Rad51 inhibition is an effective means of targeting DNA repair in glioma models and CD133+ tumor-derived cells
about
Connecting Malfunctioning Glial Cells and Brain Degenerative DisordersGenome management and mismanagement--cell-level opportunities and challenges of whole-genome duplicationThe efficiency of homologous recombination and non-homologous end joining systems in repairing double-strand breaks during cell cycle progressionRAD51 Is a Selective DNA Repair Target to Radiosensitize Glioma Stem Cells.Contributions of the RAD51 N-terminal domain to BRCA2-RAD51 interaction.Integrin αVβ3 silencing sensitizes malignant glioma cells to temozolomide by suppression of homologous recombination repairRad51 supports triple negative breast cancer metastasis.HOXA10 is associated with temozolomide resistance through regulation of the homologous recombinant DNA repair pathway in glioblastoma cell lines.Downregulation of BRCA1-BRCA2-containing complex subunit 3 sensitizes glioma cells to temozolomideTumor-associated mutations in a conserved structural motif alter physical and biochemical properties of human RAD51 recombinase.Complexes of mismatched and complementary DNA with minor groove binders. Structures at nucleotide resolution via an improved hydroxyl radical cleavage methodology.Acquisition of meiotic DNA repair regulators maintain genome stability in glioblastoma.A radiosensitizing effect of RAD51 inhibition in glioblastoma stem-like cells.Exonuclease 1 (Exo1) is required for activating response to S(N)1 DNA methylating agents.Retinoblastoma Binding Protein 4 Modulates Temozolomide Sensitivity in Glioblastoma by Regulating DNA Repair Proteins.FoxM1 inhibition sensitizes resistant glioblastoma cells to temozolomide by downregulating the expression of DNA-repair gene Rad51.In vitro and in vivo radiosensitization induced by hydroxyapatite nanoparticles.Complex DNA repair pathways as possible therapeutic targets to overcome temozolomide resistance in glioblastoma.CpG oligodeoxyribonucleotide 7909 enhances radiosensitivity via downregulating Oct-4 expression in radioresistant lung cancer cellsRegulation of DNA Alkylation Damage Repair: Lessons and Therapeutic Opportunities.Paradoxical Role of High Mobility Group Box 1 in Glioma: A Suppressor or a Promoter?Triggering of the TRPV2 channel by cannabidiol sensitizes glioblastoma cells to cytotoxic chemotherapeutic agents.Chemotherapeutic Drugs: DNA Damage and Repair in Glioblastoma.Increased sensitivity to ionizing radiation by targeting the homologous recombination pathway in glioma initiating cells.Endoplasmic reticulum stress-inducing drugs sensitize glioma cells to temozolomide through downregulation of MGMT, MPG, and Rad51Akt1 Stimulates Homologous Recombination Repair of DNA Double-Strand Breaks in a Rad51-Dependent Manner.Radiosensitization of esophageal cancer cells ECA109 by knockdown of H2AX.Radiosensitization by CpG ODN7909 in an epidermoid laryngeal carcinoma Hep-2 cell line.Inhibition of RAD51 by siRNA and Resveratrol Sensitizes Cancer Stem Cells Derived from HeLa Cell Cultures to Apoptosis.Targeting Long Noncoding RNA HMMR-AS1 Suppresses and Radiosensitizes Glioblastoma.
P2860
Q26744443-BD8B071E-6868-4098-84F1-6C131312A933Q26774293-06109CF5-8A6C-435A-8717-60EBFCF575FDQ28534631-B94E0BA8-3151-44E8-A8B7-25A6D8819C81Q30365298-6C3DAE0C-3A3A-4979-9603-4D5AAE2F9963Q30551528-93118249-CE10-4463-8C47-EB14270F41B5Q33709432-3370ABDA-96D8-4070-9780-D65EF58C16DCQ33917622-E3026458-E8E4-4C31-9494-7E47E8CF6D1AQ33925330-E974F0B6-FF23-4490-A9E0-C603EE94401FQ34786598-45BC7650-2B3E-4282-81B0-4C03BDD07F75Q35097983-5B83E772-DDB2-4A13-A8FB-FFB7800EE56DQ35300516-9AE4B2D2-34E5-4752-A62D-0B70B931E501Q35612804-CE31AC5D-251F-4D3E-9336-86F6358291F8Q36096420-43D34F4B-3A44-4903-B142-DD089AE71A97Q36421123-68913053-F237-4028-93DB-5202FC70AEB7Q36723302-860C32DC-471B-4696-8A68-97855DFE03FCQ36803405-755C7C78-C2F9-4DF8-B299-0009E54B3DEDQ36943880-2B7BD06B-47ED-491D-8F74-8B00A22D5C6BQ38066230-60334468-F385-4DB9-BEBE-16283CB34C2CQ38860264-FEF39E19-1E91-489D-9E70-224710D9117DQ38999756-BA54D19F-3DEC-43E6-912F-E85CC191E834Q39223354-30CC599E-ED88-440D-89B6-9F4A5138AAA2Q39256859-0D89A757-6627-494C-801F-0EFBDCDBD08FQ39354541-A0E108A1-BD32-4874-A103-C296D3EC9629Q41135252-1D02B25B-E086-4A5C-948E-587D30E730F0Q42014579-E3C375F5-879C-43D6-B4E4-F6A03CECDD18Q47383621-6D35E191-F550-423A-A0E7-B88A7C7D4619Q47896370-4276580D-B071-4044-91C1-498595E050A6Q49799180-099A29B4-877C-4000-B9A9-392C2EF46643Q52571383-2AE8E361-17BF-4EF8-BBE2-F29A8BFADFA5Q55313011-9EF130A7-667D-4D0C-8F96-5C87E0513CD2
P2860
Rad51 inhibition is an effective means of targeting DNA repair in glioma models and CD133+ tumor-derived cells
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@ast
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en-gb
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@nl
type
label
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@ast
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en-gb
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@nl
prefLabel
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@ast
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en-gb
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@nl
P2093
P2860
P356
P1433
P1476
Rad51 inhibition is an effecti ...... and CD133+ tumor-derived cells
@en
P2093
Christine Martindale
George Sioftanos
Ka Ian Lio
Marisa Alcaide-German
Maya Shaked-Rabi
Mulugeta Worku
Sara Bourne
P2860
P304
P356
10.1093/NEUONC/NOR010
P577
2011-03-01T00:00:00Z