Inducible protein knockout reveals temporal requirement of CaMKII reactivation for memory consolidation in the brain - Wikidata - awgldk - TriplyDB

Inducible protein knockout reveals temporal requirement of CaMKII reactivation for memory consolidation in the brain

Inducible protein knockout reveals temporal requirement of CaMKII reactivation for memory consolidation in the brain

http://www.wikidata.org/entity/Q34923757

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Calmodulin-kinases: modulators of neuronal development and plasticity Structural, Synaptic, and Epigenetic Dynamics of Enduring Memories Advances in cortical modulation of pain Mass Spectrometry-Based Approaches to Understand the Molecular Basis of Memory Stage-specific inhibition of TrkB activity leads to long-lasting and sexually dimorphic effects on body weight and hypothalamic gene expression Identification of hypothalamic neuron-derived neurotrophic factor as a novel factor modulating appetite The organization of recent and remote memories Directed evolution of a small-molecule-triggered intein with improved splicing properties in mammalian cells Plastic synaptic networks of the amygdala for the acquisition, expression, and extinction of conditioned fear Neural encoding of the concept of nest in the mouse brain.Transgenic up-regulation of alpha-CaMKII in forebrain leads to increased anxiety-like behaviors and aggression.Inducible and selective erasure of memories in the mouse brain via chemical-genetic manipulation.Transient inhibition of TrkB kinase after status epilepticus prevents development of temporal lobe epilepsy.Identification of network-level coding units for real-time representation of episodic experiences in the hippocampus.Investigation of molecular mechanism of chronic pain in the anterior cingulate cortex using genetically engineered mice Frizzled-5, a receptor for the synaptic organizer Wnt7a, regulates activity-mediated synaptogenesis.A semisynthetic epitope for kinase substrates.genetic overexpression of NR2B subunit enhances social recognition memory for different strains and species Assignment of protein function in the postgenomic era.Cre-Lox Neurogenetics: 20 Years of Versatile Applications in Brain Research and Counting…Optimizing small molecule inhibitors of calcium-dependent protein kinase 1 to prevent infection by Toxoplasma gondii Bidirectional control of BK channel open probability by CAMKII and PKC in medial vestibular nucleus neurons The molecular and cellular biology of enhanced cognition.New pyrazolopyrimidine inhibitors of protein kinase d as potent anticancer agents for prostate cancer cells BET bromodomain proteins are required for glioblastoma cell proliferation.Temporal and region-specific requirements of αCaMKII in spatial and contextual learning.Visceral hyperalgesia induced by forebrain-specific suppression of native Kv7/KCNQ/M-current in mice.Dentate gyrus-specific manipulation of beta-Ca2+/calmodulin-dependent kinase II disrupts memory consolidation.Neuronal calcium/calmodulin-dependent protein kinase II mediates nicotine reward in the conditioned place preference test in mice.The CaMKII/NMDAR complex as a molecular memory TrkB kinase is required for recovery, but not loss, of cortical responses following monocular deprivation Efficient reproduction of cynomolgus monkey using pronuclear embryo transfer technique.A novel behavioral paradigm for assessing the concept of nests in mice Tissue-specific PKA inhibition using a chemical genetic approach and its application to studies on sperm capacitation.TrkB signaling is required for both the induction and maintenance of tissue and nerve injury-induced persistent pain.Neural activity changes underlying the working memory deficit in alpha-CaMKII heterozygous knockout mice.Towards transgenic primates: What can we learn from mouse genetics?APP Deletion Accounts for Age-Dependent Changes in the Bioenergetic Metabolism and in Hyperphosphorylated CaMKII at Stimulated Hippocampal Presynaptic Active Zones.Guiding neuronal development with in situ microfabrication.A role for cerebellum in the hereditary dystonia DYT1.

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Inducible protein knockout reveals temporal requirement of CaMKII reactivation for memory consolidation in the brain

http://www.wikidata.org/entity/Q34923757

description

2003 nî lūn-bûn

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2003 թվականի մարտին հրատարակված գիտական հոդված

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2003年の論文

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Inducible protein knockout rev ...... ory consolidation in the brain

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Inducible protein knockout rev ...... ory consolidation in the brain

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Inducible protein knockout rev ...... ory consolidation in the brain

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Inducible protein knockout rev ...... ory consolidation in the brain

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Proceedings of the National Academy of Sciences of the United States of America

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Inducible protein knockout rev ...... ory consolidation in the brain

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Chao Zhang

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Daphne A Robinson

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Eiji Shimizu

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Hiromi Morimoto

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Huimin Wang

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Joe Z Tsien

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Kevan M Shokat

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Maureen Kyin

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Min Zhuo

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Peter J Alaimo

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P2860

Genetic enhancement of learning and memory in mice

A synaptic model of memory: long-term potentiation in the hippocampus

The protein kinase complement of the human genome

The protein kinase family: conserved features and deduced phylogeny of the catalytic domains

Deficient hippocampal long-term potentiation in alpha-calcium-calmodulin kinase II mutant mice

Alpha-CaMKII-dependent plasticity in the cortex is required for permanent memory

Impaired spatial learning in alpha-calcium-calmodulin kinase II mutant mice

The essential role of hippocampal CA1 NMDA receptor-dependent synaptic plasticity in spatial memory

Enrichment induces structural changes and recovery from nonspatial memory deficits in CA1 NMDAR1-knockout mice

Conditional restoration of hippocampal synaptic potentiation in Glur-A-deficient mice

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10.1073/PNAS.0636870100

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