IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system - Wikidata - awgldk - TriplyDB

IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system

IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system

http://www.wikidata.org/entity/Q33749325

about

Astrocytes in Oligodendrocyte Lineage Development and White Matter Pathology Transcriptional Control of Synaptic Plasticity by Transcription Factor NF-κB Role of microglia in CNS autoimmunity Multiple sclerosis: molecular mechanisms and therapeutic opportunities Laquinimod (ABR-215062) for the treatment of relapsing multiple sclerosis.Act1 mediates IL-17-induced EAE pathogenesis selectively in NG2+ glial cells Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.Interferon-γ activates nuclear factor-κ B in oligodendrocytes through a process mediated by the unfolded protein response.Host microbiota constantly control maturation and function of microglia in the CNS.USP18 lack in microglia causes destructive interferonopathy of the mouse brain.Astrocyte-derived VEGF-A drives blood-brain barrier disruption in CNS inflammatory disease.Reduced astrocytic NF-κB activation by laquinimod protects from cuprizone-induced demyelination.Control of IL-17 receptor signaling and tissue inflammation by the p38α-MKP-1 signaling axis in a mouse model of multiple sclerosis Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.NF-κB signalling requirement for brain myelin formation is shown by genotype/MRI phenotype correlations in patients with Xq28 duplications.Prodegenerative IκBα expression in oligodendroglial α-synuclein models of multiple system atrophy.Astroglial NF-kB contributes to white matter damage and cognitive impairment in a mouse model of vascular dementia Microglial CX3CR1 promotes adult neurogenesis by inhibiting Sirt 1/p65 signaling independent of CX3CL1 Transient IKK2 activation in astrocytes initiates selective non-cell-autonomous neurodegeneration.The role of IκB kinase complex in the neurobiology of Huntington's disease.Molecular pharmacodynamics of new oral drugs used in the treatment of multiple sclerosis NF-κB signaling regulates myelination in the CNS.NF-κB activation protects oligodendrocytes against inflammation.CXCR3 signaling in glial cells ameliorates experimental autoimmune encephalomyelitis by restraining the generation of a pro-Th17 cytokine milieu and reducing CNS-infiltrating Th17 cells.Vinpocetine inhibits oligodendroglial precursor cell differentiation.Phosphorylation of αB-crystallin supports reactive astrogliosis in demyelination.The Effect of Melatonin on Behavioral, Molecular, and Histopathological Changes in Cuprizone Model of Demyelination.Activation of the unfolded protein response enhances motor recovery after spinal cord injury.Dysfunctional NF-κB and brain myelin formation.Astrocytes play a key role in EAE pathophysiology by orchestrating in the CNS the inflammatory response of resident and peripheral immune cells and by suppressing remyelination.Protective Effect of a cAMP Analogue on Behavioral Deficits and Neuropathological Changes in Cuprizone Model of Demyelination.Effect of glial cells on remyelination after spinal cord injury.Functional antagonism of sphingosine-1-phosphate receptor 1 prevents cuprizone-induced demyelination.Fingolimod may support neuroprotection via blockade of astrocyte nitric oxide.Resveratrol Promotes Remyelination in Cuprizone Model of Multiple Sclerosis: Biochemical and Histological Study.A TLR/AKT/FoxO3 immune tolerance-like pathway disrupts the repair capacity of oligodendrocyte progenitors.Inflammatory and cytotoxic effects of bifenthrin in primary microglia and organotypic hippocampal slice cultures.Role of nuclear factor κB in multiple sclerosis and experimental autoimmune encephalomyelitis

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IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous system

http://www.wikidata.org/entity/Q33749325

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

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IkappaB kinase 2 determines ol ...... in the central nervous system

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Daniel Erny

http://www.w3.org/2001/XMLSchema#string

Domenico Del Turco

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Geert van Loo

http://www.w3.org/2001/XMLSchema#string

Jenni Raasch

http://www.w3.org/2001/XMLSchema#string

John R Bethea

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Klaus-Peter Knobeloch

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Marco Prinz

http://www.w3.org/2001/XMLSchema#string

Markus Knust

http://www.w3.org/2001/XMLSchema#string

Nicolas Zeller

http://www.w3.org/2001/XMLSchema#string

Thomas Blank

http://www.w3.org/2001/XMLSchema#string

P2860

The POU factor Oct-6 and Schwann cell differentiation

IkappaB kinase promotes tumorigenesis through inhibition of forkhead FOXO3a

Effects of glia maturation factor overexpression in primary astrocytes on MAP kinase activation, transcription factor activation, and neurotrophin secretion

Integrating cell-signalling pathways with NF-kappaB and IKK function

IKK mediates ischemia-induced neuronal death

Axonal neuregulin-1 regulates myelin sheath thickness

Shared principles in NF-kappaB signaling

Disruption of the glucocorticoid receptor gene in the nervous system results in reduced anxiety

Regulation and function of NF-kappaB transcription factors in the immune system

Laser microdissection reveals regional and cellular differences in GFAP mRNA upregulation following brain injury, axonal denervation, and amyloid plaque deposition.

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1184-1198

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10.1093/BRAIN/AWQ359

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Pt 4

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134

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Manolis Pasparakis

Alexander Mildner

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2011-02-10T00:00:00Z

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49826287

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21310728

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oligodendrocyte

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4055835

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