis and multiple sclerosis Astroglial inhibition of NF-κB does not ameliorate disease onset and progression in a mouse model for amyotrophic lateral sclerosis (ALS)CCAAT/enhancer binding protein β expression is increased in the brain during HIV-1-infection and contributes to regulation of astrocyte tissue inhibitor of metalloproteinase-1.Astrogliosis.Astrocyte CCL2 sustains immune cell infiltration in chronic experimental autoimmune encephalomyelitis.Raloxifene suppresses experimental autoimmune encephalomyelitis and NF-κB-dependent CCL20 expression in reactive astrocytes.Inhibition of soluble tumour necrosis factor is therapeutic in experimental autoimmune encephalomyelitis and promotes axon preservation and remyelination Calpain inhibition attenuates apoptosis of retinal ganglion cells in acute optic neuritis.Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis.Transcript analysis of laser capture microdissected white matter astrocytes and higher phenol sulfotransferase 1A1 expression during autoimmune neuroinflammation.Octreotide Protects the Mouse Retina against Ischemic Reperfusion Injury through Regulation of Antioxidation and Activation of NF-κB Stimulation of the neurotrophin receptor TrkB on astrocytes drives nitric oxide production and neurodegeneration Astroglial NF-κB mediates oxidative stress by regulation of NADPH oxidase in a model of retinal ischemia reperfusion injury.Neurological diseases as primary gliopathies: a reassessment of neurocentrism Reduced astrocytic NF-κB activation by laquinimod protects from cuprizone-induced demyelination.A model of Periventricular Leukomalacia (PVL) in neonate mice with histopathological and neurodevelopmental outcomes mimicking human PVL in neonates

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Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammation

http://www.wikidata.org/entity/Q34919012

description

2009 nî lūn-bûn

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2009 թուականի Մարտին հրատարակուած գիտական յօդուած

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2009 թվականի մարտին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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Transgenic inhibition of astro ...... al nervous system inflammation

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JIMMUNOL.0802954

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Journal of Immunology

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Transgenic inhibition of astro ...... al nervous system inflammation

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Galina Dvoriantchikova

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John R Bethea

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Roberta Brambilla

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Scott R Barnum

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Shaffiat Karmally

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Trikaldarshi Persaud

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Valery I Shestopalov

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Xianchen Hu

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P2860

Increased expression of ICAM-1, VCAM-1, MCP-1, and MIP-1 alpha by spinal perivascular macrophages during experimental allergic encephalomyelitis in rats.

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Normalization of cDNA microarray data

Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

NF-kappaB and the immune response

Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

Axonal and neuronal degeneration in multiple sclerosis: mechanisms and functional consequences.

Calcineurin triggers reactive/inflammatory processes in astrocytes and is upregulated in aging and Alzheimer's models

Chemokines and chemokine receptors in inflammation of the nervous system: manifold roles and exquisite regulation.

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2628-2640

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10.4049/JIMMUNOL.0802954

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5

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182

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Lubov Nathanson

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2009-03-01T00:00:00Z

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24031962

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19234157

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experimental autoimmune encephalomyelitis

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4291126

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